JP2013209293A - Method for controlling sigatoka disease - Google Patents

Abstract

PROBLEM TO BE SOLVED: To provide a new power-saving control method having a reduced load on an environment in order to control sigatoka disease efficiently, and having the advantage of making it possible to reduce the amount of a spray agent or the like.SOLUTION: In a method for controlling sigatoka disease, leaf sheath parts (pseudostem parts) of plants of the family Musaceae infected or anticipated to be infected with the sigatoka disease are treated by injection of an effective amount of a sigatoka disease control agent to control the sigatoka disease occurring in the plants of the family Musaceae.

Description

  TECHNICAL FIELD The present invention relates to a method for controlling Sigatoka disease occurring in a Musaceae plant, particularly a banana.

  Plants belonging to the genus Bacolaceae, in particular bananas, are a collective term for a variety of edible varieties and refer to the fruits. The plant includes East Asia such as China and Taiwan, Southeast Asia such as Philippines, Indonesia, Thailand and Vietnam, West Asia such as India, Brazil, Ecuador, Costa Rica, Mexico, Guatemala, Colombia, Peru and other Central and South America, Tanzania, Congo, It is mainly cultivated in Africa such as Uganda and South Africa. Plantation is also cultivated throughout the year. Bananas grown and harvested in these countries are exported worldwide.

  In banana cultivation, the root part including the stem part is in the basement, and the part like a stalk that extends high above the ground part is called a fake stalk. A long oval leaf (leaf blade) extends greatly from the tip, and a fruit axis comes out from the tip of the pseudostem, and a whole bunch including a finger and a bunch is formed, and a banana grows in the whole bunch. After harvesting the banana, the pseudostem (parent strain) is cut at about 2 m above the ground. At that time, a new pseudostem part (child strain) newly appearing from the underground root part is propagated in the same manner, or a new pseudostem part (child strain) is bred and propagated, and banana harvesting is repeated. The number of leaves (functional leaves) is important for banana cultivation, and since there are 3 months from the last leaf extraction (before flowering) to harvest, at least 12-13 functional leaves during the flowering period It is necessary to leave a degree. If the number of functional leaves is insufficient, the plant is cut down during cultivation.

The main disease of banana cultivation is Sigatoka disease, which includes yellow Yellow Sigatoka disease and black Black Sigatoka disease.
Yellow Sigatoka disease is caused by Mycosphaerell musicola, black Sigatoka disease is a disease caused by Mycosphaerella fijiensis, and particularly Black Sigatoka disease occurs during high temperature and high humidity. It is a disease that turns the leaves black and inhibits photosynthesis to halve the yield. The incubation period of Sigatoka disease ranges from 3 weeks to 2 months, and the initial symptoms of the affected banana are small spots appearing on the back of old leaves 4-5 below the top leaf, and eventually yellow along the veins. The lesions gradually expand and become dark brown. When the damage is severe, most leaves die and hang down, leaving only one or two of the central leaves. Control of these diseases begins by monitoring the leaf portion of the banana, and the necessity of control is determined by the position of the youngest infected leaf and the degree of infection.

  When the occurrence of Sigatoka disease is confirmed, organosulfur fungicides such as Mancozeb, organochlorine fungicides such as chlorothalonil, pyrimidine fungicides such as pyrimethanil, Tridemorph, fenpropimorph Morpholine fungicides such as (Fenpropimorph), difenoconazole, diterconol, bittertanol, propiconazole, epoxyconazole such as pirocrosslobin Many strobilurin fungicides such as azoxystrobin are used Are, these fungicides several at 20 day intervals from 10 days is sprayed.

  Also, in recent years, a group of compounds called resistance inducers (SAR agents) that in itself show no bactericidal activity or are weak but induce disease resistance to plants are useful as disease control agents for various crops. Thiazinyl (generic name: tiadinil) and isothianyl (generic name: isotianil), acibenzolar-S-methyl (generic name: acibenzolar-S-methyl), carpropamide, probenazole (probenazole), salicylic acid (Salicylic acid), dichloronicotinic acid and the like are known as the resistance inducer. Further, 2 ′, 4′-dimethoxy-4-cyclopropyl-1,2,3-thiadiazole-5-carboxanilide (hereinafter referred to as “compound A”), which is a similar compound to thiazinyl, is also useful as a plant disease control agent. Although it is a known compound (see, for example, Patent Document 1), it is considered a resistance inducer. Among these resistance inducers, it is known that isotianil is effective for banana black sigatoka disease by spraying treatment (see, for example, Patent Document 2).

  The compound represented by the above general name or chemical name is a known compound, and can be produced by purchasing a commercially available product or by a method described in a known document (for example, see Non-Patent Document 1).

JP 2007-99749 A International Publication No. 2010/037482 Pamphlet

The Pesticide Manual 14th Edition (British Crop Production Council)

  As mentioned above, control of Sigatoka disease is the most important issue for banana cultivation, but the conventional technology performs many times of drug treatment, so the labor is tremendous, and the emergence of drug-resistant bacteria is more difficult to control I have to. Therefore, there is a demand for a new labor-saving control method that has advantages such as less burden on the environment for efficiently controlling Sigatoka disease, which is difficult to control, and the ability to reduce the amount of sprayed drug.

  As a result of intensive studies to solve the above-mentioned problems, the present inventors have efficiently infected this disease by injecting a banana Sigatoka disease control agent into a banana pseudostem (leaf sheath) or the like with an injector or the like. The present inventors have found a control method that can be controlled, and have completed the control method of the present invention. That is, according to the control method of injecting a chemical solution into the pseudostem part of the present invention, it is possible to efficiently control Sigatoka disease occurring in bananas, and to newly create a pseudostem part (child strain) newly emerging from the underground stem part Sigatoka disease that occurs can be controlled, it has been found that it has a significant long-term control effect compared to the spray treatment of Sigatoga disease control agent, and can reduce the burden on the environment and drug sprayers as much as possible, The present invention has been completed.

That is, the present invention relates to at least the following inventions:
(1) An effective amount of an agent for controlling Sigatoka disease is injected into a pseudostem part (leaf sheath part) of a plant of the Glyceae plant in which Sigatoka disease has occurred or is predicted to occur, and the plant of the Glyceae plant A method for controlling Sigatoka disease, which comprises controlling Sigatoka disease occurring in the body.
(2) The method for controlling Sigatoka disease according to (1), wherein the Sigatoka disease control agent is a resistance inducer or a penetrating fungicide.
(3) The method for controlling Sigatoka disease according to the above (2), wherein the resistance inducer is thiazinyl, isothianyl, compound A, acibenzoral-S-methyl, carpropamide, probenazole, salicylic acid, or dichloronicotinic acid.
(4) The method for controlling Sigatoka disease according to any one of (1) to (3) above, wherein the resistance inducer is thiazinyl, isothianyl, or compound A.

(5) The method for controlling Sigatoka disease according to (2) above, wherein the permeable fungicide is a strobilurin fungicide.
(6) Sigatoka disease according to (2) or (5) above, wherein the strobilurin fungicide is azoxystrobin, metminostrobin, picoxystrobin, pyraclostrobin, fluoxastrobin, dimoxystrobin or orisatrobin Control method.
(7) The method for controlling Sigatoka disease according to (2) above, wherein the permeable fungicide is an azole fungicide.
(8) The azole fungicide is tebuconazole, difenoconazole, propiconazole, cyproconazole, flusilazole, microbutanyl, flutriahol, triadimenol, hexaconazole, penconazole, diniconazole, triadimephone, ipconazole, bromconazole, cimeconazole, The method for controlling Sigatoka disease according to the above (7), which is imibenconazole, prothioconazole or imazalil.
(9) The method for controlling Sigatoka disease according to the above (1), wherein the Musaceae plant is a banana.
(10) The method for controlling a Sigatoka disease according to any one of the above (1) to (9), wherein the Sigatoka disease is a Sigatoka disease caused by Mycosphaerella musicola.
(11) The method for controlling a Sigatoka disease according to any one of (1) to (9), wherein the Sigatoka disease is a Sigatoka disease caused by Mycosphaella fijiensis.

(12) Sigatoka that occurs in the banana parent of the banana parent by injecting an effective amount of a Sigatoka disease control agent into the pseudostem (leaf sheath) of the banana parent that has or is predicted to have Sigatoka A method for controlling sigatoka disease in a banana seedling, characterized by controlling disease.
(13) The method for controlling Sigatoka disease according to (12), wherein the Sigatoka disease control agent is a resistance inducer or a penetrating fungicide.
(14) The method for controlling Sigatoka disease according to the above (13), wherein the resistance inducer is thiazinyl, isotianil, compound A, acibenzoral-S-methyl, carpropamide, probenazole, salicylic acid, or dichloronicotinic acid.
(15) The method for controlling Sigatoka disease according to the above (14), wherein the resistance inducer is thiazinyl, isotianil or compound A.

(16) The method for controlling Sigatoka disease according to the above (14), wherein the permeable fungicide is a strobilurin fungicide.
(17) The method for controlling Sigatoka disease according to the above (17), wherein the strobilurin-based fungicide is azoxystrobin, metminostrobin, picoxystrobin, pyraclostrobin, floxastrobin, dimoxystrobin or orisatrobin.
(18) The method for controlling Sigatoka disease according to (13), wherein the permeable fungicide is an azole fungicide.
(19) The azole fungicide is tebuconazole, difenoconazole, propiconazole, cyproconazole, flusilazole, microbutanyl, flutriahol, triadimenol, hexaconazole, penconazole, diniconazole, triadimephone, ipconazole, bromconazole, cimeconazole, The method for controlling Sigatoka disease according to the above (18), which is imibenconazole, prothioconazole or imazalil.

  The method for controlling Sigatoka disease of the present invention is effective for the Sigatoka disease control agent in the pseudostem part (leaf sheath part) of the plant body against Shigatoka disease occurring in or predicted to occur in the plantaceae, particularly banana. It is characterized by injecting treatment. The Sigatoka disease control method of the present invention is a simple treatment method compared with the conventional spraying treatment of Sigatoka disease drug, shows a remarkable control effect over a long period of time, and has an excellent control effect against drug resistant bacteria. It has an effect to prevent the onset of Sigatoka's disease on the new leaf sheath part (child strain) newly emerging from the underground rhizome part. Furthermore, the Sigatoka disease control method of the present invention is a control method that can reduce the number of treatments of the drug, reduce the amount of treatment chemicals, and can reduce the load on the environment.

  Examples of the Sigatoka disease control agent that can be used in the present invention include a resistance inducer or a penetrating fungicide. Examples of the resistance inducer include thiazinyl (generic name: tiadinil), isothianyl (generic name: isotianil), and compound A. , Acibenzoral-S-methyl (generic name: acibenzolar-S-methyl), carpropamide (generic name: carpropamid), probenazole (generic name: probenazole), salicylic acid (salicylic acid), dichloronicotinic acid (Dichloronic acid etc.) Among them, thiazinyl, compound A or isothianyl is preferred. Examples of osmotic fungicides include azoxystrobin (generic name: azoxystrobin), trifloxystrobin (generic name: trifloxystrobin), pyraclostrobin (generic name: pyraclostrobin), methinostrobin (generic name: methominostrobin), Stobilurin-based fungicides such as picoxystrobin (generic name: picoxystrobin), fluoxastrobin (generic name: fluoxastrobin), dimoxystrobin (generic name: dioxystrobin), oryastrostrobin (generic name: orysastrobin), tebuconazole (general Name: tebuconazole), epoxyconazole (generic name: epoxiconazole), difenoconazole (generic name: ifenoconazole), propiconazole (generic name: propiconazole), cyproconazole (generic name: cyproconazole), flusilazole (generic name: flusilazole), metconazole (generic name: metconazole), microbutanil (general name: metconazole) (Generic name: tetraconazole), fluquinconazole (generic name: flequinconazole), flutriahol (generic name: flutriafol), triadimenol (generic name: triadimenol), hexaconazole (generic name: hexaconazole), penconazole (general name) Name: penconazole), Vitertanol (generic name: bitteranol) Triticonazole (generic name: triticonazole), fenbuconazole (generic name: fenbuconazole), diniconazole (generic name: diniconazole), triadimephone (generic name: triadimefone), ipconazole (generic name: ipconazole), broconazole : Bromuconazole), cimeconazole (generic name: simconazole), imibenconazole (generic name: imibenconazole), prothioconazole (generic name: prothioconazole) (generic name: prochloraz), izobenazole (generic name: prochloraz) Use azole fungicides such as nazole (generic name: oxpoconazole). However, the Sigatoka disease control agent that can be used in the present invention is not limited to these active ingredients. These drugs are commercially available and are readily available from the market. Compound A can be produced by the method described in Patent Document 1.

The method for controlling Sigatoka disease of the present invention can be performed by injecting a commercially available liquid drug containing the above compound as an active ingredient into the leaf sheath of a plant body such as a banana with an injector such as a syringe. In addition, a drug containing Compound A as an active ingredient is prepared by dissolving Compound A in an appropriate inert carrier together with a surfactant and, if necessary, an auxiliary agent in an appropriate ratio, For example, it may be used in the form of a suspension, milk suspension, emulsion, liquid or the like.
The addition amount of the active ingredient in 100 parts by mass of the control agent for Sigatoka disease may be appropriately selected from the range of 0.1 to 50 parts by mass, and preferably in the range of 1 to 50 parts by mass. .

  As the inert carrier that can be used in the control method of the present invention, a liquid or solid inert carrier can be used. In the method of injecting into the leaf sheath, a liquid carrier is preferable, and the material that can be a liquid carrier is itself a solvent ability. In addition, the liquid carrier is selected from those having no solvent ability and capable of dispersing the active ingredient compound with the aid of an auxiliary agent, such as water, alcohols (for example, methanol, ethanol, isopropanol, butanol). , Ethylene glycol etc.), ketones (eg acetone, methyl ethyl ketone, methyl isobutyl ketone, diisobutyl ketone, cyclohexanone etc.), ethers (eg ethyl ether, dioxane, cellosolve, dipropyl ether, tetrahydrofuran etc.), aliphatic hydrocarbons ( For example, kerosene, mineral oil, etc.) Aromatic hydrocarbons (eg, benzene, toluene, xylene, solvent naphtha, alkylnaphthalene, etc.), halogenated hydrocarbons (eg, dichloroethane, chloroform, carbon tetrachloride, etc.), esters (eg, ethyl acetate, diisopropyl phthalate, Dibutyl phthalate, dioctyl phthalate, etc.), amides (eg, dimethylformamide, diethylformamide, dimethylacetamide, etc.), nitriles (eg, acetonitrile, etc.), dimethyl sulfoxides, and the like. It can be used in the form of a mixture.

  As other adjuvants, typical adjuvants exemplified below can be mentioned, and these adjuvants are used depending on the purpose, and used alone, in some cases, two or more kinds of adjuvants are used together. In some cases it is possible to use no adjuvants at all. Surfactants can be used for the purpose of emulsifying, dispersing, solubilizing and / or wetting of the active ingredient compound, such as polyoxyethylene alkyl ether, polyoxyethylene alkyl aryl ether, polyoxyethylene higher fatty acid ester, polyoxyethylene. Examples include surfactants such as resin acid esters, polyoxyethylene sorbitan monolaurate, polyoxyethylene sorbitan monooleate, alkylaryl sulfonates, naphthalene sulfonic acid condensates, lignin sulfonates, and higher alcohol sulfates. it can. As a peptizer for a suspension product, for example, auxiliary agents such as naphthalenesulfonic acid condensate and condensed phosphate can be used. As the antifoaming agent, for example, an auxiliary agent such as silicone oil can be used.

Examples of the plant belonging to the family Baxaceae which can control Sigatoka disease by the present control method include, for example, Gros Michel, Cavendish, Dwarf Cavendish, Dwarf Kinese, Enano, Catura, Giant Cavendish, Gran Enano, Grande Nine, Williams Hybrid, ValerieR, ballyR (Poyo), Lacatan, Monte Cristo, It can be exemplified over preparative Rondo (Bout rond) Musaceae plants such as, but not limited to these Musaceae plant. Particularly preferred are all bananas including Giant Cavendish, Dwarf Cavendish, Cavendish, Plantain, Red Banana, Apple Banana, Monkey Banana, Island Banana and the like that are generally cultivated for food.
For the genus Musaceae which is the target of the control method of the present invention, there are useful traits such as herbicide resistance genes, insecticidal pest resistance genes, antipathogenic substance production genes, oil component modification and amino acid content enhancement traits. The same plant which is a genetically modified organism (GMO) which has undergone gene conversion is also included.

  In the method of the present invention, the Sigatoka disease control agent of the Ganoderma family plant is used in an amount effective for controlling Sigatoka disease as it is, or appropriately diluted with water or the like, or suspended in order to control Sigatoka disease. What is necessary is just to inject | pour into the pseudostem part of the plant body of the banana plant which the disease generate | occur | produced or is estimated to generate | occur | produce using injection devices, such as a syringe. As long as the banana fruit is in the growing season or after harvesting the banana fruit, the injection site and the drug dose may be injected at a predetermined dose of about 30 cm to 1 m from the ground surface of the remaining pseudostem. This treatment controls the occurrence of Sigatoka disease that occurs in growing bananas, and if it is after harvesting, it can also control Sigatoka disease that occurs in the new pseudostem (child strain) that emerges from the rootstock. it can. That is, the chemical treatment of the pseudo stalk portion of the banana parent strain can not only control the Sigatoka disease of the parent strain itself, but also control the Sigatoka disease occurring in the child strain.

  The banana sigatoka disease control agent of the present invention may be used by formulating it into a normal dosage form, for example, a liquid dosage form such as an emulsion, a wettable powder, a granular wettable powder, a flowable preparation, a liquid preparation, etc. In the case of an emulsion particularly frequently used in normal plantation cultivation, the number of bananas grown per hectare is 1500 to 2000, although it varies depending on the blending ratio, weather conditions, formulation form, treatment time, treatment method, treatment place, etc. Since this is cultivated to the extent that 0.4 l to 3 l of a predetermined amount of drug is sprayed per hectare in general drug spraying, the amount corresponds to 0.2 ml to 2 ml per bottle. In the control method of the present invention, a drug solution diluted to a predetermined dose in the banana leaf sheath may be injected into the pseudostem with a dose of 0.2 ml to 2 ml per bottle, for example, with a syringe. This is in the range of 60 mg to 400 mg per banana plant as the amount of active ingredient, but the amount in the present invention is not limited thereto. With the control method of the present invention, the amount of drug used can be reduced, and unlike the case where the user sprays the user, the user is not exposed to the chemical solution (in the case of general spraying), and the Sigatoka disease can be controlled more safely. Is.

  In the method for controlling Sigatoka disease of the present invention, an agricultural or horticultural fungicide or an agricultural or horticultural insecticide other than the above-mentioned fungicide can be used by mixing as appropriate. However, the agricultural and horticultural fungicides or agricultural and horticultural insecticides are not limited to these. Examples of agricultural and horticultural fungicides include isoprothiolane, tricyclazole, azoxystrobin, pyracrostrobin, propiconazole, and organic copper (copper). , Methminostrobin, carpropamide, fthalide, kasugamycin, pyroquilon, probenazole, ciobyl-S-methyl-S-methyl (S-methyl) utolanil, mepronil, furamethpyr, pencyclone, thifluzamide, validamycin, diclomezine, ferrimzone (f). Hexaconazole, difenoconazole, bittertanol,

Epoxyconazole, triadimephone, prochloraz, fenpropimorph, tridemorph, metalloxy, basic copper chloride (p), basic copper chloride (p), basic copper chloride (p) sulfate, mancozeb, propineb, ziram, thiuram, cymoxanil, chlorothalonil, difenoconazole, difenoconazole, difenoconazole in), iprodione, procymidone, benomyl, thiophanate-methyl, carbendazim, fosetyl-aluminoxilic, oxolinic acid, fludioxonil, mepanipyrim, cyprodinil, pyrimethanil, tolcrofos-methyl, fenoxanil and phenoxil Can The

  Examples of agricultural and horticultural insecticides include benfuracarb (generic name: benfurcarb), carbosulfan (generic name: carbosulfan), PHC (generic name: propoxur), furiocarb (generic name: furathiocarb), mesomil (generic name: methomyl). , NAC (generic name: carbaryl), bendiocarb (generic name: bendiocarb), alanicarb (generic name: alaniccarb), imidacloprid (generic name: nitenpyram), nitenpyram (generic name: nitenpyram), acetamiprid (generic name: aci id) , Thiamethoxam (generic name: thiamethoxam), clothianidin (generic name: clothianidin), dinotefuran (generic name: d notofuran, propofos (generic name: propaphos), ethylthiomethone (generic name: disulfon), diazinon (generic name: diazinon), trichlorfon (generic name: trichlorfon), fenthion (generic name: fenthion), acephate (generic name: acephate) ,

Isoxathion (generic name: isoxathion), isofenphos (generic name: isofenphos), chlorpyrifos-methyl (generic name: chlorpyrifos-methyl), chlorfenvinphos (generic name: chlorfenvinphos), dimethoate (generic name: dimethoate), honophos (generic name: dimethoate) Name: fonofos), pyridafenthion (generic name: pyrifafention), monocrotofos (generic name: monocrotophos), cartap (generic name: cartap), thiocyclam (generic name: thiocyclam), bensultap (generic name: bensultap) cycloprotoline (generic name: bensultap) Name: cycloprothrin), etofenprox (generic name: etofenprox), Rafuruofen (generic name: silafluofen), tefluthrin (generic name: tefluthrin), buprofezin (generic name: buprofezin), flufenoxuron (generic name: flufenoxuron),

Cyromazine (generic name: cyromazine), rufenuron (generic name: lufenuron), tebufenozide (generic name: tebufenozide), methoxyphenozide (generic name: methoxyfenozide) (generic name: chromofenomide) General name: pyraclofos), Phostiazate (generic name: fosthiazate), Fenpyroximate (generic name: fenproximate), Fipronil (generic name: fipronil), Endosulfan (generic name: endosulfan), Methaldehyde (general name: metaldehyde), Ememactin Benzoate (generic name: emanectine-benzoate) Spinosad (common name: spinosad), chlorfenapyr (common name: chlorfenapyr), indoxacarb (common name: indoxacarb), etc. mixed to be able to use.

Although the typical formulation example and test example of this invention are illustrated below, this invention is not limited to these. In the prescription examples or test examples, “parts” means “parts by weight”.
Formulation Example 1
Active ingredient of Sigatoka disease control agent of the present invention 50 parts Xylene 40 parts Mixture of polyoxyethylene nonylphenyl ether and calcium alkylbenzenesulfonate 10 parts The above is uniformly mixed and dissolved to prepare an emulsion.

Formulation Example 2
30 parts Solpol 3105 (manufactured by Toho Chemical Co., Ltd.) 5 parts Propylene glycol 5 parts Rhodopol (manufactured by Rhone-Poulenc) 2 parts Water 58 parts or more uniformly mixed and dispersed in water And flowable.

試験規模： １処理あたり４植物体
処理器具： １０ｍｌの注射器
処理量： ２．０ｍｌ／株（散布処理では３．２リットル／ｈａに相当する量） Test Example 1 Test for controlling Sigatoka disease occurring in the banana growth period 2 ml of a chemical solution obtained by diluting the drug (active ingredient: thiazinyl) of Example 2 with water to a banana at the beginning of the development of Sigatoka disease 5 times with water. The total number of functional leaves per plant (healthy leaves functioning) is calculated from the 11th day after the chemical treatment, and every week thereafter. The calculation was made in the same manner for 9 weeks (1 ward, 4 systems). Note that the number of functional leaves in the parent pseudostem-treated section indicates the number of functional leaves of the child pseudostem that comes from the same root.
The results are shown in Table 1.

Test scale: 4 plant treatment tools per treatment: 10 ml syringe treatment amount: 2.0 ml / strain (amount corresponding to 3.2 liters / ha in spraying treatment)

  From the results of this test, the thiazinyl infusion treatment group suppressed the onset and progression of banana shigatoka disease for a long time in both the parent stem pseudo-stem injection and the child strain pseudo-stem injection treatment, compared to no treatment. 12 or more were maintained, the control effect was remarkable, and the state which does not hinder the growth of a banana was maintained for a long time. That is, it was revealed that the control method of the present invention has an effect of suppressing damage caused by Sigatoka disease in the whole plant body. Moreover, it became clear that the control method of this invention has the special effect that generation | occurrence | production of the Sigatoka disease in a next-generation plant body can be suppressed in the process to the pseudostem part of a parent strain.

Test Example 2
The same drug as in Test Example 1 was treated in the same manner. As an index of the effect of the treatment, the number of healthy leaves per strain up to the position of the youngest infected leaf counted from the newest leaf at the top was calculated (1 ward 4 continuous system). In addition, the number of healthy leaves in the parent pseudostem treatment section indicates the number of healthy leaves of the child pseudostem that comes from the same root. The results are shown in Table 2.

The incubation period of Sigatoka disease infection was about 3 weeks to 2 months, and in the untreated area, the number of functional leaves necessary for the growing season of bananas decreased due to the infection and could not grow sufficiently. On the other hand, in the section where the control method of the present invention was implemented, the spread of Sigatoka disease infection can be suppressed by the injection treatment of the Sigatoka disease control agent, resulting in an increase in the number of healthy leaves and the bananas growing sufficiently. And high quality was maintained. Moreover, the control effect was also maintained for 60 days after the chemical treatment.
In addition, the number of spraying and the amount of medicine can be reduced as compared with conventional practice control, and the burden on the sprayer and the environment is reduced.

Claims (19)

  An effective amount of a Sigatoka disease control agent is injected into the pseudostem (leaf sheath) of a plant of the plantaceae plant in which Sigatoka disease has occurred or is predicted to occur, and the plant is generated in the plant of the plant family A method for controlling Sigatoka disease, comprising controlling Sigatoka disease.   The method for controlling Sigatoka disease according to claim 1, wherein the Sigatoka disease control agent is a resistance inducer or a penetrating fungicide.   The resistance inducer is thiazinyl, isothianyl, compound A (2 ′, 4′-dimethoxy-4-cyclopropyl-1,2,3-thiadiazole-5-carboxanilide), acibenzoral-S-methyl, carpropamide, probenazole, salicylic acid, The method for controlling Sigatoka disease according to claim 2, which is dichloronicotinic acid.   The resistance inducer is thiazinyl, isotianil or compound A (2 ', 4'-dimethoxy-4-cyclopropyl-1,2,3-thiadiazole-5-carboxanilide). How to control Sigatoka disease.   The method for controlling Sigatoka disease according to claim 2, wherein the permeable fungicide is a strobilurin fungicide.   The method for controlling Sigatoka disease according to claim 2 or 5, wherein the strobilurin-based fungicide is azoxystrobin, metminostrobin, picoxystrobin, pyraclostrobin, fluoxastrobin, dimoxystrobin or orisatrobin.   The method for controlling Sigatoka disease according to claim 2, wherein the permeable fungicide is an azole fungicide.   The azole fungicides are tebuconazole, difenoconazole, propiconazole, cyproconazole, flusilazole, microbutanyl, flutriahol, triadimenol, hexaconazole, penconazole, diniconazole, triadimephone, ipconazole, bromconazole, cimeconazole, imibenconazole The method for controlling Sigatoka disease according to claim 7, which is prothioconazole or imazalyl.   The method for controlling sigatoka disease according to claim 1, wherein the plantaceae is a banana.   The method for controlling a Sigatoka disease according to any one of claims 1 to 9, wherein the Sigatoka disease is a Sigatoka disease caused by Mycosphaerella musicola.   The method for controlling a Sigatoka disease according to any one of claims 1 to 9, wherein the Sigatoka disease is a Sigatoka disease caused by Mycospharella fijiensis.   An effective amount of a Sigatoka disease control agent is injected into the pseudostem (leaf sheath) of a banana parent strain where Sigatoka disease has occurred or is predicted to occur, thereby controlling Sigatoka disease occurring in the banana parent strain of the banana parent strain. A method for controlling sigatoka disease of a banana stock.   The method for controlling Sigatoka disease according to claim 12, wherein the Sigatoka disease control agent is a resistance inducer or a penetrating fungicide.   The resistance inducer is thiazinyl, isothianyl, compound A (2 ′, 4′-dimethoxy-4-cyclopropyl-1,2,3-thiadiazole-5-carboxanilide), acibenzoral-S-methyl, carpropamide, probenazole, salicylic acid, The method for controlling Sigatoka disease according to claim 13, which is dichloronicotinic acid.   15. The method for controlling Sigatoka disease according to claim 14, wherein the resistance inducer is thiazinyl, isotianil or compound A (2 ', 4'-dimethoxy-4-cyclopropyl-1,2,3-thiadiazole-5-carboxanilide).   The method for controlling Sigatoka disease according to claim 14, wherein the permeable fungicide is a strobilurin fungicide.   The method for controlling Sigatoka disease according to claim 17, wherein the strobilurin fungicide is azoxystrobin, metminostrobin, picoxystrobin, pyraclostrobin, fluoxastrobin, dimoxystrobin or orisatrobin.   The method for controlling Sigatoka disease according to claim 13, wherein the permeable fungicide is an azole fungicide.   The azole fungicides are tebuconazole, difenoconazole, propiconazole, cyproconazole, flusilazole, microbutanyl, flutriahol, triadimenol, hexaconazole, penconazole, diniconazole, triadimephone, ipconazole, bromconazole, cimeconazole, imibenconazole The method for controlling Sigatoka disease according to claim 18, which is prothioconazole or imazalil.