MX2013009455A - Use of sdhi fungicides on conventionally bred asr-tolerant, stem canker resistant and/or frog-eye leaf spot resistant soybean varieties. - Google Patents

Abstract

The present invention relates to a method for controlling Asian soybean rust (ASR) of a conventionally bred ASR-tolerant, Stem canker resistant and/or Frog-eye leaf spot resistant soybean variety comprising the application of a succinate dehydrogenase inhibitor (SDHI) fungicide to said plant, plant propagation material, or at its locus of growth.

Description

FIELD OF THE INVENTION The present invention relates to a process for combating Asian soybean rust (ASR) from a variety of conventional selective culture soybeans, i; ASR tolerant, resistant to stem cancrosis and / or resistant to "frog eye" leaf spot, comprising the application of a fungicide inhibitor of succinate dehydrogenase (SDHI) to said plant, plant propagation material ', or in its growth locus. 1 BACKGROUND OF THE INVENTION Soybean (genus Glycine) is considered an important crop and is highly valued by world agriculture. Therefore, one of the main objectives of soybean breeders is to develop varieties that are more resistant to diseases, productive and stable. The basic motivation is to maximize the yield of grains for animal and human consumption. In order to achieve these objectives, the breeder usually selects varieties that have superior traits. i Asian soybean rust (ASR), caused by the fungus Phakopsora pachyrhizi, is considered the most destructive soybean disease, (Iviilés, MR, Frederick RD, Hartman, G. (2003) Soybean rust: Is the US soybeart drop at risk? Online, APSnet Feature, American Phytopathological Society). The disease is spread by uredoespores carried by the wind, which consequently allow long distance dispersion to new rust-free regions. As a result, ASR has already caused losses in many soybean growing regions of the world. The impact of the pathogen on productivity is drastic: up to 80% of yield loss was observed in some regions (Yorinori JT (2004) Ferrugem "Asian" soybean not Brazil: evolution, economic importance and control In: Junior JN Lazzarotto JJ (eds.) Documents 247. Embrapa, Londrina, Brazil, 36). : In order to combat the disease, fungicides are applied or resistant or tolerant varieties are used.

The application of fungicides commonly entails the problem of unfavorable toxicological or environmental effects due to the high dosage rates needed to effectively combat the disease.

It is known that resistance to the fungus occurs naturally in genotypes of the genus Glycine (Burdon, JJ; Marshall, DR (1981) Evaluation of Australia's native species of Glycine canescens, a wild relative of soybean, Theoretical Applied Genetics, 65: 44-45. Burdon, JJ (1988) Major gene resistance to Phakopsora pachyrhizi in Glycine canescens, a wild relative of soybean, Theoretical Applied Genetics, 75: 923-928). Five qualitative dominant resistance genes have been identified (Meyer JDF, et al., (2009) Identification and analyzes of candid genes for rpp4-mediated resistance to ASR in soybean, Plant Physiology, 150: 295-307; Oloka HK; Tukamuhabwa P (2008) Reaction of exotic soybean germplasm to Phakopsora pachyrhizi in Uganda, Plant Disdase, 92: 1493-1496): rppl in 1200492 (McLean, RJ; Byth, DE (1980) Inheritance of resistance to rust (Phakopsora pachyrhizi) in soybean Australian Journal Agricultural research, 31: 951-956); rpp2 in ?? 230970 (Bromfield, K. R .; Hartwig E. E. (1980) Resistance to soybean rust and mode of inheritance, Crop Science, 20: 254-255); rpp3 in ?? 462312 (Bromfield K. R .; Melching, J. S. (1982) Sources of specific resistance to soybean rust, Phytopathology, 72: 706); rpp4 in ?? 459025 (Hartwig RR (1986) Identification of a fourth major gene conferring to resistance to soybean rust Crop Science, 2 (3: 1135-1136) and rpp5 (eyer JDF (2009) Identification and analyzes of candidate genes for Rpp4-mediated resistance to Asian soybean rust in soybean, Plant Physiol 150: 295-307) The resistance presented by each gene is limited to the specific pathogen variety d and can be overcome in a short period of time due to the coevolution of pathogenic virulence and resistance of the host Although there are varieties of soybean that have superior traits, for example, tolerance to ASR, they are not completely resistant but are still only tolerant to said disease.

Therefore, tolerance to cultivation and activity against fungi nb always satisfies the needs of agricultural practice in many events and aspects. HE investigates the use of new sources of resistance with the focus on both: application of fungicide more effective and identification and / or modification of genes that confer tolerance / resistance to ASR.

WO 2010/049405 teaches a method for improving the health of a plant by applying a combination containing an alanine compound substituted with heteroaryl and an agriculturally active compound such as for example fungicides fluxapiroxad, bixafenoj, boscalid, sopirazam or pentiopirad. In the same, the improvement of the health of the ho plant indicates a teaching for the combat of the ASR.

Furthermore, previously US 2010/0093715 has reported that the application of a carboxamide fungicide to a 0a transgenic plant results in a synergistic increase in the health of the plant. Although! He mentions a procedure to combat ASR, teaching is limited to plants with transgenic modifications. No transgenic plants tolerant to ASR are described.

WO 2010/046380 describes a method for controlling pests and / or increasing the health of a plant by treating a plant grown with a carboxamide compound. The soybean varieties that are referred to in the description or examples of this reference are I transgenic plants. The variety of soybean used in the biological example of WO 2010/046380 contains a transgene conferring tolerance to herbicides. Do not i! there is indication in document WO 2010/046380 that the application of unifinuclide inhibitor of succinate dehydrogenase (SDHI) to a variety of soya. Conventional selective culture, tolerant to ASR, resistant to canker of the stem and / or resistant to the leaf spot "frog's eye", would result in a synergic effect between the trait of said plant and the applied fungicide. Therefore, no sufficient teaching is given regarding the control of ASR in conventional selective soybean varieties, tolerant to ASR, resistant to stem cancrosis and / or resistant to the leaf spot "frog's eye". :: The general advantages of conventionally grown plants vs. Transgenic plants, include the following: j: In many cases, traits introduced by conventional cultivation are more stable than traits introduced by transgenic procedures. For conventional selective culture plants, there is less risk of environmental contamination, for example, by horizontal gene transfer. In particular, for conventional selective culture plants there is no risk that could be conferred by an antibiotic resistance gene1 which is usually introduced into transgenic plants for the purpose of selection. Also, there is a strong economic advantage generated, since there is no need for costly and somewhat long deregulation procedures; in each country for conventional selective cultivation plants. Also, the period of time and costs necessary to develop a conventionally cultivated variety are much smaller than for transgenic varieties. In addition, in some countries the acceptance by the consumer is generally greater. Furthermore, for plants of conventional selective cultivation there is no risk that the beneficial plant properties will be disturbed or even eliminated by the introduction of the transgene that is randomly produced in the genome.

DESCRIPTION OF THE INVENTION Accordingly, it is an object of the present invention to provide a method, which allows the combat of ASR in a culture ' conventional selective, tolerant to ASR, resistant to stem and / or resistant to leaf spot "frog's eye" by using a fungicide.

Furthermore, it is an object of the present invention to provide a method, which allows the combat of ASR in a variety of conventional selective culture, ASR-tolerant, canker-resistant and / or leaf-spot resistant. frog's eye "by using a fungicide, in which there is a synergistic effect of the traits of the plant and the fungicide.

Another objective of the present invention is to provide a procedure that allows the combat of ASR in which the superior traits of uni pjárita conventional selective culture, tolerant to ASR, resistant to cancrosis destallo and / or I! 'J resistant to leaf spot "frog's eye", lead to a synergic effect pontra ASR with the application of a fungicide.

Another objective of the present invention is to provide a method, which allows the combat of ASR in a variety of soybean of conventional selective culture, tolerant to ASR, resistant to canker of the stem and / or resistant to the leaf spot "eye of frog "by using a fungicide, in which there is a synergistic effect of the plant and fungicide traits, and in which there are additional advantages in the use of a conventionally cultivated plant compared to a selected transgenic plant 1 of the group constituted by greater stability of the new / s trait / s, lower 'risk of environmental contamination, no risk of contamination by a gene with! resistance to antibiotics, economic advantage, greater acceptance by the consumer, and no risk that the beneficial plant properties are disturbed or even eliminated by the introduction of the transgene that is randomly produced.

Surprisingly, it has been discovered that the use of an SDHI fungicide for the treatment of ASR in a variety of soybean of selective cultivation conventional, tolerant to ASR, resistant to stem cancrosis and / or resistant to leaf spot. frog "shows a synergistic effect between the trait of said plant and the applied fungicide.

Synergistic in the present context means that the use of the fungicide SDHI in combination with the variety of soybean of conventional selective culture, tolerant to ASR, resistant to canker of the stem and / or resistant to the stain of the "frog's eye" exceeds the cumulative effect, which is expected in Phakopsora pachyrñizi and thereby extends the range of action of fungicide SDHI and active pririclpio expressed by the variety of soybean conventional selective culture, tolerañté to ASR, resistant to stem cancrosis and / or resistant to the leaf spot "ojo de frog".

This surprising discovery involves an inventive step: from the prior art. For example, according to what is detailed above, the difference with WO 2010/046380 is not only that the biological specimen in soybean in WO 2010/046380 (page 139-140) corresponds to a variety: transgenic soya compared to a conventional selective crop variety, but also that the transgene of the soybean variety used in WO 2010/046380 confers tolerance to herbicides (glyphosate), while the altered property of the conventionally cultivated variety claimed in the present invention is linked to tolerance to ASR, resistance to the cancrosis of the stem and / or resistance to the leaf spot "frog's eye". It could not be expected that the treatment with the fungicide / s according to the invention would result in a synergistic effect with respect to the combat of the ASR between the trait of said plategta and the fungicide applied.

Especially, it has been found that the application of at least one SDHI fungicide to a conventional, tolerant selective soybean variety; ASR, resistant to stem cancrosis and / or resistant to leaf spot "eye of frog" leads to a synergistically enhanced action against Pha opsora pachyrhizi compared to the eradication rates that are possible with the same fungicide SDHI in natural-type plants when applied to a variety of soybean 1 | of conventional selective culture, tolerant to ASR, resistant to canker: stem and / or resistant to leaf spot "frog's eye", parts of that plant , material of propagation of the plant or in its locus of growth.

Accordingly, the present invention relates to a method for combating ASR from a variety of conventional selective soybean, ASR-tolerant, resistant to stem canker and / or stain resistant to "frog's eye". it comprises the application of an SDHI fungicide to said plant, propagation material of the plant, or at its growth locus. ! ''. ' According to a preferred embodiment of the present invention, the SDHI fungicide is applied at least in parts of the variety of soya "conventional selective culture, tolerant to ASR, resistant to stem canker and / or stain resistant. "frog eye" leaf, preferably leaves, roots, flowers and / or the stem of the conventional selective crop plant, and its seed.

SDHI Fungicides In a preferred embodiment of the present invention it has been found that the application of at least one SDHI fungicide selected from the group consisting of penflufen, isopyrazam, bixafen, sedaxane, fluxapiroxad, fluopyram, pentiopyrad, boscalid, N- [1- (2,4-dichlorophenol) -1-methoxypropan-2-yl] -3- (difluoromethyl) -1-rnetyl-1 H-pyrazole-4-carboxamide, N - [9- (dichloromethylene) -1, 2,3,4-tetrahydro-1,4-methanonaphthalen-5-yl] -3- (d-fluoro-methyl) -1-methyl-1 H-prazol-4-carboxamide , N - [(1S, 4R) -9- (Dichloromethylene) -1, 2,3,4-tetrahydro-1,4-methanonaphthalen-5-yl] -3- (d-fluoro-methyl) -1- methyl-1 H-pi carboxamide, and N- ^ IR ^ SJ-g-dichloromethylene-I ^. S ^ -tetrahydro-l ^ -metanqnaphthalen-5-yl] -3- (difluoromethyl) -1-methyl-1 H-pyrazole-4- carboxamide to a soybean variety of conventional selective culture, tolerant to ASR, resistant to stem cancrosis and / or 1 resistant to leaf spot "frog's eye" leads to a synergistically enhanced action against Phakopsora pachyrhizi compared to eradication rates that are possible with the same fungicide SDHI preferably chosen from the group consisting of the compounds as defined above in wild-type plants when applied to a variety of conventional selective culture soybeans, plant parts, propagation of the plant or its locus of growth.

According to another preferred embodiment of the present invention, at least one SDHI fungicide is selected from the group consisting of pehflifen, isopyrazam, bixafen, sedaxane, and fluxapiroxad.

Penflufen (chemical name: N- [2- (1,3-dimethylbutyl) phenyl] -5-fluoro-1,3-dimethyl-1H-pyrazole-4-carboxamide) and the processes for its production based on the commercially available compounds can be found in WO 03/010149. j.

Isopyrazam is a mixture comprising both isomers without 3- (difluoromethyl) -l-methyl-N - [(1 RS, 4SR, 9RS) -1, 2,3,4-tetrahydro-9-isopropy | -1, 4-methanonaphthalen-5-yl] pyrazole-4-carboxamide and both anti isomers of 3- (difluoromethyl) -1-methyl-N - [(1 RS, 4SR, 9SR) -1, 2,3,4-tetrahydro- 9-isopropyl-1,4-methanonaphthalene-5r L] pyrazole-4-carboxamide. Isopirazam further comprises isopyrazam (anti-epimeric 1-RS, 4SR, 9SR), isopyrazam (anti-epimeric enantiomer (R) 4S, 9S), isopyrazam (anti-epimeric enantiomer 1S, 4R, 9R), isopyrazam (Rifamate without epimeric 1 RS, 4SR, 9RS), isopyrazam (rinantiomers without epimeric 1R, 4S, 9R), and ,; isopyrazam (enantiomer without epimeric 1S, 4R, 9S). Isopyrazam: and the Methods for their production based on commercially available compounds are provided in WO 2004/035589. 1 Bixafen (chemical name: N- (3 ', 4'-dichloro-5-fluoro-1, 1' -biphenyl-2-yl) -3- (difluoromethyl) -1-methyl-1 H-pyrazole-4- carboxamide) and processes for their production based on commercially available compounds can be found in WO 03/070705.

Sedaxane is a mixture comprising both cis isomers of 2 '- [(1 RS, 2RS) -1' -bibcloprop ^ -ylJ-S-idifluoromethyl-1-methylpyrazole ^ -carboxanilide, and both trans isomers of 2 '- [ (1RS, 2SR) -1, 1'-bicycloprop-2-yl] -3- (diflupromethyl) -1-methylpyrazole-4-carboxanilide. Sedaxane and methods for its production on the basis of commercially available compounds can be found in WO 03/074491, WO 2006/015865 and WO 2006/015866. ';,.

Fluxapiroxad (chemical name: 3- (difluoromethyl) -1-methyl-N- (3 ', 4', 5'-trifluorobiphenyl-2-yl) -1H-pyrazole-4-carboxamide) and the procedures for their production on the basis of commercially available compounds' can be found in WO 2005/123690. ! " Fluopyram (chemical designation: N- { [3-chloro-5- (trifluoromethyl) -2-pyridinyl] ethyl} -2,6-dichlorobenzamide and processes for its production on the basis of commercially available compounds can be found in EP-A-1 531 673 and WO 2004/016088 ..: jT Pentiopyrad (chemical designation: (RS) -N- [2- (1,3-dimethylbutyne-9-thienyl] -1-methyl-3- (trifluoromethyl) -pyrazol-4-carboxamide), the processes for its production based on Commercially available compounds can be found in EP 0737682.

Boscalid (chemical name: 2-chloro-N- (4'-chlorobiphenyl-2-yl) nicotinamide) and processes for its production based on commercially available compounds can be found in DE 19531813.

N- [1- (2,4-Dichlorophenyl) -1-methoxypropan-2-yl] -3- (difluoromethyl) r1-ri) etl-1-pyrazol-4-carboxamide is known from WO 2010 / 000612. .

N- [9- (Dichloromethylene) -1, 2,3,4-tetrahydro-1,4-methanonaphthalen-5-yl] -3- (difluoromethyl) -1-methyl-1 H -pyrazole-4-carboxamide, N - [(1S, 4R) -9- (dichloro ethylene) - 1, 2,3,4-tetrahydro-1,4-methanonaphthalen-5-yl] -3- (difluoromethyl) -1-methyl-1 H-pyrazole-4-carboxamide, and N - [(1 R, 4S) -9- (dichloromethylene) -1, 2,3,4-tetrahydro-1,4-methanonaphthalen-5-yl] -3- (d-fluoro-methyl) -1-methylene-1H-pyrazole-4 -carboxamida are known - by WO 2007/048556.

The SDHI fungicides mentioned above can be used alone or in combination with other active ingredients such as: (The active ingredients specified herein by their "common name" are known and described, for example, in the Pesticide Handbook ("The Pesticide Manual", 14th Edition, British Crop Protectibn Council 2006) or can search on the Internet (for example, http://www.alanwood.net/pesticides)).

Inhibitors of ergosterol biosynthesis, for example aldimorf, azaconazole, i'bitertanol, bromuconazole, cycloconazole, diclobutrazol, difenoconazole, diniconazole, diniconazole-M, dodemorf, dodemorf acetate, epoxiconazole, etaconazole, fenarimol, fenbuconazole, fenhexamid, fenpropidin, fenpropimorf , fluquinconazole, flurprimidol, flusilazole, flutriafol, furconazole, furconazole-cis, hexaconazole, imazalil, imazalil sulfate, imibenconazole, ipconazole, metconazole, myclobutanil, naftifine, nuarimol, oxpoconazole, paclobutrazol, pefurazoate, penconazole, piperaline, 1 prochloraz, propiconazole , protioconazole, piributicarb, pirifenox, quinconazole, simeeonazole, spiroxamine, tebuconazole, terbinafine, tetraconazole, triadimefonone, triaclimenol, tridemorph, triflumizole, triforine, triticonazole, uniconazole, uniconazole-p, vini-nazole, voriconazole, 1- (4-chlorophenyl) - 2- (1 H-1, 2,4-triazol-1-yl) cycloheptanol, 1- (2,2 ^ dimethyl-2,3-dihydro-1 H -inden-1-yl) -1 H-imidazole-5 -methylcarboxylate, N 5- (diflu ro'métil) -2-methyl-4- [3- (trimethylsilyl) propoxy] fen¡l} -N-ethyl-N-methylimidoformamide, N-ethyl-N-methyl-N '- ^ - methyl-5- (trifluoromethyl) -4- [3- (trimethylsilyl) propoxy] phenyl} imidoformamide and: 0- [1- (4-methoxyphenoxy) -3,3-dimethylbutan-2-yl] -1H-imidazole-1-carbothioate.

Inhibitors of the respiratory chain in complex I or II, for example carboxin, diflumetorim, fenfuram, flutolanil, furametpir, furmeciclox, m pronyl, oxycarboxin, trifluzamide, 1-methyl-N- [2- (1, 1, 2 , 2-tetrafluoroethoxy) phenyl] -3- (trifluoromethyl) -1H-pyrazole-4-carboxamide, 3- (difluoromethyl) -1-methylene-N- [2- (1, 1,2,2-tetrafluoroethoxy! ) phenyl] - 1 H-pyrazole-4-carboxamide, 3- (difluoromethyl) -N- [4-fluoro-2- (1, i, 2,3,3,3-hexafluoropropoxy) phenyl] -1-methyl-1Hp Razol-4-carboxamide, and salts of njiismos.

Inhibitors of the respiratory chain in complex III, for example ametoctradin, amisulbrom, azoxystrobin, ciazofamid, dimoxystrobin, enéstroburina, famoxadona, fenamidona, fluoxastrobina, kresoxim-methyl, metomínostrobina, orisastrobina, picoxystrobin, pyraclostrobina, pirametostrobina, pirapxistrobina, piribencarb, trifloxystrobin, (2E) -2- (2- { [6- (3-chloro -2-methylphenoxy) -5-fluoropyrimidin-4-yl] oxy}. Phenyl) -2- (methoxyimino) -N-methyletanamide, (2E) -2- (methoxyimino) -N-niethyl-2- (2- {([( { (1 E) -1- [3- (trifluoromethyl) phenyl] ethylidene}. Amino) oxy] methyl} phenyl) ethanamide,, (2E) -2- (methoxyimino) - N-methyl-2-. { 2 - [(E) - ( { 1 - [3- i (trifluoromethyl) phenyl] ethoxy} imino) methyl] phenyl} Ethanamide, (2E) -2-. { 2 - [( { [(1 E) - - (3. {[[(E) -1-fluoro-2-phenylethenyl] oxy} phenyl) ethylidene] amino.} Oxy) methyl] phen L} -2- (methoxyimino) -N-; methylentanamide, (2E) -2-. { 2 - [( { [(2E, 3E) -4- (2,6-dichlorophenyl) but-3-en-2-ylidene] amino} oxy) methyl] phenyl} -2- (methoxyimino) -N-methyleneteamide, 2-chloro-N- (1, 1, 3-trimethyl-2,3-dihydro-1 H -inden-4-yl) pyridine-3-carboxamide, 5-methoxy -2-methyl-4- (2-. {[[( { (1 E) -1- [3- (trifluoromethyl) phenyl] ethylidene}. Amino) oxy] methyl.}. Phenyl) - 2,4-dihi triazol-3-one, (2E) -2-. { 2 - [( { Cyclopropyl [(4-methoxyphenyl) imino] methyl]} sulfanyl) methy! Phenyl} Methyl-3-methoxyprop-2-enoate, N- (3-ethyl-3,5,5-trimethylcyclohexyl) -3- (forrnilanriino) -2-hydroxybenzamide, 2-. { 2 - [(2,5-dimethylphenoxy) methyl] phenyl} -2-methoxy-N-methylacetamide, (2R) -2-. { 2 - [(2,5-dimethyphenoxy) methyl] phenyl} -2-methoxy-N-methylacetamide and salts; '"' thereof; Inhibitors of mitosis and cell division, for example benomyl, carbendazim, chlorphenazole, dietofencarb, etaboxam, fluopicolid,: füb§rdadazole, pencycuron, thiabendazole, thiophanate-methyl, thiophanate, zoxamide, 5 ??? 6G? -7 - (4-methylpiperidin-1-yl) -6- (2,4,6-trifluorophenyl) [1,4] triazolo [1,5-a] pyrimidine, 3-chloro-5- (6-chloropyridin- 3-yl) -6-methyl-4- (2,4,6-trifluorophenyl) pyridazine and salts thereof; Compounds capable of having a multisite action, such as, for example, mixture of bordeaux, captafol, captan, chlorothalonil, copper hydroxide, copper rhealfate, copper oxide, copper oxychloride, copper sulfate (2+), diclofluanid, dithianon , dodin, free base dodin, ferbam, fluorofolpet, folpet, guazatine, guazatin acetate, iminoctadine, iminoctadine albesilate, iminoctadine triacetate, i mancobre, mancozeb, maneb, metiram, zinc metiram, oxina-copper, propamidine, propineb, sulfur and sulfur preparations including calcium polysulfide, thiram, tolylfluanid, zineb, ziram, 2,6-dimethyl-1H, 5H- [1, 4] dithino [2,3-c: 5,6-G '] dipyrrole-1, 3,5,7 (2H, 6H) -tetron and salts thereof.

Compounds capable of inducing host defense, such as, for example, acibenzolar-S-methyl, isothianyl, probenazole, thiadinyl and salts thereof.

Inhibitors of amino acid and / or protein biosynthesis, for example, andoprim, blasticidin-S, cyprodinil, kasugamycin, kasugamycin hydrate hydrochloride, mepanipyrim, pyrimethanil and salts thereof.

Inhibitors of ATP production, for example, fentin acetate, fentin chloride, fentin hydroxide and silthiofam.

Inhibitors of cell wall synthesis, eg bentiavalicarb, dimetomorf, flumorf, iprovalicarb, mandipropamid, polyoxins, polioxorim, validamycin A and valifenalate.

Inhibitors of membrane synthesis and lipids, for example biphenyl, chloroneb, dichloran, edifenfos, etridiazol, iodocarb, iprobenfos, isoprortholan, propamocarb, propamocarb hydrochloride, protiocarb, pyrazophos, quintozene, tecnazene and tolclofos-methyl.

Inhibitors of melanin biosynthesis, for example carpropamid, idiclocimet, fenoxanil, phthalide, pyroquilon and tricyclazole. 'J, Inhibitors of nucleic acid synthesis, for example benalaxyl, benalaxyl- (kiralaxyl), bupirimate, clozilacon, dimethirimol, etirimol, fiiiralaxyl, himexazole, metalaxyl, metalaxyl-M (mefenoxam), ofurace, oxdixil. and oxolinic acid.

Inhibitors of signal transduction, for example clozolinate, phenolonyl, fludioxonil, iprodione, procymidone, quinoxifene, and vinozozoline.

Compounds capable of acting as a decoupler, as for example binapacril, dinocap, ferimzone, fluazinam and meptildinocap.

Other compounds, such as, for example, benthiazole, betoxazine, capsymycin, carvone, cinometionat, clazafenone, cufraneb, ciflufenamide, cymoxanil, , .1. ciprosulfamide, dazomet, debacarb, dichlorophene, diclomezine, difenzoquat, diphenzoquat methylsulfate, diphenylamine, ecomato, fenpirazamina, flumetover, fluoroimida, flusulfamida, flutianilo, fosetilo-aluminio, fosetilo-calcio, fosetyl-sodium, hexachlorobenzene, irumamycin, metasulfocarb, methyl isothiocyanate, metrafenone, mildiomycin, natamycin, nickel dimethyldithiocarbamate, nitrotal-isopropyloxy octyl ammonia, oxamocarb, oxyfentin, pentachlorophenol and salts, phenothrin, phosphorous acid and its salts, propamocarb-fosetilate, propanosine -sodium, proquinazid, pyrrolnitrine, tebufloquin, tecloftalam, tolnifanide, triazoxide, triclamide, zarilamide, 1- (4- { 4 - [(5R) -5- (2,6-difluorophenyl) -4,5-dihydro- 1,2-oxazol-3-yl] -1,3-thiazol-2-yl}. Piperidin-1-yl) -2- [5-methyl-3- (trifluoromethyl) -1 H-pyrazole-1- il] ethanone, 1- (4- { 4 - [(5S) -5- (2,6-difluorophenyl) -4,5-dihydro-1,2-oxazol-3-yl] -1, 3- thiazol-2-yl.}. picperidin-1-yl) -2- [5-methyl-3- (trifluoromethyl) il] ethanone, 1- (4- { 4- [5- (2,6-difluorophenyl) -4,5-dihydro-1,2-oxazol-3-yl] -1,3-thiazole-2- il.}. piperidin-1-yl) -2- [5-methyl-3- (trifluoromethyl) -1 H -pyrazol-1-yl] ethanone, 1 H-imidazole-1-carboxylic acid 1- (4-methoxyphenoxy) ) -3,3-dimethylbutan-2-yl, 2,3,5,6-tetrac-oro-4- (methylsulfonyl) pyridine, 2,3-dibutyl-6-chlorothieno [2,3-d] pyrimidine -4 (3H) -one, 2; - [5-methyl-3- (trifluoromethyl) -1 H -pyrazol-1-yl] -1- (4-. {4 - [(5R) -5-phenyl) -4,5-dithy-1, 2-oxazol-3-ylH-thiazol-2-yl.}. Pyridin-1-yl) ethanone, 2- [5-methyl-3- (trifluoromethyl) ) -1 H-pyrazol-1i-yl] -1- (4-. {4 - [(5S) -5-phenyl-4,5-dihydro-1,2-oxazol-3-yl] -1. S-thiazole ^ -ylJpiperidin-li ethanone, 1 2- [5-methyl-3- (trifluoromethyl) -1 H -pyrazol-1 -yl] -1 -. {4- [4- (5-phenyl-4 , 5-dihydro-1,2-oxazo-K3-yl) -1, 3-thiazol-2-yl] piperidin-1-yl-ketanone, 2-butoxy-6-iodo-3-propyl-4H-chromen-4-one, '2-chloro-5- (4-chlorophenyl) -5- (2,6-difluorophenyl) -3,6-dimethylpyridazine, 5-amino-1, 3,4-thiadiazk > l72-thiol, 5-chloro-N'-phenyl-N '- (prop-2-yn-1-yl) thiophene-2-sulfonohydrazide, 5-methyl-6-octyl [1, 2,4] triazolo [1, 5 -a] pyrimidin-7-amine, (2Z) -3-amino-2-cyano-3-phenylpropane-2-ethyl enoate, N- (4-chlorobenzyl) -3- [3-methoxy-4- (prop -2-in-1-yloxy) phenyl] propanamidei N - [(4-chlorophenyl) (cyano) methyl] -3- [3-methoxy-4- (prop-2-yn-1-yloxy) phenyl] propanamide , :: N - [(5-bromo-3-chloropyridin-2-yl) methyl] -2,4-dichloropyridine-3-carboxamide, N- [1- (5-bromo-3-chloropyridin-2) -yl) ethyl] -2,4-dichloropyridine-3-carboxamide, N- [1- (5-bromo-3-chloropyridin-2-yl) ethyl] -2-fluoro-4-iodopyridine-3-carboxamide , N-. { (E) - [(cyclopropylmetho) i) i nno] [6- (d'-fluoro-methoxy) -2,3-difluorophenyl] methyl} -2-phenylacetamide, 1; N-. { (Z) - [(cyclopropylmethoxy) imino] [6- (difluoro (trifluoromethyl) -l H-pyrazol-1-yl] acetyl} piperidin-4-yl) -N - [(1 R) -1, 2,3,4-tetrahydrnaphthalen-1-yl] -1,3-thiazole-4-carboxamide, N-methyl-2- (1- { [5-Methyl-3- (trifluoromethyl) -lH-pyrazol-1-yl] acetyl}. Piperidin-4-yl) -N - [(1S) -1,2,3,4-tetrahydronaphthalen-1 -yl] -1, 3-thiazole-4-ca ^ . { 6 - [( { [(1-methyl-1 H-tetrazol-5-yl) (phenyl) metW of pentyl, phenazine-1-carboxylic acid, quinolin-8-ol and quinol-8-ol sulfate (2: 1).

Other compounds, such as, for example, l-methyl-S-trifluoromethyl-N- ^ '- (trifluoromethyl) b-phenyl-2-yl] -1H-pyrazole-4-carboxamide, N- ( 4-chlorobiphenyl-2-yl) -3- (d-fluoro-methyl) -1-methyl-1 H-prazol-4-carboxamide, N- (2 \ 4'-dichlorob-phenyl-2-yl); l) -3- (difluoromethyl) -1-methyl-1 H -pyrazole-4-carboxamide, 3- (difluoromethyl) -1-methyl-N- [4 '- (trifluoromethyl) biphenyl-2-yl) ] -1 H-pyrazole-4-carboxamide, N- (2 ', 5'-difluorobiphenyl-2-yl) -1-methyl-3- (trifluoromethyl) -1 H -pyrazole-4-carboxamide, 3- (difluoromethyl) -1-methyl- - [4 '- (prop-1-yn-1-yl) biphenyl-2-yl] -1 H -pyrazole-4-carboxamide, 5-fluoro-1, 3-dimethyl-N- [4 '- (prop-1-yn-1-yl) biphenyl-2-yl] -1 H -pyrazole-4-carboxamide, 2-chloro-N- [4' - (prop- 1-in-1-yl) biphenyl-2-yl] pyridine-3-carboxamide, 3- (difluoromethyl) -N- [4X3,3-d'metNbut-1-n-1-yl) biphenol -2-yl] -jl-methyl-1 H ^ pyrazole-4-carboxamide, N- [4 '- (3,3-dimethylbut-1-in-1-yl) b-phenyl-2-yl ] -5-fluoro-1, 3-dimethyl-1 H-p'irazol-4-carboxamide, 3- (difluoromethyl) -N- (4'-ethynyl-phenyl-2-yl) -1-methyl -1 H-pyrazole-4-carboxamide , N- (4'-ethynylbiphenyl-2-yl) -5-fluoro-1,3-dimethyl-1H-pyrrazol-4-carboxarinide, 2-. i;;; Chloro-N- (4'-ethynylbiphenyl-2-yl) pyridine-3-carboxamide, 2-chloro-N- [4 '- (3,3-dimethyl-1-y-1-yl) ) biphenyl-2-yl] pyridine-3-carboxamide, 4- (d-fluoro-methyl) -2-nitnet-N- [4 '- (trifluoromethyl) biphen-2-yl] -1, 3-Tazo-5-carboxamide, 5-fluoro-N- [4 '- (3-hydroxy-3-rjiethylbut-1-y-1-yl) biphenyl-2-yl] -1, 3-dimethyl-1 H-pyrazole-4-carboxamide, 2-chloro-N- [4 '- (3-hydroxy-3-) Acetylcholinesterase inhibitors (AChE), for example carbamates, for example Alanicarb, Aldicarb, Bendiocarb, Benfuracarb, Butoxicarboxim, Carbaryl, Carbofuran, Carbosulfan, Etiofencarb, Formetanate, Furathiocarb, Isoprocarb, Methiocarb, Methomyl, Metolcarb, Oxamyl, i I.

Pirimicarb, Propoxur, Thiodicarb, Tiofanox, Triazamate, Trimetacarb, XMC, and Xililcarb; or organophosphates, for example, acephate, azamethiphos, azinphos-ethyl, azinphos-methyl, cadusafos, chloretoxyphos, chlorfenvinphos, chloro, Chlorpyrifos, Clorpi ifos-methyl, Coumafos, Cyanophos, Demeton-S-methyl, Diazinon, Dichlorvos / DDVP, Dícrotofos, Dimetoato, Dimetilvinfos, Disulfoton, EPN, Etion, Etoprofos, Famfur, Fenamiphos, Fenitrotion, Fention, Fostiazato, Heptenofos , Imiciafos, Isofenfos, Is! Opropil O- (methoxyminothio-phosphoryl) salicylate, Isoxation, Malation, Mecarbam, Méthamidophos, Metidation, Mevinfos, Monocrotophos, Naled, Ometoate, Oxidemeton-methyl, Paration, Paration-methyl, Pentoate, Porato, Posalona , Fosmet, Fosfamidon, Foxino Pirimifos-methyl, Profenofos, Propetamfos, Protiofos, Piraclofos, Piridafention, Quinalfos, Sulfotep, Tebupirimfos, Temefos, Terbufos, Tetraclorvinfos, Tiometon, Triazofos, Triclorfon, and Vamidotion. ': GABA-dependent chloride channel antagonists, for example organochlorinated cyclodiene, for example Chlordane and Endosulfan; or phenylpyrazoles (fiproles), for example Etiprole and Fipronil.

Sodium channel modulators / sodium channel blockers I '. voltage-dependent, for example pyrethroids, for example Acrinatrin, Aletrin, d-cis-trans Aletrin, d-trans Aletrin, Bifentrin, Bioallethrin, S-cyclopentenyl isomer of Bioallethrin, Bioresmethrin, Cycloprothrin, Ciflutrin, beta-Cyfluthrin, Cih! albtrin , lambda-Cihalotrina, gamma-Cihalotrina, Cipermetrina, alfa-Cipermetfi av beta-Cypermetrina, teta-Cypermetrina, zeta-Cypermetrina, Cifenotrina [isomers (fi R) -trans], Deltametrina, Empentrina [isomers (EZ) - (1 R )), Esfenvalerate, Etofenprox, Fenpropatrine, Fenvalerate, Flucitrinate, Flumethrin, Tau-Fluvalinate, Halfenprox, Imiprotrin, Kadethrin, Permethrin, Phenothrin [isomer (1R) -trans), iPralethrin, i * Pyrethrin (pyrethrum), Resmethrin, Silafluofen, Tefluthrin, Tetramethrin, Tetramethrin [isomers (1 R))], Tralometrine, and Transfluthrin; or DDT; or Metoxychlor.

Nicotinic acetylcholine receptor agonists (nAChR), for example neonicotinoids, for example Acetamiprid, Clotianidin, Dinotefuran, Irhidacloprid, Nitenpyram, Thiacloprid, and Thiamethoxam; or Nicotine.

Allosteric activators of the nicotinic acetylcholine receptor (nAChR), for example spinosyns, for example Espinetoram and Espinosad.

Activators of the chloride channel, for example avermectins / milbemycins, for example Abamectin, Emamectin benzoate, Lepimectin, and Milbemectin.

Juvenile hormone mimetics, for example juvenile hormone analogs, for example Hydrorene, Chenoprene, and Metoprene; or Fenoxicarb; or Pyriproxyfen.

Various non-specific inhibitors (multisite), for example alkyl halides, for example methyl bromide and other alkyl halides; or Chloropycrine; or Sulfuryl fluoride; or Borax; or Tartar emetic.

Selective homopteran feeding blockers, eg Pimetrozine; or Flonicamid.

Mite growth inhibitors, for example Clofentezin, Hexithiazox, and Diflovidazine; o Etoxazole Microbial disruptors of insect midgut membranes, eg, Bacillus thurinogiensis subsp. Israelensis, Bacillus sphaericus,; Bacillus i thuríngiensis subspecie aizawai, Bacillus thuríngiensis subspecie kurstaki,; Bacillus thuríngiensis subspecies tenebrionis, and BT culture proteins: CryIAb,, Cry1Ac, CryI Fa, Cry2Ab, mCry3A, Cry3Ab, Cry3Bb, Cry34 / 35Ab1. ] Inhibitors of mitochondrial ATP synthase, for example Diafenthiuron; or organotin miticides, for example Azocyclotin, Cihexatin, and Feñbutatin oxide; or Propargita; or Tetradiphon.

Disengagement of disruption through oxidative phosphorylation of the proton gradient, for example Clorfenapyr, DNOC, and Sulfluramid.

Blockers of the nicotinic acetylcholine receptor channel (nAChR), for example Bensultap, Cartap hydrochloride, Tiociclam, and Tiosultap-sodium. ·; Inhibitors of chitin biosynthesis, type 0, for example isififluron, Clorifluzuron, Diflubenzuron, Flucycloxuron, Flufenoxuron, Hexaflumuron, Lufenuron, Novaluron, Noviflumuron, Teflubenzuron, and Triflumuron.

Inhibitors of chitin biosynthesis, type 1, for example Buprofezin.

Disruptors of the molt, for example Ciromazine.

Ecdysone receptor agonists, for example Chromafenozide, Halofenozide, Methoxyfenozide, and Tebufenozide.

Octopamine receptor agonists, for example Amitraz.

Inhibitors of electron transport of mitochondrial complex III, for example Hydramethylnon; Acequinocyl; or Fluacripirim.

Inhibitors of electron transport of the mitochondrial complex I, for example METI acaricides, for example Fenazaquine, Fenpyroximate, Pirirnidifen, Piridaben, Tebufenpyrad, and Tolfenpirad; or Rotenone (Derris).

Voltage-dependent sodium channel blockers, for example Indoxacarb; or Metaflumizone.; Acetyl CoA carboxylase inhibitors, for example derivatives of tetronic and tetrotic acid, for example Spirodiclofen, Spiromesifen, and Spirotetramat.

Inhibitors of mitochondrial IV complex electron transport, for example phosphines, for example aluminum phosphide, calcium phosphide, phosphine, zinc phosphide; or cyanide.

Inhibitors of electron transport of the mitochondrial complex II, for example Cienopyrafen.

Modulators of the ryanodine receptor, for example diamides, for example chlorantraniliprole and flubendiamide.; Other active ingredients with uncertain or unknown mode of action, for example Amidoflumet, Azadirachtin, Benclotiaz, Benzoximate, Bifenazate, Bromopropylate, Cinomethionate, Cryolite, Cyantraniliprol (Ciazipir), Ciflumetofen,; difluoroethyl) amino} furan-2 (5H) -one (known from WO2007 / 115644), 4-. { [(2-chloro-1,3-thiazol-5-yl) methyl] (2-fluoroethyl) amino} furan-2 (5H) -one (known, from WO2007 / 115644), 4-. { [(6-chloropyridin-3-yl) methyl] (2-fluoroethyl) amino} furan-2 (5H) -one (known from WO2007 / 115644), 4-. { [(6-chloropyridin-3-yl) methyl] (2,2-difluoroethyl) amino} furan-2 (5H) -one (known from WO2007 / 1 15644), 4-. { [(6-chlor-5-fluoropyridin-3-yl) methyl] (methyl) amino} furari-2 (5H) -one WO2007 / 1 15643), 4-. { [(6-chloropyridin-3-yl) methyl] (cyclopropyl) amide} furan-2 (5H) -one (known from EP-A-0 539 588), 4-. { [(6-cyor-iridin-3-yl) methyl] (methyl) amino} furan-2 (5H) -one (known from EP-A-0 5 398 588),. { [1- (6-chloropyridin-3-yl) ethyl] (methyl) oxide-A4-sulfanilidene} cyanamide (known by the WO2007 / 149134 document) and its diastereomers. { [(1 R) -1- (6-chloro] r¡d¡n-3-yl) ethyl] (methyl) oxide-A4-sulfanilidene} cyanamide (A) and. { [(1 S) -1- (6-chloropyridin-3-yl) ethyl] (methyl) oxide-A4-sulfanylidene} cyanamide (B) (also known from WO2007 / 149134) as well as Sulfoxaflor (also known from WO2007 / 149134) and its diastereomers [(R) -methyl (oxide). { (1 R) -1- [6- (Trifluoromethyl) pyridin-3-yl] ethyl} -A4-sulfanylidene] cyanamide (A1) and [(S) -methyl (oxide). { (1 S) -1- [6- (Trifluoromethyl) pyridin-3-yl] ethyl} -A4-sulfanylidene] cyanamide (A2), mentioned as a group of diastereomers A (known from WO2010 / j074747, WO2010 / 074751), [(R) -methyl (oxide). { (1 S) -1- [6- (trifluoromethyl) pindin-3-yl] etl} -A4-sulfanylidene] cyanamide (B1) and [(S) -methyl (oxid). { (1 R) -1- [6- (trifluoromethyl) pyridin-3-yl] ethyl} -A4-sulfanilidene] cyanamide (B2), referred to as a group of diásterémes B (also known as WO2010 / 074747, WO2010 / 074751), and 11- (4-chloro-2,6-dimethylphenol) -12-hydroxy-1,4-dioxa-9-azadiespiro [4.2.2] tetradec-11: -eh- 0- (trifluoromethyl) -1 H-1, 2,4-triazol-5-amine (known from WO2006 / 043635), cyclopropanecarboxylate of [(3S, 4aR, 12R, 12aS, 1: 2bS) -3- [(cyclopropylcarbonyl) oxy] -6 2-dihydroxy-4-b-dimethyl-11-oxo-9- (pyridin-3-yl, 3,4,4a, 5,6,6a, 12, 12a, 12b-decahydro -2H, 11 H-benzo [f] pyrano [4,3-b] chromen-4-yl] methyl (known from WO2008 / 066153), 2-cyano-3- (difluoromethoxy) -N, N- dimethylbenzenesulfonamide (known from WO2006 / 056433) [2 * qano-3- (difluoromethoxy) -N-methylbenzenesulfonamide (known from WO2006 / 100288), 2-cyano-3- (difluoromethoxy) -N-ethylbenzenesulfonamide j (known by WO2005 / 035486), 1,1-dioxido (difluoromethoxy) -N-ethyl-N-methyl-1,2-benzothiazol-3-amine (known from WO2007 / 057407); N- [ 1- (2,3-dimethylphenyl) -2- (3,5-dimethylphenyl) ethyl] -4,5-dihydro-1,3-thiazol-2-amine (known, from WO2008 / 104503),. { 1 '- [(2E) -3- (4-chlorophenyl) prop-2-en-1-yl] -5-fluorospiro [indole-3,4'-piperidin] -1 (2H) -yl.}. (2-chloropyridin-4-yl) methanone (known from WO2003 / 106457), 3- (2,5-dimethylphenyl) -4-hydroxy-8-methoxy-1,8-diazaspiro [4.5] dec-3- en-2-ona (know reported in WO2009 / 049851), 3- (2,5-dimethylphenyl) -8-methoxy-2-oxo-1,8-diazaspiro [4.5] dec-3-en-4-yl carbonate and ethyl carbonate (known from WO2009 / 049851), 4- (but-2-in-1-ylxi) -6- (3,5- il) oxy] quinolin-4-yl and methyl (known from JP2008 / 110953);, 2-ethyl-7-methoxy-3-methyl-6 - [(2,2,3,3-tetrafluoro- 2,3-dihydro-1,4-benzodioxin-6- |: · (known from WO2007 / 075459), 4- [5- (3,5-dichlorophenol) -5- (trifluoromethyl) -4,5-dihydro-1,2-oxazol-3-yl] -2- methyl-N-. { 2-oxo-2 - [(2,2,2- i! · trifluoroetyl) amino] ethyl} benzamide (known from WO2005 / 085216), 4-? . { [(6-chloropyridin-3-yl) methyl] (cyclopropyl) amino} -1, 3-oxazole-2 (5H) -one, 4-. { [(6-chloropyridin-3-yl) methyl] (2,2-difluoroethyl) amino} -1, 3-oxazole-2 (5H) -one > 4-. { [(6-chloropyridin-3-yl) methyl] (ethyl) amino} -1, 3-oxazole-2 (5H) -one, 4-. { [(6-chloropyridin-3-yl) methyl] (methyl) amino} -1, 3-oxazole-2 (5H) -one (all known from WO2010 / 005692), NNI-0711 (known from WO2002096882), 1-acetyl-N- [4- (1, 1, 1 , 3,3,3-hexafluoro-2-methoxypropan-2-yl) -3-isobutylphenyl) - ^ carboxamide (known from WO2002096882), 2- [2- ( { [34> romo-1- (3-chloropyridin-2-yl) -1 H -pyrazol-5-yl] carbonyl} amino. Methyl) -5-chloro-3-methylbenzoyl] -2- 1 methylhydrazinecarboxylate (known from WO2005 / 085216), 2- [2- ( { [3-bromo-1- (3-chloropyridin-2- yl) -1 H -pyrazol-5-yl] carbonyl}. amino) -5-cyano-3-methylbenzoyl] -2-ethylhydrazinecarboxylic acid methyl ester (known from WO2005 / 085216), 2- [2- ( { [3-bromo-1- (3-chloropyridin-2-yl) -1H-pY-amol-5-yl] carbonyl}. Amino) -5-cyano-3-methylbenzoyl] -2-methylhydrazinecarboxylate. methyl (known from WO2005 / 085216), 2- [3,5-dibromo-2- ( { [3-bromo-1- (3-chloropyridin-2-yl) -1 H-pyrazole-5- il] carbonyl.} amino) benzoyl (known from WO2005 / 085216), 2- [3,5-dibromo-2- ( { [3-bromo-1- (3-chloropyridine -2-yl) -1H-pyrazol-5-yl] carbonyl} amino) benzoyl] -2-ethylhydrazincarb methyl ester (known from WO2005 / 085216), (5RS, 7RS, 5RS, 7SR) - 1- (6-chloro-S-pyridylmethyl-I ^. S.S.e.-hexahydro ^ -methyl-S-nitro-S-propoxyimidazofli ^ j 'a] pyridine (known from WO2007 / 101369), 2-. { 6- [2- (5-fluoropyridin-3-yl) -1, 3-thiazol-5-yl] pyridin-2-yl} pyrimidine (known from WO2010 / 006713), 2- (methylcarbamoyl) phenyl] -3-. { [5- (trifluorom carboxamide (known from WO20 0/069502), 'N-j [2- (tert-butylcarbamoyl) -4-cyano-6-methylphenyl] -1- (3-chloropyridin-2-yl) -3-. { [5- (trifluoromethyl) -l tetrazol-1-yl] methyl} -1 H-pyrazole-5-carboxamide (known from WO2010 / 069502), N- [2- (tert-butylcarbamoyl) -4-cyano-6-methylphenyl] -1- (3-chlorophyl) rdin-2 il) -3-. { [5- (trifluoromethyl) -2H-tetrazol-2-yl] methyl} -1 H-pyrazole-5-carboxamide (known from WO2010 / 069502), and (1 E) -N - [(6-chloropyridin-3-yl) methyl] -N'-cyano-N- (2, 2-difluoroethyl) ethanimidamide (known from WO2008 / 009360).

Variety of soybean of conventional selective cultivation, tolerant to ASR, resistant to the cancrosis of the stem and / or resistant to the leaf stain "eye of frog" The variety of conventional selective soybean, ASR tolerant, resistant to stem cancrosis and / or resistant to leaf spot "eye frog" and its plant parts can be treated in accordance with the present invention. It is understood that "plant parts" means all aerial and underground parts and plant organs, such as sprout, leaf, flower and root, examples which may be mentioned as leaves, stems, flowers, fruiting bodies, and seeds , jy; also roots and rhizomes. The plant parts also include cultivation material and generative and vegetative propagation material, for example cuttings, rhizomes, shoots and seeds. Preferably, it is understood that the plant parts mean the leaves, the roots, the flowers and / or the stem of the conventional selective cultivation plant, and its seed. More preferably, it is understood that the plant parts mean: leaves.

The plants or plant varieties used in accordance with the present invention are tolerant to ASR, resistant to stem cancrosis and / or resistant to the leaf spot "frog's eye". Preferably, tolerance to ASR from the plant or plant varieties according to the present invention is conferred by a gene selected from the group consisting of Rpp1, Rpp2, Rpp3, Rpp4 and Rpp5 or a combination thereof. Much more preferably, the tolerance ja ASR is conferred by a gene selected from the group consisting of Rpp2, Rpp4i and! Rpp5 or a combination thereof.

The plants or plant varieties used in accordance with the present invention are not transgenic. Transgenic organisms are produced by introducing an exogenous gene (a transgene) into a living organism using genetic engineering so that the organism will present its own property. The genetic material of transgenic plants has been modified by use. of recombinant DNA techniques that in natural circumstances can not be easily obtained by cross-culture, mutations or natural recombination, so the modification confers tolerance, ASR, resistance to stem cancrosis and / or resistance to leaf spot "frog's eye", or confers the increase of tolerance to ASR, resistance to stem cancrosis and / or resistance to leaf spot "frog's eye".

Genetic engineering is the direct human manipulation of an organism gerioma using genetic transformation techniques. DNA sequences, either from different species or from the same species, which are inserted into the genome using the transformation, are collectively referred to herein as "transgenes". A "transgene" also encompasses antisense sequences, or sense and antisense sequences capable of silencing the gene. i Numerous methods have been developed for the transformation of plants, including biological and physical plant transformation protocols (see, for example, Glick, BR and Thompson, JE Eds. (CRC Press, Inc., Boca Raton (1993): 67- 88 and Armstrong, "The First Decade of Maize Transformed:! '; Review and Future Perspective" Maydica 1999 (44): 101-109.) Additionally, expression vectors and in vitro culture procedures are available for the transformation of tissue or plant cells and plant regeneration See, for example, Glick, BR and Thompson, JE Eds. (CRC Press, Inc., Boca Raton (1993): 89-1 19).

In contrast, non-transgenic plants are produced by traditional selective breeding techniques (eg, crossing / recurrent crossing / self-fertilization, etc.). ! Preferably, the soybean variety of conventional selective culture, tolerant to ASR, resistant to stem cancrosis and / or resistant to spotting; "frog eye" blade according to the present invention is obtained; making segregating generations, preferably the procedure to granéí,. SSD (offspring of a single seed) and / or recurrent crossing. |: More preferably, the variety of conventional selective soybean, tolerant to ASR, resistant to the cancrosis of the stem and / or resistant to the leaflet "frog's eye" leaf according to the present invention is obtained by means of the bulk procedure. In the bulk process, the segregating generations, usually F2 and F5 are cultivated with the sowing and harvesting of all the plants mixed in a single population. Therefore, in the bulk process, the seeds used to grow each segregating generation are one of the seeds harvested in the previous generation. After five generations of self-fertilization cultures, the plants will present a high degree of honjiocigosis and can be selected for the individual harvest (Souza, AP Biology! Molecular Applied to or melhoramento In: Genetic Resources and Melhoramento - Plants Luciano L. Nass, Alfonso CC Valois, Itamar S. de Melo, Maria Cléli Valadres-Inglis (Org.) 1st Ed. Rononópolis, 2001, 1: 939-966).

In another embodiment, the soy variety of conventional selective culture, tolerant to ASR, resistant to stem cancrosis and / or resistant to leaf spot "frog eye" according to the present invention is obtained by the SSD method. The SSD procedure was described by Brim in 1966 (Brirn, CA, 1966. A modified pedigree method of selection in soybeans, Crop Science, i 6: 20) and consists of the segregating generation advance (from F2 to F5), and a unique flower pot (2 to 3 seeds) per plant; however, only one plant from each pot is used to grow the next generation. A sample is harvested and preserved. In this way, at the end of the process, each line corresponds to a different plant F2 and, therefore, there is a reduction in the loss caused by deficient sampling or natural selection. i; In another preferred embodiment the conventional selective, ASR-tolerant cultivar variety, resistant to stem cancrosis and / or resistant to the "frog's eye" leaf spot in accordance with the present invention is; obtains through recurrent crossing. Recurrent crossing is not exactly a procedure to cultivate segregation populations. It is a strategy used to improve the phenotypic expression of a deficient trait, especially ';, if this trait is of a qualitative inheritance. The use allows the transfer of üñ gene or a few genes of a parent called donor parent (RP) ': to another parent called recurrent parent (PR), and the parent! Recurrent is usually a cultivar of commercial interest that has some kind of i I, Deficiency in your crop that needs to be improved. This deficiency can be corrected through the process of transferring the gene from the donor parent, who does not have a deficiency, to the recurrent parent. This procedure, that is, the crossing of individuals from the segregating population with the recurrent parent, is called recurrent crossing and is responsible for the recovery of almost 100% of the genotype of the recurrent parent (Souza, AP Molecular Biology Applied to or melhoramento. Genetic Resources and Melhoramento - Plants Luciano L. ass, Alfonso CC Valois, Itamar S. de Melo, Maria Clélia Valadres-lnglis (Org.) 1st Edition, Rononópolis, 2001, 1: 939-966). '! Preferably, at the end of the selective processes, according to what is described above, one or a few pure lines with superior traits that will give rise to a new cultivar are identified.

Much more preferably, the plant varieties used according to the present invention are TMG 801 and / or TMG 803 d Tropical Melhoramento e Genetica LTDA, Brazil.

Soy type superior feature like ASR, resistant to stem cancrosis and / or resistant to leaf spot "frog's eye" but is in any other property as similar as possible to soybean, used in the process of the present invention. The plant of natural type is in its genome, transcriptome, proteome or metabolome as similar as spa; possible to a plant, used in the process of the present invention. P re re ntérri ent, natural-type soybeans are a Miyagishirome variety.

The treatment of soybean varieties of conventional selective cultivation, tolerant to ASR, resistant to canker of the stem and / or resistant to the leaf spot "frog's eye", its parts of plant, material of propagation of the plant, Their growth locus with the SDHI fungicide according to the present invention is carried out directly or by means of the action in the lambing medium, habitat or storage space using usual treatment methods, for example by immersion, spraying, atomization, fogging, evaporation, dedusting, mist treatment, sweep, treatment with foam, paint, dissemination, injection, soaking,! drip irrigation and, in the case of propagation material, in particular in the seed casserole, in addition by the dry seed treatment procedure, the wet process seed treatment procedure, the suspension treatment process, by incrustation, by coating with one or more coatings and the like. It is also possible to apply the SDHI fungicide by the ultra-low volume method. Preferably, the fungicide SDHI is sprayed on the variety of soybeans, parts of the plant, or its growth locus.

The present invention further provides formulations and application forms prepared from fungicidal compositions described above comprising at least one of the SDHI fungicides of the invention. The fungicidal formulations or application forms in question are preferably those comprising adjuvants, such as extenders, solvents and vehicles, for example, and / or other adjuvants, such as tetrasubjective substances, for example.

Examples of customary formulations include solutions, emulsions, wettable powders, water-based and oil-based suspensions, water-based and oil-based suspension concentrates, powders, dusts, pastes, soluble powders, granules, dispersed granules. , Soluble granules, granules for diffusion, suspension-emulsion concentrates, and natural materials impregnated with active compound, synthetic materials impregnated with active compound, fertilizers and ultra-fine encapsulation in polymeric compounds. Preferably, the application form prepared from the SDHI fungicide is an emulsion.

These formulations are produced in a conventional manner, for example by mixing the active compounds with such adjuvants; as extenders, solvents and / or solid carriers, for example, and / or other adjuvants such as surfactants, for example. The formulations are produced in appropriate equipment or also before or during application. ! ||| The adjuvants used can be substances capable of providing the SDHI fungicide formulation, and the forms of application prepared? from these formulations (such as crop protection compositions for use, for example, such as spraying mother liquors or seed coatings) particular properties, such as certain physical, technical and / or biological properties.

Examples of suitable extenders include water, polar and non-polar organic chemical liquids, such as those, for example, of the aromatic and non-aromatic hydrocarbon classes (such as paraffins, alkylbenzenes, alkylnaphthalenes, chlorobenzenes), alcohols and polyols (which in corjvient case may also be substituted, etherified and / or esterified), ketones (such as acetone, cyclohexanone), esters (including fats and oils) and (poly) ethers, simple and substituted amines, amides, lactams, (such as N- alkylpyrrolidones) and jactones, sulfones and sulfoxides (such as dimethyl sulfoxide).

When water is used as an extender, organic solvents can also be used as coadjuvant solvents. The liquid solvents contemplated are essentially the following: aromatics, such as xylene, toluene or alkylnaphthalenes, chlorinated aromatics or chlorinated aliphatic hydrocarbons, such as chlorobenzenes, chloroethylenes or methylene chloride, aliphatic hydrocarbons, such as cyclohexane or paraffins, such as petroleum, mineral and vegetable oils, alcohols, such as butanol or glycol and their "ethers and esters, ketones, such as acetone, methyl ethyl ketone, methyl butylbutyl ketone or cyclohexanone, strongly polar solvents such as dimethylformamide and dimethyl sulfoxide, and also water. such as chlorobenzene, chloroethylene or methylene chloride, aliphatic hydrocarbons, such as cyclohexane, paraffins, petroleum fractions, mineral and vegetable oils, alcohols, such as methanol, ethanol, isopropanol, butanol or glycol and their ethers and esters, ketones such as acetone, methyl ethyl ketone, methyl isobutyl or cetpna cyclohexanone, strongly polar solvents, such as dimethyl sulfoxide, and water. Preferably, acetone is used as the solvent.

In principle, it is possible to use all appropriate vehicles. The vehicles contemplated more particularly are the following: for example, ammonium salts and finely ground, natural minerals, such as kaolins, aluminas, talc, czeta, quartz, attapulgite, montmorillonite or diatomaceous earth, and finely ground, synthetic minerals, such as highly dispersed silica, alumina and natural or synthetic silicates, resins, waxes and / or solid fertilizers. In the same way mixtures of said vehicles can be used. The vehicles contemplated for pellets include the following: for example, fractionated and crushed natural minerals such as calcite, marble, pumice, sepiolite, dolomite, and also synthetic pellets of inorganic and finely ground organic substances, 1 and also pellets of material organic such as sawdust, paper, coconut husk, corn cobs and tobacco stems.

Liquefied solvents or gaseous extenders can also be used. The applicability is more particularly those extenders or vehicles! which are gaseous at standard temperature and under atmospheric pressure, being it? propellant aerosol examples, such as halogenated hydrocarbons, and also; butane, propane, nitrogen and carbon dioxide.; The surfactants for the purposes of the invention are emulsifiers and / or foaming agents, dispersants or wetting agents having ionic or non-ionic properties, or mixtures of these surfactant substances. Examples thereof are salts of polyacrylic acid, salts of lignosulfonic acid, salts of phenolsulfonic acid or naphthalenesulfonic acid, polycondensates of ethylene oxide with fatty alcohols or with fatty acids or with fatty amines, with substituted phenols (preferably alkylphenols or arylphenols) , salts of sulfosuccinic esters, taurine derivatives (preferably alkyl taurates), phosphoric esters of polyethoxylated alcohols or phenols, fatty acid esters of polyols, and derivatives of compounds comprising sulphates, sulfonates and phosphates, for example alkylaryl polyglycol ethers, alkylsulfonates, sulfates of alkyl, arylsulfonates, protein hydrolysates, lignin-sulfite wastewater and methylcellulose. The presence ide Surfactant is advantageous if one of the SDHI fungicides and / or inert vehicles is insoluble in water and if the application occurs in water. Preferably, polyoxyethylene alkyl phenyl ether is used as an emulsifier.

Other adjuvants present in the formulations and the application forms obtained therefrom may include dyes, such as inorganic pigments, the examples being iron oxide, titanium oxide and Prussia Blue, and organic dyes, such as alizarin dyes , azo dyes and metal phthalocyanine dyes, and nutrients, including oligonutrient.es, such as salts of iron, manganese, boron, copper, cobalt, molybdenum and zinc. : Additionally, stabilizers such as low temperature stabilizers, preservatives, antioxidants, light stabilizers and other agents that enhance physical and / or chemical stability may be present. Additionally, foamers or defoamers may be present. : The formulations and application forms obtained therefrom may further comprise, as additional adjuvants, adhesives such as carboxymethylcellulose, natural and synthetic polymers in powder, granule or latex form, such as gum arabic, polyvinyl alcohol, polyvinyl acetate and also natural phospholipids, such as cephalins and lecithins, and synthetic phospholipids. Other possible adjuvants include mineral and vegetable oils.

If desired, other adjuvants may also be present, in the formulations and in the forms of application obtained therefrom. Examples of such adjuvants include fragrances, proteomeous colloids, binders, adhesives, thickeners, thixotropic agents, penetrants, retention promoters, stabilizers, sequestering agents and binding agents. In general terms, the active compounds can be combined with any solid or liquid adjuvant which is commonly used for formulation purposes.

The SDHI fungicide can be present in its commercially customary formulations and also in the application forms prepared from those formulations, in a mixture of other active compounds such as insecticides, attractants, sterilants, bactericides, acaricides, nematicides, fungicides, growth regulators, herbicides, plagjuicide antidotes, fertilizers, semiochemical compounds or other agents to enhance the properties of the plant.

The formulations preferably contain between 0.00000001% and 98% by weight of the fungicide SDHI or, with particular preference, between 0.01% and 95% by weight of the fungicide SDHI, more preferably between 0.5. % and 90%: by weight, much more preferably 1% by weight of the SDHI fungicide, based on the weight of the formulation. In the above context the term "SDHI fungicide" also includes combinations of SDHI fungicides. .

Preferably, the formulations comprise at least one of the SDHI fungicides, a solvent, an emulsifier and / or water. 1 ! The SDHI fungicide content of the application forms (compositions preferably between 0.2 ppm and 500 ppm, more preferably between! 0, 2 ppm and 100 ppm. In the above context, the term "SDHI fungicide" also includes combinations of SDHI fungicides. 1 \ - | Most preferably, penflufen is applied as an emulsion, it contains 0.2 ppm to 50 ppm of said fungicide.

Most preferably, isopyrazam is applied as an emulsion containing 0.2 ppm to 1 ppm of said fungicide.

Most preferably, bixafen is applied as an emulsion containing 0.2 ppm to 100 ppm of said fungicide. \ Most preferably, sedative is applied as an emulsion containing 0.2 ppm to 25 ppm of said fungicide.

Most preferably, fluxapiroxad is applied as an emulsion containing 0.2 ppm to 25 ppm of said fungicide. II The application volume of the fungicide SDHI to a variety of conventional selective cultivation soybeans, propagation material of the plant, or its growth locus is in the range of 0.01 kg / ha to 3 kg / ha, preferably 0 , 01 kg / ha to 1, 5 kg / ha, more preferably 0.02 kg / ha to 0.5 kg / ha. Preferably, the Application volume of SDHI fungicide is 25-500 l / ha, preferably 25-250 l / ha.

Preferably, the present invention relates to a kit of parts comprising a variety of conventional selective soybean, tolerant to ASR, resistant to stem cancrosis and / or resistant to leaf spot "frog eyes" and a fungicide SDHI selected from the group consisting of penflufen, jsppirazam, bixafen, sedaxane, fluxapiroxad, fluopiram, pentiopirad, boscalid, - [1- (2,4-dichlorophenyl) -1-methoxypropan-2-yl] -3- (difluoromethyl) -1 -methyl-1 H-pyrazole-4-carb; oxamide, N- [9- (dichloromethylene) -1, 2,3,4-tetrahydro-1,4-methanonaphthalen-5-yl] -3- (difluoromethyl) - 1-methyl-1 H-pyrazole-4-carboxamide, N - [(1 S, 4 R) -9- (dichloromethylene) -1, 2,3,4-tetrahydro-1,4-methanonaphthalene-5-yl ] -3- (difluoromethyl) -1-methyl-1 H-pyrazole-4-carboxamide, and N - [(1 R, 4S) -9- (Dichloromethylene) -1, 2,3,4-tetrahydro-1, 4-methanonaphthalen-5-yl] -3- (d-fluoroethyl) l ÷ -methyl-1 H-pyrazole-4-carboxamide.

According to another embodiment of the present invention, the kit of parts comprises a variety of conventional selective soybean, tolerant to ASR, resistant to stem cancrosis and / or resistant to leaf spot "eye of a frog" and a SDHI fungicide selected from the group consisting of penflufen, sopirazam, bixafen, sedaxane, and fluxapiroxad. According to another embodiment of the present invention, the kit of parts comprises the varieties of soybean TI \) 1G.:801 and / or TMG 803 of conventional selective culture tolerant to ASR, resistant to canker of the stem and / or resistant to the leaf spot "frog's eye".

Other preferred embodiments in relation to this kit of parts comprise the embodiments as defined above.; The present invention also comprises a process for the production of an agricultural product comprising the application of an SDHI fungicide to a conventional selective soybean variety, tolerant to ASR, resistant to stem canker and / or resistant to leaf spot "ojo de riaria", its propagation material of the plant, or its locus of growth, and the production of an agricultural product from said plant or parts of said plant or material of 1; propagation of the plant.

According to a preferred embodiment of the present invention, the SDHI fungicide applied in said process for the production of an agricultural product is selected from the group consisting of penflufen, isopyrazam, bixafen, sedaxane, fluxapiroxad, fluopyram, pentiopyrad, boscalid, N- [1- (2,4-dichlorophenyl) -1-methoxypropan-2-yl] -3- (difluoromethyl) -1-methyl-1 H -pyrazole-4-carboxamide, N- [9- (dichloromethylene) -l, 2,3,4-tetrahydro-1,4-methanonaphthalen-5-yl] -3- (difluoromethyl) -1-methyl-1 H-pyrazole-4-carboxamide, N - [(1 S, 4R) -9- (dichloromethylene) -1, 2,3,4-tetrahydro-1,4-methanonaphthaien-5-yl] -3- (difluoromethyl) -1-methyl-1 H-pyrazole-4-carboxamide, and N - [(1 R, 4S) -9- (dichloromethylene) -1, 2,3,4-tetrahydro-1,4-methanonaphthalen-5-yl] -3- (difluoromethyl) -1-methyl-1 H -pyrazole-4-carboxamide . i In accordance with another preferred embodiment of the present invention, the fungicide SDHI applied in said process for the production of an agricultural product is applied at least in parts of the soybean variety of conventional selective culture, tolerant to ASR, resistant to canker stem and / or resist to the leaf spot "frog's eye", preferably leaves, roots, flowers and / or the stem of the conventional selective cultivation plant, and its seed. According to another preferred embodiment, the SDHI fungicide is applied as an emulsion, preferably containing 0.2 to 500 ppm of said fungicide. According to still another preferred embodiment, the application volume of the SDHÍ fungicide is in the range of 0.01 to 3 kg / ha, preferably 0.01 to 1.5 kg / Ha; more preferably 0.02 to 0.5 kg / ha. According to yet another preferred embodiment, the tolerance to ASR in the variety of soybean of conventional selective culture, tolerant to ASR, resistant to the cancrosis of the stem and / or resistant to the leaf spot "frog's eye" is conferred by the gene selected from the group consisting of Rpp1, Rpp2, Rpp3, Rpp4 and Rpp5 or a combination thereof. Even more preferably, tolerance to ASR is conferred by the gene selected from the group consisting of Rpp2, Rpp4 and Rpp5 or a combination of the (s), and also preferably, the soybean variety of conventional selective culture, tolerant to ASR, resistant to cancrosis of the stem and / or resistant to the stain ; i.; .. "eye of a frog" is TMG 801 and / or TMG 803.

I ' Another preferred embodiment of the present invention is also the use of an SDHI fungicide to combat ASR from a variety of conventional selective soybean, ASR tolerant, resistant to stem canker and / or leaf spot resistant. frog "comprising the application of an SDHI fungicide to said plant, propagation material of the plant, or at its growth locus.

Example A susceptible variety (Miyagishirome) and soybean tolerant soybean rust varieties (Phakospora pachyrhizi) (TMG 801 and TMG; 803) were grown in a greenhouse to a leaf stage of 1, 5. There was adequate preparation of I! Fungicide SDHI by mixing 1 part by weight of the SDHI ingredient with 28.5 parts by weight of acetone as solvent and 1.5 parts by weight of polyoxyethylene alkyl phenyl ether as emulsifier. The concentrate was diluted with water: to the desired concentrations. ¡¡ Young plants of the susceptible and tolerant soybean varieties were sprayed with SDHI fungicide preparation at the application rate established to evaluate the combat activity of Phakopsora pachyrhizi.

One day after spraying, soybean plants were inoculated with an aqueous suspension of spores of the causative agent of soybean rust. { Phakopsora pachyrhizi). The plants were then placed in a greenhouse at about 20 ° C and a relative atmospheric humidity of about 80% Se; applied the following test program: The plants were evaluated 1 day after the inoculation. The percentage of infected leaf area was determined visually. The results were calculated in accordance with ABBOTT (% efficiency). 0% means an efficacy that corresponds to that of the control, while an efficacy of 100% sjgriífíca that no disease was observed. j " The expected efficiencies for the combinations of fungicide and SDHI and ASR tolerant variety were determined in accordance with the Coiby formula and compared with the observed efficiencies (Coiby, SR (1967); Calculating synergistic and antagonistic responses of herbicide combinations, Weeds,? ? d? 20-22): E = x + y-xy / 100 wherein E is the expected efficacy as a percentage of control when using the combination of a fungicide and the ASR tolerant variety; x is the effectiveness of the percentage control when using the fungicide; and it is the efficiency of the percentage control when using the ASR tolerant variety.

By comparing the expected efficacy and the observed efficacy, the synergistic effect can be analyzed. If the percentage efficiency observed is greater than the estimated percentage efficiency calculated, there is a synergistic effect.

Table 1. Penflufen Table 2. Isopyrazm Table 3. Bixafen I.

Table 4. Sedaxane Table 5. Fluxapiroxad The results clearly demonstrate that the combination of an SDHI fungicide and a Phakopsora pachyrhizi tolerant variety provides a synergistic effect in relation to the combat of soybean rust. The observed activity of the SDHI fungicide combination and tolerance tolerant variety: with the invention was greater than the activity calculated using the Colby formula; The most pronounced synergistic effect was found when applying fluxapiroxad with 1 ppm of i.a. to TMG 803. The effect in low concentrations of said fungicide is more pronounced than the effect in concentrations up to 25 ppm of: i.a! The same synergistic effect trend for sedaxan that is applied to TMG 803 can be found. The application of this fungicide at lower concentrations (0.2 ppm ai to 1 ppm ai) provided higher synergism than the application of sedative at concentrations of 5 ppm ai at 25 ppm i.a.

In contrast to this, the application of bixafen to TMG 803 shows the highest synergistic effect for concentrations varying from 100 ppm of La. at 25 ppm i.a. i :: When penflifenum is applied at a concentration of 10 ppm i i.á., the highest deviations of actual efficacy of the expected value are obtained. This is regardless of the type of plant (TMG 801 or TMG 803).

Taken together, TMG 803 shows a higher potential for synergistic behavior independent of the SDHI fungicide used. ::

Claims (15)

1. A method for combating Asian soybean rust (ASR) from a variety of conventional selective soybean, tolerant to ASR, resistant to stem cancrosis and / or resistant to leaf spot "frog eye", characterized in that comprises the step of::; apply a fungicide inhibitor of succinate dehydrogenase (SDHI) to said plant, propagating material of the plant, or at its growth locus,!

2. The method according to claim 1, characterized in that the SDHI fungicide is selected from the group consisting of penflufen, isopyrazam, bixafen, sedaxane, fluxapiroxad, fluopyram, pentyopril, boscalid, N- [1- (2,4-dichlorophenyl) -1. -methoxypropan-2-yl] -3- (difluoromethyl) -1-methyl-1 H -pyrazole-4-carboxamide, N- [9- (dichloromethylene) -1, 2,3,4-tetrahydro-1, 4- methanonaphthalen-5-yl] -3- (difluoromethyl) -l-methyl-1 H-pyrazole-4-carboxamide, N - [(1S, 4R) -9- (diclote ometylene) -1, 2,3, 4-tetrahydro-1,4-methanonaphthalen-5-yl] -3- (difluoromethyl) -1-methyl-1 H-pyrazol-4-carboxamide, and N - [(1R, 4S) -9- (dichloromethylene) - 1, 2,3,4-tetrahydro-1,4-methanonaphthalen-5-yl] -3- (difluoromethyl) -1-methyl-1 H-pyrazole-4-carboxamide.

3. The method according to the claims ·; j or 2, characterized in that the fungicide SDHI is applied at least in parts of the soy variety of conventional selective culture, tolerant to ASR, resistant to canprosis of the stem and / or resistant to the leaf spot "frog's eye" , preferably the leaves, the roots, the flowers and / or the stem of the conventional selective cultivation plant, and its seed. |; · |

4. The method according to any of claims 1 to 3, characterized in that tolerance to ASR is conferred by; the gene selected from the group consisting of Rpp1, Rpp2, Rpp3, Rpp4 and Rpp§ or a combination thereof.

5. The method according to any of claims 1 to 3, characterized in that tolerance to ASR is conferred by the gene selected from the group consisting of Rpp2, Rpp4 and Rpp5 or a combination thereof. i

6. The method according to any of the claims 1 to 3, characterized in that the variety of conventional selective soybean, tolerant to ASR, resistant to canker of the stem and / or resistant to leaf spot "frog's eye" is TMG 801 and / or TMG 803.

7. The method according to any of the claims 1 to 6, characterized in that the SDHI fungicide is applied as an emulsion, preferably containing 0.2 to 500 ppm of said fungicide.; i

8. The process according to any of claims 1 to 7, characterized in that the application volume of the fungicide SDHlj is in the range of 0.01 to 3 kg / ha, preferably 0.01 to 1.5 kg / ha, more preferably 0.02 to 0.5 kg / ha. 'í

9. A kit of parts comprising a variety of soybean of conventional selective culture, tolerant to ASR, resistant to canker of the stem and / or resistant to leaf spot "frog's eye" and an SDHI fungicide selected from the group consisting of penflufen, isopyrazam, bixafen, sedaxane or fluxapiroxad, fluopiram, pentyopril, boscalid, N- [1- (2,4-dichlorophenyl) -1-methoxyprpapan-2-yl] -3- (difluoromethyl) -1-methyl-1 H-pyrazole 4-carboxamide, N- [9- (dichloromethylene; no) f1, 2,3,4-tetrahydro-1,4-methanonaphthalen-5-yl] -3- (difluoromethyl) -1-methyl-1 H-pyrazole 4-carboxamide, N - [(1 S, 4R) -9- (dichloromethylene) -1, 2,3,4-tetrahydro-1,4-methanonaphthalen-5-yl] -3- H (difluoromethyl) -l -methyl-1 H-pyrazole-4-carboxamide, and N - [(1 R, 4 S) -9- (dichloromethylene) -1, 2,3,4-tetrahydro-1,4-methanonaphthalen-5-yl] - 3- (difluoromethyl) -1-methyl-1 H-pyrazole-4 '. carboxamide. '| · |

10. The kit of parts according to claim 9, characterized in that the variety of conventional selective soybean, tolerant to ASR, resistant to stem cancrosis and / or resistant to leaf spot "frog's eye" is TMG 801 and / or TMG 803.

1 1. A procedure for the production of an agricultural product, characterized in that it comprises the passage of apply an SDHI fungicide to a variety of conventional selective soybean, tolerant to ASR, resistant to stem cancrosis and / or resistant to leaf spot "frog eye", its plant propagation material, or in its locus of growth, and the production of the agricultural product from said plant or parts of said plant or propagating material of the plant.

12. The method according to claim 1, characterized in that the SDHI fungicide is defined as in claim 2.!:

13. The process according to claims 1, 1 or 12, characterized in that the SDHI fungicide is applied as defined in claims 3, 7 and 8.

14. The process according to claims 11 to 13, characterized in that the variety of soybean of conventional selective culture, tolerant to ASR, resistant to the cancrosis of the stem and / or resistant to the leaf spot of "frog's eye" is defined as in any one of claims 4 to 6. ¡•; |: \

15. Use of an SDHI fungicide to combat ASR from a variety of conventional selective soybeans, ASR-tolerant, canker-resistant, stem-resistant and / or frog-eye leaf spot-resistant. application of an SDHI fungicide to said plant, propagation material of the plant, or in its growth locus. i .i, i