CA2761068A1 - Nouveaux regimes posologiques et methodes de traitement, de prevention ou de gestion d'au moins un trouble du systeme nerveux central
- Google Patents
Nouveaux regimes posologiques et methodes de traitement, de prevention ou de gestion d'au moins un trouble du systeme nerveux central
Download PDF
Legal status (The legal status is an assumption and is not a legal conclusion. Google has not performed a legal analysis and makes no representation as to the accuracy of the status listed.)
Current Assignee (The listed assignees may be inaccurate. Google has not performed a legal analysis and makes no representation or warranty as to the accuracy of the list.)
Sumitomo Pharma Co Ltd
Original Assignee
Sumitomo Dainippon Pharma Co Ltd
Priority date (The priority date is an assumption and is not a legal conclusion. Google has not performed a legal analysis and makes no representation as to the accuracy of the date listed.)
Filing date
Publication date
Application filed by Sumitomo Dainippon Pharma Co LtdfiledCriticalSumitomo Dainippon Pharma Co Ltd
Publication of CA2761068A1publicationCriticalpatent/CA2761068A1/fr
Pharmaceuticals Containing Other Organic And Inorganic Compounds
(AREA)
CA 27610682011-12-022011-12-07Nouveaux regimes posologiques et methodes de traitement, de prevention ou de gestion d'au moins un trouble du systeme nerveux central
AbandonedCA2761068A1
(fr)
Pimavanserin, a selective serotonin (5-HT) 2A-inverse agonist, enhances the efficacy and safety of risperidone, 2 mg/day, but does not enhance efficacy of haloperidol, 2 mg/day: comparison with reference dose risperidone, 6 mg/day
Mirtazapine add‐on therapy in the treatment of schizophrenia with atypical antipsychotics: a double‐blind, randomised, placebo‐controlled clinical trial
A comparison of the effects of loxapine with ziprasidone and thioridazine on the release of dopamine and acetylcholine in the prefrontal cortex and nucleus accumbens
Synaptic dysfunction and dysregulation of extracellular matrix-related genes in dopaminergic neurons derived from Parkinson’s disease sporadic patients and with GBA1 mutations