RU2149625C1 - Method and device for correcting cardiac arrhythmia - Google Patents

Abstract

FIELD: medicine. SUBSTANCE: method involves evaluating organism state in dynamic mode using Akabane test. Channels of outmost influence upon heart beat rate are determined by means of regression analysis data. Their inlet/outlet points are stimulated until the number of heart contractions comes to norm. Stimulation is carried out with modulated infrared bandwidth radiation of 780-2500 nm in the mode of operation composed of 15-30 s action and 60-120 s pause. The device has casing having pulse gage unit, unit for recording information, control unit, switching unit, infrared radiation generator, infrared radiation emitter mounted therein. The pulse gage unit as photodetector and the infrared radiation emitter are mounted above the entry/exit point of the corresponding channel. Photodetector outlet is connected to control unit inlet through the unit for recording information. Control unit outlet and infrared radiation generator outlet are connected to the infrared radiation emitter through the switching unit. EFFECT: enhanced effectiveness of treatment. 2 cl, 1 dwg, 1 tbl

Description

 The present invention relates to medicine, in particular to reflexology, by means of which a number of cardiac arrhythmias that are not associated with gross organic changes are corrected, and is a method of correcting cardiac arrhythmias and a device for its implementation.

 Known methods for the reflex treatment of cardiac arrhythmias, in particular paroxysmal tachycardia, in which at the time of attacks they press on the eyeballs or moisten the face with cold water / N.K. Furkalo, "Clinical lectures on the most important internal diseases," Kiev, 1975, p. 184 /. However, this method of reflex exposure is effective only with a narrow circle of arrhythmias and not for all patients.

 It is known the treatment of heart disease using reflexology. So, with angina pectoris in the interictal period without severe organic heart damage, massage of the projection sites of the meridians of the heart and pericardium on the left hand is recommended. In this case, a significant effect is achieved by using massage and acupuncture at the points of ge-guan and yang-gan. / G.Luvsan, "Essays on the methods of oriental reflexology." Novosibirsk, Science, Sib. Dep. 1991, pp. 360-362 /.

 There is also a method of correcting cardiac arrhythmias (RF Application N 92016109/14 A CL A 61 H 39/06, 1995, "Inventions", N 21), in which auricular points are affected by electromagnetic wavelength-modulated electromagnetic pulses of infrared radiation with a wavelength 780-2500 nm with a constant frequency of exposure until the rhythm normalizes, with tachycardia they perform a stimulating effect on points associated with the kidney and bladder channels, and inhibitory effects on points associated with the heart channel, and with bradycardia stimulating type on the points associated with the channel of the heart, and the impact of the inhibitory type on the points associated with the channels of the kidney and bladder.

 The method is intended only for the correction of pathology associated with rhythm disturbance in the type of tachy- and bradycardia and not associated with pronounced organic changes in the body.

 To implement the method, a device was used in the form of a clip on the ear, the block diagram of which includes pulse sensor, control, switching blocks, two generators for fixed frequencies, a timer and two IR emitters installed opposite the selected auricular points. The heart rate sensor unit is made in the form of a pair of a photosensor-photodetector of a pulse blood supply mounted on the auricle for recording the number of heartbeats (HR) connected to the inputs of the control unit, pulse generators and a timer, and the output of the switching unit is connected to IR emitters. The device implements the “on demand” operating mode and can be programmed for certain operating modes. The device works on the principle of feedback with a heart rate sensor for pulse blood supply to the earlobe and IR emitters installed opposite the auricular points.

 The objective of the invention is to develop a method that allows non-invasively using reflexology to correct cardiac arrhythmias such as tachy and bradycardia, not associated with pronounced organic changes in the heart / for example, WPW syndrome, sinus node weakness syndrome / SSS / etc /.

 This goal is achieved by the fact that the proposed method for the correction of cardiac arrhythmias, not associated with organic changes, in which the acupuncture points are affected by electromagnetic, modulated rectangular pulses, infrared radiation with a wavelength of 780 - 2500 nm at a power density of 100-150 mW / cm with a constant frequency of 15-30 Hz and a duty cycle of 0.5-0.7. At the same time, in the dynamics of observation at different values of the number of heart contractions / heart rate /, the Akabane test is carried out, according to the regression analysis, the channel / channels / with the greatest influence on the normalization of the number of heart contractions is determined and the points on its / their / entry-exit are normalized to normalization Heart rate in the 15-30 s exposure mode and 60-120 s break.

 The entry-exit points of the energy channels are not chosen randomly. It is known that the more distal the point is located on which the method of reflexology is applied, the more pronounced is the effect of the procedure. In this regard, the entry-exit point from the channel has the maximum therapeutic effect, however, these points are not used in traditional reflexology, since the introduction of a needle into the area of the nail root is extremely painful.

 In the proposed method, the impact on the entry-exit points of the energy channels is carried out by a non-invasive painless method, using infrared electromagnetic radiation modulated by rectangular pulses with a wavelength of 780-2500 nm / IR radiation /. Such an effect gives a pronounced effect that exceeds the effect of introducing a needle.

 The channel selection strategy and the principle of influence on it are decided taking into account its contribution to the overall heart rate regulation model, as well as the specific value of the channel energy at the time of exposure. Specific channels for exposure through entry and exit points are selected individually based on the test results, for example, according to the Akabane / cm test. F.G. Portnov, "Electropuncture reflexotherapy", Riga, "Zinatra" 1983, p. 103./. Its essence lies in the fact that certain points of each channel are affected by a point heat source, which sends heat pulses, for example, in time with the number of heartbeats. The number of such thermal pulses from the beginning of exposure to biologically active points / BAP / to the occurrence of the first pain sensations depends on the thresholds of BAP perception of this channel and is representative of the energy and functional state of each channel. In the proposed method, the Akabane test is carried out using infrared radiation to the beat with pulse circulation with a frequency of 1-2 Hz.

 In patients with cardiac arrhythmias, dynamic testing is performed with simultaneous registration of heart rate. In the presence of a dynamic series of synchronous changes in heart rate indicators and the Akabane test by regression analysis with the construction of a regulation model in the form of a formula, channels / channel / are identified that have the largest specific contribution to individual heart rate regulation with different signs / contributing to an increase or decrease in heart rate /. Then, those channels that, according to the regulation formula, have a normalizing effect on heart rate, are exposed to infrared radiation through their entry-exit points until the heart rate normalizes. The methodology for constructing the formula for the heart rate regulation model is described in detail in the example.

 The Akabane test and the correction of cardiac arrhythmias by affecting the calculated points with modulated infrared radiation can be performed using the Reflexomaster device specially developed for this purpose.

 The proposed method for the correction of cardiac arrhythmias can also be implemented using a device containing a housing, inside which there is an element for influencing the BAP in the form of an IR emitter, a physiological parameter sensor, a control unit, an information recording unit, an IR pulse generator and a switching unit. The physiological parameter sensor for detecting heart rate and the BAT exposure element are made in the form of a pair of an infrared emitter-photodetector of a pulse blood supply connected through an information recording unit to the input of a control unit, the output of which and the output of an infrared pulse generator through a switching unit are connected to an infrared emitter.

 The device can be made in the form of a ring or thimble, worn on the end phalanx of the finger where the entry-exit point of the channel is located, which, when exposed to it, normalizes the heart rate according to the formula obtained, while the IR emitter and photodetector are placed in the case so in order to respond to changes in heart rate and to influence the entry-exit points of the energy channel.

 The device operates in automatic mode by the principle of feedback, performing the function "on demand" (demand) and can be reprogrammed into different operating modes. In contrast to the prototype, the proposed device used one generator and one control unit. For correction, only one type of effect is applied - exciting.

 So, in case of tachycardia, if a certain programmed threshold of heart rate is exceeded / for example, 90 beats / min /, it produces an effect associated with a decrease in heart rate, while the IR emitter is installed above the entry-exit point of that channel, the stimulation of which according to an individual model has a lowering heart rate act.

 In bradycardia, it is reprogrammed to the lower threshold of operation, set above the entry-exit point of the channel, stimulation of which leads to an increase in heart rate, and is included in the exposure mode, for example, when the heart rate is below 60 beats / min.

 The effect on the entry and exit points in both cases can be of the same type, by exposure to modulated infrared radiation at a fixed frequency and power. The main factor in the impact is the correct choice of channel with the maximum normalizing regulatory influence.

 In the proposed figure / Fig. 1 / shows a schematic block diagram of the proposed device. The device comprises a housing / 1 /, a physiological parameter sensor / 2 /, an information recording unit / 3 /, a control unit / 4 /, a switching unit / 5 /, a pulse generator / 6 /, an element for influencing the BAT / 7 /. The latter in the form of an infrared emitter is introduced into the physiological parameter sensor. The housing can be made in the form of a thimble or ring worn on a finger. The physiological parameter sensor / 2 / is made in the form of a pulse blood supply IR-emitter-photodetector pair installed above the entry-exit point from the corresponding energy channel, the IR emitter simultaneously acts as an element of influence on the BAT.

 The device operates as follows. The entire phalanx of the finger, where there is an entry / exit point to the corresponding energy channel, is superimposed in the form of, for example, a thimble in which all the blocks are placed. The heart rate sensor unit in the form of an optocoupler IR emitter-photodetector is located directly above the input-output point of the channel that is being affected, while the IR emitter illuminates the photodiode in the observation mode. During pulse blood supply at this point, its optical transparency changes and, therefore, the current passing through the photodiode changes. Information about this goes to the information recording unit / 3 /, containing a comparator or Schmitt trigger, which generates strobe pulses corresponding to heart rate, arriving at the control unit / 4 /, where the signals are analyzed in frequency and only if they deviate from the specified normal limit , the control unit / 4 / turns on the device in the active exposure mode. The control unit / 4 / as a whole is a programmable threshold device that monitors heart rate changes. It can be performed in several variants, for example, as a threshold comparator or microprocessor device, for example, on a Z-80 or 81S51JB processor or their analogs. The control unit / 4 / implements signal tracking in a certain frequency range, while the specific mode of operation and the response frequency of the device are recorded. At a certain point, when the heart rate goes beyond the set limits, the control unit switches from the monitoring mode to the exposure mode with the inclusion of an infrared emitter / 7 / to the generator load / 6 /.

 So, for example, in a patient with tachycardia, the threshold frequency of the device’s response at a heart rate of 90 beats / min can be set. If it is exceeded, the command receives a command by which the transition from the monitoring mode to the active exposure mode occurs. In this case, the pulse generator / 6 /, operating at a frequency of 15-30 Hz, is connected via the switch / 5 / to the IR emitter / 7 / and for a certain time, which can be programmed in the control unit / 4 / individually, for example 15 -30 s, the effect on this BAP. During a break after exposure, the heart rate sensor unit is reconnected and, if the heart rate is not normal, the control unit / 4 / authorizes a second action, which can be repeated until the heart rate normalizes, after which the device again operates in the monitoring mode.

 In bradycardia syndrome, the lower limit of heart rate is similarly monitored. The device may have micro-connectors for reprogramming it into other modes.

 To track along the upper and lower boundaries of the standard corridor, two or more devices can be used, each of which is located on different channels, depending on their sign and level of participation in heart rate regulation and controls its portion of the range.

 The following example illustrates the invention.

Example 1. Patient Mes ... n M.E., 63 years old. Diagnosis: ischemic heart disease. Myocarditis cardiosclerosis. Sick sinus syndrome, tachy - brady form. Complaints about the presence of periods of a rare number of heart contractions from 40 to 50 beats / min, which are sometimes replaced by attacks of tachycardia with a heart rate of more than 100 beats / min. The usual daily pulse is 50 - 65 beats / min. EFI data from 6.10.94 g: sinus rhythm with a heart rate of 50 beats / min. VVFSU - 1550 ms. KVVFSU - 350 ms. Wenckebach point - 130 beats / min. Filed Holter monitoring - migration of the pacemaker from the sinus node to the atria and AB connection with periods of bradycardia up to 38 beats / min. Short paroxysms of atrial tachycardia with a heart rate of 115 beats / min. ECHO-KG: PL - 3b8 cm, Ec - 38%, Ep - 69%. The patient underwent 15 tests in the dynamics of observations with the simultaneous registration of heart rate during testing. The data of the regression analysis with the construction of the model are presented in table. 1
The model has a 72% prediction reliability coefficient (Rsq = 0.72), and both components have a significant (t> 4.0) effect on heart rate from the gallbladder channels on the left (VBs) and the triple heater on the right (TRd), but with different signs. Their graphs of regulatory influences are presented in FIG. 2 and 3, respectively, and the total ratio of observed and predicted values according to this formula is shown in FIG. 4. Thus, the general formula for channel regulation of heart rate in this patient has the form:
Heart rate = 81 + 12VBs - 35TRd
In this model, an increase in the values of the scaled indicators is greater than 1.0 (obtained by dividing the absolute values of each channel by the arithmetic mean of the entire test) of the gallbladder (VB) channel when it is hypofunctional, leads to an increase in heart rate with an influence coefficient of 12. Similarly, the translation of the indicators of this channel values lower than 1.0 are accompanied by a decrease in heart rate in proportion to the degree of hyperfunction of a given channel and its influence coefficient.

 The most powerful effects on heart rate in this model come from the triple heater channel (TR), since its influence coefficient on heart rate is 34. In the case of certain effects on this channel, translating it into a state of hyperfunction with values of scaled indicators less than 1.0, there will be an increase in heart rate is noted, and with its hypofunction, on the contrary, a marked decrease in heart rate. Thus, these channels are a kind of leverage on heart rate in a wide range of its values.

During one of the observations, the heart rate reached 100 beats / min. In this case, the energy state of the channels was estimated using the Akabane test on a Reflexomaster-007 device, performed using an IR emitter operating in a pulsed mode synchronously with the patient’s heart rate, counting the number of pulses for each channel until a temperature threshold occurs pain In this case, the following Akabane test took place:
P; GI; MC; TR; C; IG; RP; F; E; VB; R; V - channels
6; eleven; thirteen; 5; eight; eleven; nine; 12; ten; 18; 32; 36 - right
ten; ten; thirteen; ten; 7; fourteen; thirteen; eleven; eleven; 35; thirteen; 25 - left
Since the general threshold of pain sensitivity during different observations can vary, to compare different observations, it becomes necessary to bring their data to a single denominator, which is done by scaling the indicators by dividing them by the arithmetic mean of each test.

 The total value of 24 channels amounted to 343, the arithmetic average value (343: 24) of the indicator was 14.2. In this case, the scaled value of TRd (5: 14.2) was 0.35. On the channel VBs it amounted to 2.4.

 Thus, the estimated heart rate = 81 + 12 • 2.4 - 35 • 0.35 = 98 beats / min, which corresponded to the existing clinical picture.

 To stop the attack of tachycardia, the method of stimulating type influence on the entry-exit point from the gallbladder channel on the left (i.e. zu-tseyao-yin) was used. The exposure was carried out by modulated rectangular pulses of infrared radiation with a wavelength of 780-2500 nm with a power density of 150 mW / cm with a constant frequency of 15 Hz and a duty cycle of 0.5-0.7 in the mode of 15 s exposure and 120 s break.

 After a double exposure of 15 s with intervals between exposures of 60 s, there was a decrease in the number of heart contractions to 75 beats / min due to an increase in the activity energy of the gallbladder channel from the left from 2.4 to 1.42, while a decrease in the energy of the channel of the triple heater from 0 , 35 to 0.65.

 In this case, the estimated heart rate = 81 + 12 • 1.42 - 35 • 0.65 = 75 beats / min.

In another of the observations for this patient, there was an attack of bradycardia with a heart rate of 50 beats / min. At the same time, the Akabane test looked like:
Before exposure
P; GI; MC; TR; C; IG; RP; F; E; VB; R; V channels
eight; 5; 7; nine; 6; 5; 7; 5; 7; thirteen; 17; 16 - right
6; 7; 5; 4; 3; 6; eight; nine; ten; 6; 24; 15 - on the left
After exposure
7; 7; 6; 6; 5; 5; 7; 6; eight; eight; eight; 11 - right
7; ten; eight; 7; 4; 6; 6; eight; thirteen; ten; nine; 18 - left
The total sum of the test values before exposure was 198, the arithmetic average of 8.25. Moreover, the calculated value of heart rate = 81 + 12 • 0.72 - 35 • 1.09 = 52 beats / min.

 To increase the heart rate, the most rational taking into account the obtained formula is to transfer TRd to a hyperfunction state, which was carried out by modulating IR radiation at the input-output point of this channel (i.e., guan-chun) according to the claimed method at a frequency of 28 Hz in a mode of 30 s and 120 with a break. After such a triple exposure, after 6 min, the heart rate increased to 68 beats / min, and according to the results of control testing, the energy of the TRd channel increased from 9 to 6 conv / units according to the Akabane test, which confirms the exciting (stimulating) effect of the effect. The arithmetic mean value of the test was 7.9 conv / unit, and the calculated heart rate by the formula was 71 beats / min, (heart rate = 81 + 12.4 • 1.26 - 34.6 • 0.75 = 71 beats / min), which was not at variance with the clinic data at that time.

 These effects in this example were carried out using the device "Reflexomaster-007", which is specifically designed for such effects. However, they could be carried out using the claimed specialized device. Moreover, if the patient is dominated by bradyssystole, then to increase the heart rate, as shown in the example, the effect can be carried out from the entry-exit point of the triple heater channel (TR). For this, the claimed device, made, for example, in the form of a thimble, is put on the end phalanx of the fourth finger of the right hand, and its emitter is placed opposite the entry-exit point of this channel.

 When tachysystole predominates, this device is worn on the terminal phalanx of the fourth toe of the left foot, and the emitter is placed opposite the entry-exit point of the gallbladder channel (VB) located there.

 When tachy- and bradyssyndrome, it is advisable to use immediately two declared devices located on both channels participating in the obtained regulation formula. At the same time, one of the devices will track and level tachysystolic crises, and the other bradysystolic crises.

 In addition, using the claimed device, it is possible to cause a braking effect on the channels interested in the regulation of heart rate. For this purpose, the brake connections between the channels, which are known from the theory of the five primary elements and widely used in reflexology, are used. So, if we need to weaken the energy of the gallbladder channel (VB), we must stimulate the colon channel (GI). To weaken the energy (activity) of the triple heater (TR), we must stimulate the channel of the bladder V. (G. Luvsan "Essays on the methods of eastern reflexology", Novosibirsk, Nauka, 1991, pp. 12-14). The specific laterality of such an indirect effect, as well as the channel itself, which has a inhibitory inhibitory effect, taking into account the individual pathology presence factor, can be obtained from the inter-channel correlation effects matrices obtained from the results of preliminary dynamic testing in a particular patient.

 In the shown example, if it is necessary to inhibit the channels indicated in the formula, the claimed devices that perform stimulating effects will be located on the second finger of the hand (there is the entry / exit point of the colon GI channel) and on the fifth toe, where the entry / exit point is from the channel of the bladder (V).

 Thus, the claimed device, operating on the principles described above, can provide both excitatory and inhibitory effects on any channels involved in the regulation of various physiological parameters, and can have more universal, widespread use in all cases where there are indications for reflexology.

Claims (2)

1. A method for correcting cardiac arrhythmias not associated with organic changes in the body, including assessing the level of energy status of the channels with the Akabane test and subsequent exposure to BAT with electromagnetic modulated rectangular pulses of infrared radiation with a wavelength of 780 - 2500 nm (modulated IR radiation) at a power density emission 100 - 150 mW / cm ² at a constant frequency of 15 - 30 Hz, characterized in that the test is carried out in Akabane observing the dynamics for various values of heart abbr scheny, according to the mathematical (regression) analysis determined the channels with the greatest influence on the regulation of heart rate and conduct affect its point (s) entry and exit to the normalization of cardiac contractions in the mode 15 - 30 with exposure and 60 - 120 with a break.  2. A device for reflexology containing an influence element on the BAP in the form of an IR emitter, a physiological parameter sensor, a pulse generator and a control unit connected via a switch to an influence element, characterized in that an information recording unit connected to the control unit is inserted into the device impact element, while the impact element is inserted into the physiological parameter sensor, the body is a thimble or ring, worn on the terminal phalanx of the finger, and an infrared emitter and photodetectors to placed in the housing so as to respond to changes in heart rate and to influence the input-output points of the energy channel.

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