JP2023511277A5 - - Google Patents

Info

Publication number
JP2023511277A5
JP2023511277A5 JP2022542918A JP2022542918A JP2023511277A5 JP 2023511277 A5 JP2023511277 A5 JP 2023511277A5 JP 2022542918 A JP2022542918 A JP 2022542918A JP 2022542918 A JP2022542918 A JP 2022542918A JP 2023511277 A5 JP2023511277 A5 JP 2023511277A5
Authority
JP
Japan
Prior art keywords
cancer
ccr5
tumor
seq
metastatic
Prior art date
Legal status (The legal status is an assumption and is not a legal conclusion. Google has not performed a legal analysis and makes no representation as to the accuracy of the status listed.)
Pending
Application number
JP2022542918A
Other languages
English (en)
Japanese (ja)
Other versions
JP2023511277A (ja
Filing date
Publication date
Application filed filed Critical
Priority claimed from PCT/US2021/013289 external-priority patent/WO2021146323A1/en
Publication of JP2023511277A publication Critical patent/JP2023511277A/ja
Publication of JP2023511277A5 publication Critical patent/JP2023511277A5/ja
Pending legal-status Critical Current

Links

JP2022542918A 2020-01-13 2021-01-13 Ccr5陽性の転移性のがんの処置のためのccr5結合剤 Pending JP2023511277A (ja)

Applications Claiming Priority (7)

Application Number Priority Date Filing Date Title
US202062960613P 2020-01-13 2020-01-13
US62/960,613 2020-01-13
US202062968954P 2020-01-31 2020-01-31
US62/968,954 2020-01-31
US202062977023P 2020-02-14 2020-02-14
US62/977,023 2020-02-14
PCT/US2021/013289 WO2021146323A1 (en) 2020-01-13 2021-01-13 Ccr5 binding agent for treatment of ccr5 positive metastatic breast cancer

Publications (2)

Publication Number Publication Date
JP2023511277A JP2023511277A (ja) 2023-03-17
JP2023511277A5 true JP2023511277A5 (https=) 2024-03-18

Family

ID=76760851

Family Applications (1)

Application Number Title Priority Date Filing Date
JP2022542918A Pending JP2023511277A (ja) 2020-01-13 2021-01-13 Ccr5陽性の転移性のがんの処置のためのccr5結合剤

Country Status (11)

Country Link
US (1) US20210214448A1 (https=)
EP (1) EP4090675A4 (https=)
JP (1) JP2023511277A (https=)
KR (1) KR20220127859A (https=)
CN (1) CN115003691A (https=)
AU (1) AU2021207851A1 (https=)
BR (1) BR112022012821A2 (https=)
CA (1) CA3163060A1 (https=)
IL (1) IL294113A (https=)
MX (1) MX2022008632A (https=)
WO (1) WO2021146323A1 (https=)

Families Citing this family (1)

* Cited by examiner, † Cited by third party
Publication number Priority date Publication date Assignee Title
BR112021019405A2 (pt) * 2019-04-01 2021-11-30 Cytodyn Inc Agentes anti-ccr5 e métodos de tratamento que bloqueiam a metástase do câncer ou aumentam a morte celular induzida pela quimioterapia prejudicial ao dna

Family Cites Families (13)

* Cited by examiner, † Cited by third party
Publication number Priority date Publication date Assignee Title
US7122185B2 (en) * 2002-02-22 2006-10-17 Progenics Pharmaceuticals, Inc. Anti-CCR5 antibody
NZ534947A (en) * 2002-02-22 2008-03-28 Progenics Pharm Inc Anti-CCR5 antibody that binds to CCR5 on the surface of a human cell.
WO2007113648A2 (en) * 2006-04-05 2007-10-11 Pfizer Products Inc. Ctla4 antibody combination therapy
JP6518585B2 (ja) * 2012-05-14 2019-05-22 リチャード ジー. ペステル がんの治療のためのccr5の修飾薬の使用
CN106413751A (zh) * 2014-05-21 2017-02-15 辉瑞大药厂 用于治疗癌症的抗ccr4抗体和4‑1bb激动剂的组合
WO2016073380A1 (en) * 2014-11-03 2016-05-12 Genentech, Inc. Method and biomarkers for predicting efficacy and evaluation of an ox40 agonist treatment
CN107847514A (zh) * 2015-06-23 2018-03-27 西托戴恩股份有限公司 炎症、癌症、自身免疫和其它病症中ccl5配体结合ccr5受体的抑制作用和ccr5/ccl5轴信号传导的改变
KR20190003958A (ko) * 2016-04-15 2019-01-10 제넨테크, 인크. 암의 치료 및 모니터링 방법
WO2018085513A1 (en) * 2016-11-04 2018-05-11 Genentech, Inc. Treatment of her2-positive breast cancer
CN111886249A (zh) * 2017-09-18 2020-11-03 西托戴恩股份有限公司 用于鉴定和治疗适合长期抗-ccr5剂治疗的hiv-1感染患者亚群的筛选方法
EP3773676A4 (en) * 2018-04-03 2022-05-18 Dragonfly Therapeutics, Inc. Proteins binding nkg2d, cd16 and an antigen associated with tumors, mdscs and/or tams
CN112639137A (zh) * 2018-07-02 2021-04-09 茵赛德斯有限公司 用于检测癌症相关细胞群体、进行转移性癌症筛查及治疗的方法
BR112021019405A2 (pt) * 2019-04-01 2021-11-30 Cytodyn Inc Agentes anti-ccr5 e métodos de tratamento que bloqueiam a metástase do câncer ou aumentam a morte celular induzida pela quimioterapia prejudicial ao dna

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