JP2007505825A5 - - Google Patents

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Publication number
JP2007505825A5
JP2007505825A5 JP2006525791A JP2006525791A JP2007505825A5 JP 2007505825 A5 JP2007505825 A5 JP 2007505825A5 JP 2006525791 A JP2006525791 A JP 2006525791A JP 2006525791 A JP2006525791 A JP 2006525791A JP 2007505825 A5 JP2007505825 A5 JP 2007505825A5
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JP
Japan
Prior art keywords
nucleic acid
sequence
polypeptide
acid molecule
cell cycle
Prior art date
Legal status (The legal status is an assumption and is not a legal conclusion. Google has not performed a legal analysis and makes no representation as to the accuracy of the status listed.)
Pending
Application number
JP2006525791A
Other languages
English (en)
Japanese (ja)
Other versions
JP2007505825A (ja
Filing date
Publication date
Application filed filed Critical
Priority claimed from PCT/EP2004/010308 external-priority patent/WO2005025624A2/en
Publication of JP2007505825A publication Critical patent/JP2007505825A/ja
Publication of JP2007505825A5 publication Critical patent/JP2007505825A5/ja
Pending legal-status Critical Current

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JP2006525791A 2003-09-15 2004-09-15 増殖性疾患の診断および治療における、細胞周期制御または細胞周期進行に影響を与える真核生物遺伝子の利用 Pending JP2007505825A (ja)

Applications Claiming Priority (2)

Application Number Priority Date Filing Date Title
US50265103P 2003-09-15 2003-09-15
PCT/EP2004/010308 WO2005025624A2 (en) 2003-09-15 2004-09-15 The use of eukaryotic genes affecting cell cycle control or cell cycle progression for diagnosis and treatment of proliferattive diseases

Publications (2)

Publication Number Publication Date
JP2007505825A JP2007505825A (ja) 2007-03-15
JP2007505825A5 true JP2007505825A5 (https=) 2007-11-08

Family

ID=34312411

Family Applications (1)

Application Number Title Priority Date Filing Date
JP2006525791A Pending JP2007505825A (ja) 2003-09-15 2004-09-15 増殖性疾患の診断および治療における、細胞周期制御または細胞周期進行に影響を与える真核生物遺伝子の利用

Country Status (6)

Country Link
US (1) US7648827B2 (https=)
EP (1) EP1682573B1 (https=)
JP (1) JP2007505825A (https=)
AU (1) AU2004271725B2 (https=)
CA (1) CA2538442A1 (https=)
WO (1) WO2005025624A2 (https=)

Families Citing this family (5)

* Cited by examiner, † Cited by third party
Publication number Priority date Publication date Assignee Title
US20160083397A1 (en) * 2013-04-19 2016-03-24 Narsimha Reddy Penthala Parthenolide derivatives and their modulation of processes controlled by regulated translation
US9487536B2 (en) 2013-11-08 2016-11-08 Board Of Trustees Of The University Of Arkansas Melampomagnolide B derivatives
US9469650B2 (en) 2013-11-08 2016-10-18 Board Of Trustees Of The University Of Arkansas Melampomagnolide B derivatives
WO2016090166A1 (en) 2014-12-03 2016-06-09 Board Of Trustees Of The University Of Arkansas Melampomagnolide b dimers
CA3012179A1 (en) 2016-01-29 2017-08-03 Bioventures, Llc Triazole derivatives of melampomagnolide b and methods of use thereof

Family Cites Families (8)

* Cited by examiner, † Cited by third party
Publication number Priority date Publication date Assignee Title
WO1995024223A1 (en) * 1994-03-08 1995-09-14 Thomas Jefferson University Inhibition of cell proliferation by e2f-1 antisense oligonucleotides
WO1996017933A2 (en) 1994-12-09 1996-06-13 Takeda Chemical Industries, Ltd. Dna encoding a cell growth inhibiting factor and its product
US6809193B2 (en) * 1997-08-13 2004-10-26 Isis Pharmaceuticals, Inc. Antisense oligonucleotide compositions and methods for the modulation of JNK proteins
US6183961B1 (en) * 1997-09-22 2001-02-06 The Regents Of The University Of California Methods and compositions for regulating cell cycle progression
US6506559B1 (en) 1997-12-23 2003-01-14 Carnegie Institute Of Washington Genetic inhibition by double-stranded RNA
WO2001029221A2 (en) * 1999-10-20 2001-04-26 Zymogenetics, Inc. Proteins and polynucleotides encoding them
FR2827866B1 (fr) * 2001-07-27 2004-12-10 Pasteur Institut Peptides synthetiques ou naturels liant la proteine phosphatase 2a, methode d'identification et utilisations
EP1506004A2 (en) 2002-05-15 2005-02-16 Max-Planck-Gesellschaft zur Förderung der Wissenschaften e.V. Means for use in treating diseases correlated with or caused by non-physiological levels of microtubule-associated pp2ac

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