IL119119A - Rhodanine derivatives process for their preparation and pharmaceutical compositions containing them - Google Patents
Rhodanine derivatives process for their preparation and pharmaceutical compositions containing themInfo
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- IL119119A IL119119A IL11911993A IL11911993A IL119119A IL 119119 A IL119119 A IL 119119A IL 11911993 A IL11911993 A IL 11911993A IL 11911993 A IL11911993 A IL 11911993A IL 119119 A IL119119 A IL 119119A
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Description
ΙΓΠΝ 0Ί7Ί3ΠΠ ΠΙΠΡΙΊ ""ΤϋΟΓΠ 1ΠΟΠ7 Τ7ΠΠ ,ΠΤΠ ΏΜΏΙ RHODANINE DERIVATIVES, PROCESS FOR THEIR PREPARATION AND PHARMACEUTICAL COMPOSITIONS CONTAINING THEM *¾-6715Α - 1 - This application is a division of application no. 106877, filed September 2, 1993 and claiming a priority of September 10, 1992.
The present invention relates to certain rhodanine derivaties, pharmaceutical formulations comprising them, processes for preparing them, and their use as medicaments.
Diabetes mellitus is a systemic disease characterized by disorders in the metabolism of insulin, carbohydrates, fats and proteins, and in the structure and function of blood vessels. The primary symptom of acute diabetes is hyperglycemia, often accompanied by glucosuria, the presence in urine of large amounts of glucose, and polyuria, the "excretion of large volumes of urine. Additional symptoms arise in chronic or long standing diabetes. These symptoms include degeneration of the walls of blood vessels. Although many different organs are affected by these vascular changes, the eyes and kidneys appear to be the most susceptible. As such, long-standing diabetes mellitus, even when treated with insulin, is a leading cause of blindness.
There are two recognized types of diabetes.
Type I diabetes is of juvenile onset, ketosis-prone, develops early in life with much more severe symptoms and has a near-certain prospect of later vascular involvement. Control of this type of diabetes is difficult and requires exogenous insulin administration.
Type II diabetes mellitus is of adult onset, ketosis- resistant, develops later in life, is milder and has a more gradual onset.
One of the most significant advancements in the history of medical science came in 1922 when Banting and r:\0library\karchive\up-4500\4107nopc.doc X-6715A -2- Best demonstrated the therapeutic effects of insulin in diabetic humans. However, even today, a clear picture of the basic biochemical defects of the disease is not known, and diabetes is still a serious health problem. It is believed that two percent of the United States' population is afflicted with some form of diabetes.
The introduction of orally effective hypoglycemic agents was an important development in the treatment of hyperglycemia by lowering blood glucose levels. Oral hypoglycemic agents are normally used in the treatment of adult onset diabetes .
A variety of biguanide and sulfonylurea derivatives have been used clinically as hypoglycemic agents. However, the biguanides tend to cause lactic acidosis and the sulfonylureas, though having good hypoglycemic activity, require great care during use because they frequently cause serious hypoglycemia and are" most effective over a period of ten years.
In Chem cal & Pharmaceutical Bulletin. 3_Q_, 3563 (1982), Chemical & Pharmaceutical Bulletin. 3J2, 3580 (1982) and Chemical & Pharmaceu ical Bulletin. 22., 2267 (1984) , reference is made to a variety of thiazolidinediones which have blood glucose and lipid lowering activities.
Antidiabetic activity of ciglitazone was also reported in Diabetes r 32. 804 (1983) . However, these compounds have proven difficult to use because of insufficient activities and/or serious toxicity problems .
Furthermore, Alzheimer's disease, a degenerative disorder of the human brain, continues to afflict more and more persons throughout the world. Such disease results in progressive mental deterioration manifested by memory loss, confusion, disorientation and the concomitant loss, of r.\0librar \karchive\up-4500\ 107nopc.doc X-6715A -3 - enjoyment of life associated therewith. At the present time there is no scientifically recognized treatment for Alzheimer's disease. Because of this, and because of the debilitating effects of the disease, there continues to exist an urgent need for effective treatments.
The invention of application no. 106877 relates to a series of compounds having cathepsin inhibitory activity. As will be discussed more fully below, compounds capable of inhibiting cathepsin (and, in particular, cathepsin D) may be useful for treating Alzheimer's disease. Accordingly, an object of the invention of application no. 106877 is to provide compounds for use in the preparation of medicaments for the treatment of Alzheimer's disease. n\01ibrar \karchive\up-4500\4107nopc.doc X-6715A -4- The invention of application no. 106877 provides compounds of formula (la) wherein : Ar is (i) phenyl, (ii) phenyl substituted with from one to three substituents independently selected from Ci-Cg alkyl, Ci-C8 alkoxy, Ci-C8 alkylthio, trifluoromethyl, C1 - C4 alkylphenyl, phenyl, NO2, F, CI, hydroxy, phenoxy, Cx-C4 alkyloxyphenyl, thiophenyl, C1-C4 alkylthiophenyl, -COOR7, -N(R7)S02R7 or -N(R7)2, where each R7 is independently hydrogen or Ci-Cg alkyl or (iii) 1- or 2-naphthyl; R1 is C1 - C6 alkyl, C1-C4 alkylphenyl, hydrogen, phenyl or phenyl substituted with one or two substituents independently selected from Cl, Br, F, I, Ci-C4 alkyl, Ci- C4 alkoxy, hydroxy, trifluoromethyl, -NH2, -NH(Ci-C4 alkyl), -N(Ci-C4 alkyl)2 or C1-C4 alkylthio; R2 and R3 are each hydrogen or when taken together form a bond; R4 and R5 are each hydrogen or when taken together are =S, or when one of R4 and R5 is hydrogen, the other is -SCH3; R6 is hydrogen, C1- C6 alkyl, C3 - C8 cycloalkyl, C2-C6 alkenyl, -S02CH3, or -(CH2)p-Y where p is 0, 1, 2, or 3 and Y is O II cyano, -OR8, -CR9, tetrazolyl, - R10R1:1-, -SH, C1-C4 alkylthio, or r.\0librar \karchive\up-4500\4107nopc.doc X-6715A -5- 0-(CrC4 alkyl) O II where R8 is hydrogen, C1-C4 alkyl or -C-d-C4 alkyl, R9 is hydrogen, 0χ-04 alkyl, C1-C4 alkoxy, hydroxy or NH2/ and R10 and R11 are each independently hydrogen, Ci-Cg alkyl, C2-C6 alkenyl, C2-C6 alkynyl, phenyl, x-C^ alkylphenyl, -(CH2)qOH, - (CH2)qN(C1-C4 alkyl) 2( or - (CH2) S (¾-σ alkyl) , where q is an integer from 1 to 6, both inclusive, or R10 and R11, taken together with the nitrogen atom to which they are attached, form a morpholinyl, piperidinyl, piperizinyl, or N-methylpiperazinyl ring; and m is .0 , 1, or 2; or a pharmaceutically acceptable salt thereof, for use as medicaments for treating Alzheimer's disease in a mammal suffering from or susceptible to such disease, for example by administering a therapeutically effective amount of a compound of formula (la) to a mammal in need of such treatment .
The present invention provides novel compounds of the formula (II) wherein : Ar is (i) phenyl, (ii) phenyl substituted with from one to three substituents independently selected from Ci-C8 alkyl, C1-C3 alkoxy, Ci-Ca alkylthio, trifluoromethyl, C2-C alkylphenyl, N02, F, Cl, phenoxy, > ' Λ n\0libraiy\karchive\up-4500\ 107nopc.doc X-6715A -6- C!-C4 alkyloxyphenyl, thiophenyl, C1-C alkylthiophenyl, -COOR7, -N(R7)S02R7 or -N(R7) 2,· "where each R7 "is independently' hydrogen or Ci-Cg alkyl, (iii) 1- or 2-naphthyl, (iv) 2- or 3-benzofuranyl, (v) 2- or 3-' benzothiophenyl , (vi) 2- or 3-thienyl, (vii) 2-, 3- or 4-pyridyl, (viii) 2- or 3-furanyl r:\01ibrary\karchive\up-4500\4107nopc.doc X-6715A -7- (ix) 1, 3-benzodioxanyl, (x) substituted 1,3-benzodioxanyl ,\ (xi) quinolinyl, (xii) 2- or 3-indolyl or (xiii) N-substituted 2- or 3-indolyl; 1 is Ci-Cg alkyl, C1 - C alkylphenyl, hydrogen, phenyl or phenyl substituted with one or two substituents independently selected from CI, Br, F, I, C1-C4 alkyl, C1-C alkoxy, hydroxy, trifluoromethyl , - H2 - H(Ci-C4 alkyl), -N(Ci-C4 alkyl) 2 or Q1. - C4 alkylthio; R2 and R3 are each hydrogen or when taken together form a bond; R4 and R5 are each hydrogen or when taken together are =S, or when one of R4 and R5 is hydrogen, the other is -SCH3 ; R6 is hydrogen, Ci-Cg alkyl, C3-Ca cycloalkyl, C2-C6 alkenyl, .-SO2CH3 or -(CH2)p-Y where p is 0, 1, 2, or 3 and Y is O II cyano, OR8' -CR^- tetrazolyl, -NR10R1:L, -SH, C1-C4 alkylthio or alkyl) O II where R8 is hydrogen, C3.-C4 alkyl, or -C-C1-C4 alkyl; R9 is hydrogen, C1-C4 alkyl or H2; and R10 and R11 are each independently hydrogen, Ci-Cs alkyl, C2-C6 alkenyl, -(CH2)qOH, - (CH2)qN(d-C4 alkyl)2, - (CH2) qS ( C1-C4 alkyl) , C2. Ce alkynyl, phenyl, or C1-C4 alkylphenyl, where q is 1 to 6, both inclusive, or R10 and R11, taken together with the nitrogen atom to which they are attached, form a morpholinyl, piperidinyl, piperazinyl or N-methylpiperazinyl ring; and m is 0, 1, or 2; with the provisos that r:\0library\karchive\up-4500\4107nopc.doc X-6715A -8- when Ar is (i) phenyl, (ii) phenyl substituted with from one to three substituents selected from Ci-Ce alkyl, Ci-Ce alkoxy, F, Cl, trifluoromethyl, phenoxy, Ci-C4 alkyloxyphenyl , Ci-Cg alkylthio, N02, -N(R7)2 or -COOR7, where each R7 is independently hydrogen or ^-CQ alkyl, (iii) 1- or 2-naphthyl, (iv) 2- or 3-benzofuranyl, (v) 2- or 3-benzothiophenyl , (vi) 2- or 3-thienyl, (vii) 2- or 3-indolyl, (viii) 2- or 3- furanyl, (ix) quinolinyl or (x) 2-, 3- or 4-pyridyl; R1 is hydrogen or Ci-C6 alkyl; R2 and R3 taken together form a bond; m is 0; and R4 and R5 taken together are =S, R6 must be other than hydrogen or Ci-Cs alkyl; when Ar is phenyl; R1 is hydrogen, methyl or ethyl; R2 and R3 taken together form a bond; m is 0; R4 and R5 taken together are =S; R6 must be other than phenyl or Ci-C alkylphenyl; Ar cannot be phenyl substituted solely with one chloro substituent at the 4 -position of the phenyl ring; when Ar is phenyl substituted with two ethoxy moieties at the 3- and 4-positions of the phenyl ring, R1 must be hydrogen,- Ar cannot be phenyl substituted with a COOH moiety at the 2 -position of the phenyl ring; and when R4 and R5 are each hydrogen R6 cannot be Ci-Cg alkyl, and the pharmaceutically acceptable salts thereof. These compounds may be employed, for example, in the invention of application no. 106877.
Other objects, features and advantages of the present invention will become apparent from the subsequent description and the appended claims.
In addition to the genus of novel compounds described by formula II, above, certain other of the compounds which can be employed in the methods of the invention of application no. 106877 also appear to be r:\01ibrary\karchive\up-4500\4107nopc.doc X-6715A -9- novel. These compounds, while structurally similar to compounds specifically known in the art (see, for example, European Patent Application Nos. 343643, 391644 and 39817 as well as U.S. Patent No. 4,552,891), are not actually described in any of those patents or applications . As such, the present invention also encompasses the following novel compounds and their pharmaceutically acceptable salts : 5- [ (2-nitrophenyl) methylene] -2-thioxo-4- thiazolidinone; 5- [ (4 -fluorophenyl) methylene] -2-thioxo-4- thiazolidinone; 5- [ (2-thienyl) methylene] -2-thioxo-4- thiazolidinone ; 5- [ ( 2 -furanyl) methylene] -2-thioxo-4- thiazolidinone ; 5- [ (3 , 4 , 5-trimethoxyphenyl) methylmethylene] -2- thioxo-4 -thiazolidinone; 4- [ (2 -thioxo-4-thiazolidinone) methylene] benzoic acid; 5- [ (3 -hydroxy-4-nitrophenyl) methylene] -2- thioxo-4-thiazolidinone; 5- [ (3 -hydroxyphenyl) methylmethylene] -2-thioxo- -thiazolidinone; 5- [ (3 -methoxy-4-pentoxyphenyl) methylene] -2- thioxo-4 -thiazolidinone ; 5- [ (3 -hydroxy-4-ethoxyphenyl) methylene] -2- thioxo-4-thiazolidinone ; 5- [ (4 -pentoxyphenyl) methylene] -2-thioxo-4- thiazolidinone; 5 - [ (3 -ethoxy-4-propoxyphenyl) methylene] -2- thioxo-4 -thiazolidinone ; 5- [ ( 3 -propoxy-4-ethoxyphenyl) methylene] -2- thioxo- -thiazolidinone ; r:\01ibrary\karchive\up-4500\4107nopc.doc X-6715A - 10- 5- [ (3 , -dipropoxyphenyl) methylene] -2-thioxo-4- thiazolidinone ; 5- [ [3- (methyloxyphenyl) henyl] methylene] -2- thioxo- -thiazolidinone ; 5 - [ [3 , 5 -bis (1, 1-dimethylethyl) -4 -hydroxy- phenyl] methylene] -4-oxo-2-thioxo-3-thiazolidine acetic acid; 5- [ (3 , 5-dichloro-4-hydroxyphenyl) methylene] -2- thioxo-4-thiazolidinone; 5- [ (3 -ethoxy-4-butoxyphenyl) methylene] -2- thioxo-4 -thiazolidinone ; 5- [ (3 -ethoxy-4-methoxyphenyl) methylene] -2- thioxo- -thiazolidinone ; 5- [ [3 , 5-bis (1-methylpropyl) -4- hydroxyphenyl] methylene] - -oxo-2 -1hioxo-3 - thiazolidine acetic acid; 5- [ (4 -b toxyphenyl) methylene] -2-thioxo-4- thiazolidinone; 5- [ (3 -methoxy-4 -pentoxyphenyl) methylene] -2- thioxo-3 -methyl-4 -thiazolidinone ; 5- [ (3-methoxy-4-octoxyphenyl) methylene] -2- thioxo-4 -thiazolidinone ; 5- [ (3 , 5 -dimethoxy-4 -pentoxyphenyl) methylene] -2- thioxo- -thiazolidinone; 5- [ [3- (1, 1-dimethylethyl) -4-hydroxy-5- (methylthiophenyl) phenyl] methylene] -2-thioxo-4- thiazolidinone ; 5- [ [3-ethoxy-4-hydroxy-5- (methylthiophenyl) - phenyl] methylene] -2-thioxo-4-thiazolidinone; 5- [ [3 -ethoxy-4 -hydroxy-5 - (methylthiophenyl) - phenyl] methylene] -2-thioxo-3 -methyl-4-thiazolidinone ; r\01ibrar \karchive\up- 500\4107nopc.doc X-6715A -11- 5- [ [3-ethoxy-4-hydroxy-5- (methylthiophenyl) - phenyl] methylene] -4-oxo-2-thioxo-3-thiazolidine acetic acid.
Certain of the above compounds and, in particular, 5- [ ( -pentoxyphenyl) methylene] -2-thioxo-4-thiazolidinone; 5- [ (3 -propoxy- -ethoxyphenyl) methylene] -2-thioxo-4-thiazolidinone; 5- [ (3 -ethoxy-4-butoxyphenyl) -methylene] -2-thioxo-4-thiazolidinone; 5- [ (4-butoxyphenyl) methylene] -2-thioxo-4-thiazolidinone 5- [ (3-methoxy-4 -pentoxyphenyl) methylene] -2-thioxo-4-thiazolidinone; 5- [ (3 , 5-bis (1 , 1-dimethylethyl) -4-hydroxyphenyl] methylene] -4-oxo-2-thioxo-3 -thiazolidine acetic acid; and 5- [ [3 , 5-bis (1-methylpropyl) -4-hydroxy-phenyl] methylene] -4-oxo-2-thioxo-3-thiazolidine acetic acid (especially the latter three compounds) , appear to possess a surprising ability to lower blood glucose levels in mammals compared to structurally similar compounds known in the art. Because of such surprising activity, these compounds are particularly preferred compounds of the present invention.
In addition, 5- [ [3- (1, 1-dimethylethyl) -4-hydroxy-5- (methylthiophenyl) phenyl] methylene] -2-thioxo-4-thiazolidinone appears to possess a surprising ability to inhibit cathepsin D levels compared to structurally similar compounds known in the art. Because of such surprising activity, such compound is also a particularly preferred compound of the present invention.
Finally, the present invention also provides pharmaceutical formulations comprising a compound of the present invention, or a pharmaceutically acceptable salt thereof, in combination with one or more pharmaceutically acceptable carriers, diluents or excipients therefor.
As used herein, the term "Ci-Cs alkyl" represents a straight or branched alkyl chain having. from r.\01ibrary\karchive\up-4500\4107nopc.doc X-6715A -12- one to eight carbon atoms. Typical Ci-Cs alkyl groups include methyl, ethyl, n-propyl, isopropyl, n-butyl, sec-butyl, isobutyl, t-butyl, n-pentyl, and the like. The term "Ci-Cs alkyl" includes within its definition the terms "C1-C4 alkyl" and "C1-C6 alkyl".
"C1-C4 alkylphenyl" represents a straight or branched chain alkyl group having from one to four carbon atoms attached to a phenyl ring. Typical C1-C4 r:\0Iibrar \karchive\up-4500\4107nopc.doc X-6715A -13- alkylphenyl groups include methylphenyl , ethylphenyl , n-propylphenyl , isopropylphenyl , n-butylphenyl, isobutylphenyl , and tert-butylphenyl .
The term "C1-C4 alkylthiophenyl " represents a straight or branched chain alkyl group having from one to four carbon atoms attached to a thiophenyl moiety.
Typical CX-C4 alkylthiophenyl groups include methylthiophenyl, ethylthiophenyl , isobutylthiophenyl and the like.
In a similar fashion, the term "C1-C4 alkyloxyphenyl " represents a straight or branched chain alkyl group having from one to four carbon atoms attached to phenoxy moiety. Typical C1-C4 alkyloxyphenyl groups include methyloxyphenyl , ethyloxyphenyl, propyloxyphenyl and the like. ' "C1-C8 alkoxy" represents a straight or branched alkyl chain having one to eight carbon atoms, . which chain is attached to the remainder of the molecule by an oxygen atom. Typical C1-C8 alkoxy groups include methoxy, ethoxy, propoxy, isopropoxy, butoxy, isobutoxy, sec-butoxy, tert-butoxy, pentoxy, hexoxy, heptoxy, and the like. The term "Ci-Cs alkoxy" includes within its definition the term "C1-C4 alkoxy".
"Ci-Ce alkylthio" represents a straight or branched alkyl chain having one to eight carbon atoms, which chain is attached to the remainder of the molecule by a sulfur atom. Typical Ci-Cs alkylthio groups include methylthio, ethylthio, propylthio, butylthio, tert-butylthio, octylthio and the like. The term "Ci-Cg alkylthio" includes within its definition the term "Οχ- C4 alkylthio" .
The term "C2-C6 alkenyl" refers to straight and branched chain radicals of two to six carbon atoms, both inclusive, having a double bond. As such, the term r.\01ibrar \karchive\up- 500\4107nopc.doc X-6715A - 14- includes ethylene, propylene, 1-butene, 2-butene, 2-methyl-l-propene, 1-pentene, 2 -methyl -2-butene and the like .
The term " 2~ Q alkynyl" refers to straight and branched chain radicals of two to six carbon atoms, both inclusive, having a triple bond. As such, the term includes acetylene, propyne, 1-butyne, 2-hexyne, 1-pentyne, 3 -ethyl -1-butyne and the like.
The term "C3-C8 cycloalkyl" refers to saturated alicyclic rings of three to eight carbon atoms, both inclusive, such as cyclopropyl, methylcyclopropyl, cyclobutyl, cyclopentyl, cyclohexyl, cyclooctyl and the like.
The terms "1, 3-benzodioxanyl" and "substituted 1, 3-benzodioxanyl" refer to structures of the formulae where each R is independently hydrogen or C1-C4 alkyl .
"Quinolinyl" refers to a quinoline ring system which is attached to the rest of the molecule at the 4, 5, 6, 7 or 8 position of such ring system.
"N-substituted 2- or 3- indolyl" refers to a 2- or 3- indolyl ring system substituted on the nitrogen atom of that ring system with a C]_-C6 alkyl, C1-C4 alkylphenyl, or C3-C8 cycloalkyl group.
The term "pharmaceutically acceptable salts" refers to salts of the compounds of the above formulae which are substantially non-toxic to living organisms.
Typical pharmaceutically acceptable salts include those salts prepared by reaction of the compounds of the above formulae with a pharmaceutically acceptable mineral or organic acid, or a pharmaceutically acceptable alkali r:\0librar \karchive\up-4500\4107nopc.doc X-6715A - 15 - metal or organic base, depending on the types of substituents present on the compounds of the formulae.
Examples of pharmaceutically acceptable mineral acids which may be used to prepare pharmaceutically acceptable salts include hydrochloric acid, phosphoric acid, sulfuric acid, hydrobromic acid, hydroiodic acid, phosphorous acid and the like.
Examples of pharmaceutically acceptable organic acids which may be used to prepare pharmaceutically acceptable salts include aliphatic mono and dicarboxylic acids, oxalic acid, carbonic acid, citric acid, succinic acid, phenyl -substituted alkanoic acids, aliphatic and aromatic sulfonic acids and the like. Such pharmaceutically acceptable salts prepared from mineral or organic acids thus include hydrochloride, hydrobromide , nitrate, sulfate, pyrosulfate, bisulfate, sulfite, bisulfite, phosphate, monohydrogenphosphate, . dihydrogenphosphate, metaphosphate, pyrophosphate, hydroiodide, hydrofluoride, acetate, propionate, formate, oxalate, citrate, lactate, p-toluenesulfonate, methanesulfonate, maleate, and the like.
Many compounds of formulae I, la or II which contain a carboxy, carbonyl, hydroxy or sulfoxide group may be converted to a pharmaceutically acceptable salt by reaction with a pharmaceutically acceptable alkali metal or organic base. Examples of pharmaceutically acceptable organic bases which may be used to prepare pharmaceutically acceptable salts include ammonia, amines such as triethanolamine, triethylamine , ethylamine, and the like. Examples of pharmaceutically acceptable alkali metal bases included compounds of the general formula n\0librar \karchive\up-4500\4107nopc.doc X-6715A - 16- MOR13, where M represents an alkali metal atom, e.g. sodium, potassium, or lithium, and R13 represents hydrogen or C1-C4 alkyl .
It should be recognized that the particular anion or cation forming a part of any salt of this invention is not critical, so long as the salt, as a whole, is pharmacologically acceptable and as long as the anion or cationic moiety does not contribute undesired qualities.
A preferred genus of compounds, which are useful, for example, in reducing blood glucose concentrations includes those compounds wherein Ar, R1, R2, R3 , m, R4, and R5 are as set forth for formula I, and R6 is hydrogen, C1-C6 alkyl, C3-C8 cycloalkyl, C2-C6 alkenyl, -SO2CH3 or -(CH2)p-Y where 0 II p is 0, 1, 2, or 3 and Y is cyano, -OR8, -CR9, tetrazolyl, R10R1:L, -SH, '-S(CI-C4 alkyl), or aIkyI) 0 II where R8 is hydrogen, C1-C4 alkyl, or -C-C1-C4 alkyl, R9 is hydrogen, C1-C4 alkyl, or NH2; and R10 and R11 are each independently hydrogen, Ci-Cg alkyl, C2-C6 alkenyl, C2-C6 alkynyl, phenyl, C1-C4 alkylphenyl, -(CH2)qOH, -(CH2)qN(Ci-C4 alkyl) 2, or - (CH2) qS (C1-C4 alkyl) where q is 1 to 6, both inclusive, or R1^ and R11' taken together with the nitrogen atom to which they are attached, form a morpholinyl, piperidinyl, piperazinyl, or N-methylpiperazinyl ring .
Of this preferred genus, those compounds in which m is 0 are more preferred.
Of this more preferred genus, those compounds in which R4 and R5 taken together are =S are even more preferred.
Of this even more preferred genus, those compounds in which R1 is hydrogen are especially preferred. r\01ibrary\karchive\up-4500\4107nopc.doc X-6715A -17- Of this especially preferred genus, those compounds in which R6 is hydrogen, C1-C6 alkyl, C2-C6 alkenyl, or - (CH2)p~Y where p is 0, 1, 2, or 3 and Y is 0 II -OR8, -CR9, -NR10R1:L, or C1-C4 alkylthio, where R8 is O II hydrogen, C1-C4 alkyl or -C-C1-C4 alkyl, R9 is hydrogen, Ci-C4 alkyl or H2; and R10 and R11 are each independently hydrogen, Ci-Cg alkyl, C2-C6 alkenyl, C2-C6 alkynyl-, phenyl, or C1-C4 alkylphenyl are particularly preferred.
Of this particularly preferred genus, those compounds in which R6 is hydrogen, Ci-Cg alkyl, or C2-C5 alkenyl are more particularly preferred.
Of this more particularly preferred genus, those compounds in which Ar is (i) phenyl, (ii) phenyl substituted with from one to three substituents independently selected from Ci-Cs alkyl, Ci-Cs alkoxy, Ci-Ca alkylthio, trifluoromethyl, C1-C4 alkylphenyl, phenyl, O2 " F, CI, hydroxy, phenoxy, (-1-C4 alkyloxyphenyl, thiophenyl, C!-C4 alkylthiophenyl, -COOR7, -N(R7)S02R7 or -N(R7)2, where each R7 is independently hydrogen or Ci-Cg alkyl, (iii) 2-, 3- or 4-pyridyl, or (iv) 2- or 3- furanyl are substantially preferred.
Of this substantially preferred genus, those compounds wherein Ar is phenyl substituted with from one to three substituents independently selected from Ci-Cs alkyl, Ci-Ca alkoxy, C1-C4 alkylphenyl, phenyl, O2, F, Cl, hydroxy, phenoxy, C1-C4 alkylthiophenyl , -COOR7 or - (R7)S02 7, where each R7 is independently hydrogen or Ci-Ce alkyl, are more substantially preferred.
Of this more substantially preferred genus, those compounds wherein Ar is phenyl substituted with from one to three substituents independently selected from Ci-Cs alkyl r:\01ibrary\karchive\up-4500\4107nopc.doc X-6715A - 18- (especially C1-C4 alkyl) , Ci-Cs alkoxy (especially Ci-C6 alkoxy) , or hydroxy are even more substantially preferred.
The most preferred compounds of the present invention which may be employed, for example, as medicaments for reducing blood glucose concentrations include 5- [(3,4-diethoxyphenyl) methylene] -2-thioxo-4-thiazolidinone; 5- [ (3-methoxy-4-butoxyphenyl) methylene] -2-thioxo-4-thiazolidinone ; 5- [ (3 -methoxy-4-pentoxy-phenyl) methylene] -2-thioxo-4-thiazolidinone; 5- [ (3 -methoxy-4-pentoxyphenyl) methylene] -2-thioxo-4-thiazolidinone, sodium salt; 5- [ (3-methoxy-4-pentoxyphenyl) methyl] -2-thioxo-4-thiazolidinone; 5 [ [3,5-bis (1, 1-dimethylethyl) -4-hydroxyphenyl] methylene] -2-thioxo-4-thiazolidinone; 5 [ (3 , 5-dimethyl-4-hydroxyphenyl) methylene] -2-thioxo-4-thiazolidinone and 5- [ (3 , 5-dimethoxy-4-hydroxyphenyl) methylene] -2-thioxo-4-thiazolidinone .
A preferred genus of compounds useful as medicaments for treating Alzheimer's disease includes those compounds wherein Ar, R1, R2, R3, m, R4 and R5 are as set forth for formula la, and R6 is hydrogen, Ci-Cg alkyl or -(CH2)pY where p is 0, 1, 2 or 3 and Y is -NR10R1:L where R10 and R11 are each independently hydrogen, alkyl, phenyl or C1-C4 alkylphenyl.
Of this preferred genus, those compounds in which m is 0 are more preferred.
Of this more preferred genus, those compounds in which R4 and R5 taken together are =S are even more preferred.
Of this even more preferred genus, those compounds in which R2 and R3 taken together form a bond are especially preferred.
Of this especially preferred genus, those compounds in which Ar is phenyl substituted with from one to three substituents independently selected from Ci-Cg alkyl, Ci-Ca alkoxy, Ci-Ce alkylthio, trifluoromethyl, C1-C4 r.\01ibrar \karchive\up-4500\4107nopc.doc X-6715A -19- alkylphenyl, phenyl, NO2, F, Cl, hydroxy, phenoxy, C1-C4 alkyloxyphenyl , thiophenyl, C1-C4 alkylthiophenyl, -COOR7, -N(R7)S02R7 or -N(R7)2, where each R7 is independently hydrogen or C1-C alkyl, are particularly preferred.
Of this particularly preferred genus, those compounds in which R1 is hydrogen are more particularly preferred.
Of this more particularly preferred genus, those compounds in which Ar is phenyl substituted with from one to three substituents independently selected from phenoxy, phenyl, C -Ca alkoxy, Ci-C8 alkyl (especially C1-C4 alkyl) , hydroxy, Cl, F, C1-C4 alkylthiophenyl, -N(R7)S02R7 and -N.(R7)2, where each R7 is independently hydrogen or Ci-Cg alkyl, are substantially preferred.
The most preferred compounds which may be employed, for example, as medicaments for treating Alzheimer's disease according to the invention of application no. 106877 include" 5- [ (4-phenoxyphenyl) -methylene] -2-thioxo-4-thiazolidinone; 5- [ (3-phenoxyphenyl) methylene] -2-thioxo-4-thiazolidinone; 5- [[(1,1' -biphenyl) -4-yl] methylene] -2-thioxo-4-thiazolidinone; 5- [ (3 -methoxy-4-hexoxyphenyl) methylene] -2-thioxo-4-thiazolidinone; 5- [ (3-methoxy-4-heptoxyphenyl) methylene] -2-thioxo-4-thiazolidinone; 5- [ (3-methoxy-4-octoxyphenyl] meth-ylene] -2-thioxo-4-thiazolidinone; 5- [ [3 , 5-bis (1, 1-dimethylethyl) -4 -hydroxyphenyl] methylene] -2-thioxo-4-thiazolidinone; 5- [ (3 , 5 -dichloro-4 -hydroxyphenyl) methylene] -2-thioxo-4-thiazolidinone; 5- [ [3- (1, 1-dimethyleth l) -4-hydroxy-5- (methylthiophenyl) phenyl] methylene] -2-thioxo-4-thiazolidinone; and 5- [ [4- (dimethylamino) phenyl] -methylene] - 2-thioxo-4-thiazolidinone .
A preferred genus of compounds of the present invention includes those compounds wherein Ar, R1, R2, R3, R4, R5 and R6 are as set forth for Formula II, and m is 0. r.\0library\karchive\up-4500\4107nopc.doc X-6715A -20- Of this preferred genus, those compounds in which R4 and R5 taken together are =S are more preferred. Of this more preferred genus, those compounds in which R2 and R3 taken together form a bond are especially preferred.
Of this especially preferred genus, those compounds in which R6 is hydrogen, Ci-Cg alkyl, C2-C5 alkenyl, or -(CH2)p-Y where p is 0, 1, 2, or 3 and Y is O II -OR8, -CR9, -NR10R1:L, or C1-C4 alkylthio, where R8 is O II hydrogen, C1-C4 alkyl or -C-C1-C4 alkyl, R9 is hydrogen, Ci-C4 alkyl or NH2; and R10 and R11 are each independently hydrogen, C1-C6 alkyl, C2-Cg alkenyl, C2-C6 alkynyl, phenyl, or C1-C4 alkylphenyl are particularly preferred.
Of this particularly preferred genus, those compounds in which R6 is hydrogen, C -CQ alkyl, or C2-C6 alkenyl are more particularly preferred. Of this more, particularly preferred genus, those compounds in which R1 is hydrogen or phenyl are even more particularly preferred.
Of this even more particularly preferred genus, those compounds in which Ar is (i) phenyl, (ii) phenyl substituted with from one to three substituents independently selected from Ci-Cs alkyl, Ci-Cs alkoxy, C^-CQ alkylthio, trifluoromethyl, C2-C4 alkylphenyl, N02, F, Cl, phenoxy, C1-C4 alkoxyphenyl , thiophenyl, C1-C4 alkylthiophenyl, -COOR7, -N(R7)S02R7 or -N(R7)2, where each R7 is independently hydrogen or Ci-Cg alkyl, (iii) 1,3-benzodioxanyl, (iv) substituted 1 , 3 -benzodioxanyl or (v) quinolinyl are substantially preferred compounds.
Of this substantially preferred genus, those compounds wherein Ar is (i) phenyl substituted with from one to three of phenoxy or -N(R7)S02R7' where each R7 is hydrogen or C1-C6 alkyl or (ii) 1 , 3 -benzodioxanyl are more substantially preferred. r\01ibrary\karehive\up-4500\ 107nopc.doc X-6715A -21 - Certain preferred compounds of the present invention include 5- (diphenylmethylene) -2-thioxo-4-thiazolidinone; 5- [ (1, 3 -benzodioxol-5-yl) methylene) -2-thioxo-4-thiazolidinone; 5- [ ( -phenoxyphenyl) methylene] -2-thioxo-4-thiazolidinone; 5- [ (3 -methoxy-4-heptoxyphenyl) methylene] -3 -amino-2-'thioxo-4-thiazolidinone ; 5- [ (3 -methoxy-4-heptoxyphenyl) methylene] -3-dimethylamino-2-thioxo-4-thiazolidinone; and 5[(3-methanesulfonamidophenyl) methylene] -2-thioxo-4-thiazolidinone .
An alternative preferred genus of compounds of the present invention includes those compounds wherein Ar, R1, R2, R3 , R4, R5, and m are as defined for formula II, and R6 is C3-C8 cycloalkyl, C2-C6 alkenyl, -SO2CH3 or -(CH2)p-Y 0 I' where p is 0, 1, 2, or 3 and Y is cyano, -OR8, -CR9, tetrazolyl, -NR10R1:L, -SH, C1-C4 alkylthio, or alkyI) O II where R8 is hydrogen, C1-C4 alkyl, or -C-C1-C4 alkyl; R9 is hydrogen, C1-C4 alkyl, or H2; and R10 and R11 are each independently hydrogen, Ci-Cg alkyl, C2 - Q alkenyl, C2-C6 alkynyl, phenyl, C1-C4 alkylphenyl, -(CH2)qOH, -(CH2)qN(Ci-C4 alkyl) 2, or - (CH2) gS (C1-C4 alkyl) where q is 1 to 6, both inclusive, or R10 and R11' taken together with the nitrogen atom to which they are attached, form a morpholinyl, piperidinyl, piperazinyl, or N-methylpiperazinyl ring.
Of this preferred genus, those compounds in which m is 0 are more preferred.
Of this more preferred genus, those compounds in which R4 and R5 taken together are =S are even more preferred. r:\01ibrai karchive\up-4500\4107nopc.doc X-6715A -22- Of this even more preferred genus, those compounds in which R2 and R3 taken together form a bond are especially preferred.
Of this especially preferred genus, those compounds in which R5 is 2~ Q alkenyl, or -(CH2)p-Y where p is 0, 1, 2, O II or 3 and Y is -OR8, -CR9, -NR10R1:l, or C1-C4 alkylthio, O II where R8 is hydrogen, C]_-C4 alkyl or -C-C1-C4 alkyl, R9 is hydrogen, C1-C4 alkyl or NH2; and R10 and R11 are each independently hydrogen, Ci-Cg alkyl, C2-C6 alkenyl, C2-C6 alkynyl, phenyl, or C1-C4 alkylphenyl are particularly preferred.
Of this particularly preferred genus, those compounds wherein R1 is hydrogen or phenyl are more particularly preferred.
Of this more particularly preferred genus, those compounds in which Ar is (i) phenyl, (ii) phenyl substituted with from one to three substituents independently selected from C1-C8 alkyl, Ci-Cs alkoxy, C -C8 alkylthio, trifluoro-methyl, C2-C4 alkylphenyl, NO2, F, Cl, phenoxy, C1-C alkoxyphenyl , thiophenyl, C1-C4 alkylthiophenyl , -COOR7, -N(R7)S02R7 or -N(R7)2/ where each R7 is independently hydrogen or Ci-Cg alkyl (iii) 2-, 3- or 4-pyridyl, or (iv) 2- or 3- furanyl are even more particularly preferred.
Of this even more particularly preferred genus, those compounds wherein Ar is phenyl substituted with from one to three substituents independently selected from Ci-Cg alkyl, Ci-Cs alkoxy, Cx-Ca alkylthio, trifluoromethyl , C2-C4 alkylphenyl, NO2, F, Cl, phenoxy, C1-C4 alkoxyphenyl, thiophenyl, C1-C4 alkylthiophenyl, -COOR7' -N(R7)S02R7 or -N(R7)2, where each R7 is independently hydrogen or Ci-Cg alkyl, are substantially preferred. r:\0library\karchive\up- 500\4107nopc.doc X-6715A -23- 119119/2 Of this substantially preferred genus, those compounds wherein Ar is phenyl substituted with from one to three substituents independently selected from C1-C8 alkyl or C]_-C8 alkoxy are most preferred.
The present invention also encompasses formulations comprising a compound of the present invention in combination with a pharmaceutically acceptable carrier, diluent, or excipient therefor.
Preferred formulations of the present invention are those formulations which contain a preferred compound or genus of compounds of the present invention, as described above .
The compounds of the present invention, as well as the compounds employed according to the invention of application no. 106877, can, typically, be prepared by methods well known to one skilled in the art of organic chemistry. For example, such compounds may be prepared by condensation of rhodanine, or an appropriately substituted rhodanine derivative, with an appropriately substituted aromatic aldehyde or aldehyde derivative such NH II as a mono or disubstituted imine of the formula Ar-C-H or NH II Ar-C-R1 . Such reaction is illustrated utilizing an appropriately substituted aromatic aldehyde as follows where Ar and are as defined in formulae I, la and II.
Compounds of the present invention (as well as those compounds employed according to the invention of r:\0library\karchive\up-4500\4107nopc.doc X-6715A -24- application no. 106877) wherein R2 and R3 are hydrogen, or when taken together form a bond, and R4 and R5 are each hydrogen can be prepared by subjecting the compound wherein R4 and R5 taken together form =S to catalytic hydrogenation. The relative proportions of compound obtained (R2, R3, R4 and R5 all hydrogen vs. R2 and R3 taken together form a bond and R4 and R5 are hydrogen) depends upon the temperature, pressure, and duration of hydrogenation, the solvent employed and the particular catalyst used. Alternatively, the above transformations may be accomplished by heating the compounds wherein R4 and R5 taken together are =S and R2 and R3 taken together are a double bond in a mixture of hydrochloric acid and an alcohol, such as ethanol, in the presence of zinc.
Reduction of the thione without affecting the benzylic double bond may be accomplished by heating the thione with a reducing agent such as tri-n-butyl tin hydride in -a non-reactive solvent, such as toluene, and preferably in the presence of a free radical initiator, such as azobisisobutyronitrile . However, for such reduction to work, an N-substituted rhodanine substrate must be employed.
The transformation of compounds wherein R2 and R3 taken together form a bond and R4 and R5 taken together are =S to those compounds wherein R2 and R3 are both hydrogen while R4 and R5 remain unchanged may be accomplished by treating the unsaturated compound with a dihydropyridine, such as diethyl 2, 6-dimethyl-1,4-dihydro-3 , 5-pyridine dicarboxylate in the presence of silica gel. The reaction is best carried out in the presence of a nonreactive solvent such as benzene or toluene, preferably under an inert atmosphere. The r\01ibraiy\karehive\up-4500\4107nopc.doc X-6715A -25- reaction may be accomplished at temperatures from about 25°C up to the reflux temperature of the mixture. At the preferred temperature of approximately 80°C, the reaction is essentially complete after about 12-18 hours.
Compounds of formulae I, la or II wherein R1 is I-CQ alkyl, phenyl, a substituted phenyl of the type described above, or C1-C4 alkylphenyl may be prepared by conventional Friedel-Crafts acylation of an appropriately substituted aromatic compound with an acyl halide of the formula R!-CCO-X, wherein R1 is as defined in formulae I or II and X is chloro, fluoro, bromo or iodo. The resulting aromatic ketone is then condensed with rhodanine, or an appropriately substituted rhodanine derivative.
The compounds of the present invention (as well as the compounds employed according to the invention of application no. 106877) allow various R6 substituents.
These R6 substituents can be prepared as follows.
Compounds of formulae I, la and II wherein R^ is hydrogen, C1-C6 alkyl, C3-C8 cycloalkyl or -(CH2)p-Y where p is as defined for formulae I, la and II and Y is cyano, or NR10R1:L where R10 and R11 are each independently hydrogen or C1-C6 alkyl may be prepared using the method set forth in the above reaction scheme.
Alternatively, rhodanine may be used for condensation with an aldehyde or aldehyde derivative forming those species wherein R^ is hydrogen, followed by alkylation or acylation with the appropriate R^-containing halide. The alkylation or acylation is usually accomplished in an inert solvent such r\0Iibrary\karehive\up-4500\4107nopc.doc X-6715 -26- as tetrahydrofuran or dimethylformamide and in the presence of a strong base such as sodium hydride.
Alternatively, compounds of formulae I, la and II wherein R6 is -(CH2)p-Y where Y is cyano may be prepared by treating the non-cyanated analog with a halo-substituted aliphatic nitrile. From this cyano derivative the tetrazolyl is prepared as by treatment with tri-N-butyl tin azide in, for example, ethylene glycol dimethyl ether.
Compounds of formulae I, la and II wherein is -(CH2)p-Y (p=0) and Y is NR!OR11, where R10 and R11 are as defined in formulae I, la and II, may also be prepared by employing an appropriately substituted hydrazine. In this reaction sequence, benzaldehyde is reacted with an appropriately substitued hydrazine, in an alcoholic solvent, yielding III. An appropriately substituted alkyl halide is the reacted with III, in the presence of triethylamine and acetonitrile, to provide IV, which is then further reacted with hydrazine to yield the R^® , R^ hydrazine V. Compound V may alternatively be prepared by the reduction of a nitroso-R^-^RH amine using zinc dust and acetic acid or aluminum and a strong base. The R^O, R^-hydrazine is then treated with carbon disulfide, chloroacetic acid and triethylamine to provide intermediate VI. Condensation of VI with an appropriately substituted aromatic aldehyde or aldehyde derivative yields the desired product, as represented by the following reaction scheme.
X-6715 -27- (IV) (V) (VI) Fxirthermore, the thione portion of the compound produced above may be reduced by treatment with a reducing agent such as tri-n-butyltin hydride in an inert solvent such as toluene, preferably in the presence of a free radical initiator such as azobisisobutyronitrile.
Preparation of compounds wherein one of RIO and is X-6715 ■28- hydrogen may be effected before or after reduction of the thione, as desired, by heating the disubstituted compound in a mixture of ethanol/water in the presence of a catalyst such as a rhodium catalyst.
Compounds of formulae I, la and II wherein is -(CH2)p-Y and Y is OR8 or R10R1:L (where R8 is hydrogen, acetyl or tosyl and R1^1 and R^ are each independently hydrogen or Ci-Cg alkyl) may also be prepared according to the following reaction scheme: Ar-CHO /(CH2)n-OH (X) X-6715 -29- where Ts = Tosyl A hydroxyalkyl rhodanine is prepared by condensing carbon disulfide, chloroacetic acid, and the appropriate hydroxyalkylamine by standard techniques. When condensed with the appropriately substituted aromatic aldehyde (or aldehyde derivative) , as described above, the resulting product is the condensed 2-thioxo-4-thiazolidinone VIII which has been transformed into the acetyl derivative. The thioxo compound VIII may optionally be converted to the methylene compound of formulae I or II as described above. The acetyl group of intermediate IX may be removed upon treatment with aqueous ammonia in a solvent such as acetonitrile to provide compound X. The hydroxy compound X is then converted to the tosyl derivative upon treatment with p-toluenesulfonyl chloride in pyridine, preferably at temperatures of around 0°C. The versatile tosyl intermediate XI may then be transformed into the compounds of formulae I or II upon treatment with an appropriate HNR^OR11 amine. This latter transformation is best accomplished by allowing XI to react in the presence of a molar excess of the amine. Once again, a solvent such as acetonitrile is useful for accomplishing this transformation.
Those compounds where m is 1 or 2 are readily prepared from the sulfide (m=0) by treatment with an oxidizing agent, such as m-chloroperbenzoic acid, in a suitable solvent for a time sufficient to generate the desired oxidative state.
X-6715A -30- Depending upon the definitions of R1, R2, and R3, the compounds of formulae I, la and II may exist in various isomeric forms. The compounds, formulations and uses according to the present invention are not related to any particular isomer but include all possible isomers and racemates.
It will be readily appreciated by one skilled in the art that the aromatic portion of the compounds of the invention (or the compounds used as medicaments according to the present invention) can be provided by compounds which are either commercially available or may be readily prepared by known techniques from commercially available starting materials. Similarly, the rhodanine or N-substituted rhodanine starting material is either commercially available or may be prepared by well known' methods from commercially available substrates.
The following Examples illustrate the preparation of the compounds of the present invention, as well as compounds which may be according to the invention of application no. 106877. The Examples are illustrative only and are not intended to limit the scope of the instant invention in any way. Passages of the description which are not within the scope of the claims do not constitute part of the invention.
Example 1 5- [ (3 -methanesulfonamidophenyl) methylene] -2-thioxo-4-thiazolidinone Thirty seven grams (185.9 mmol) of 3-methanesulfonamidbenzaldehyde, 25.0 g (187.9 mmol) of rhodanine, 48.0 g (585.3 mmol) of anhydrous sodium acetate and 950 ml of acetic acid were stirred while heating at reflux for 20 hours. The reaction was then stirred at room temperature for approximately another 60 hours. The re'sulting slurry was poured into 3000 ml of a 1:1 ethanol/water mixture. Solids precipitated and were r\0library\karchive\up-4500\4107nopc.doc X-6715 -31- recovered by filtration, washed with water and then vacuum dried to provide 54 g of title compound, m.p. 260-265°C. Analysis for C1.1H10N2O3S3 Calculated: C, 42.02; H, 3.20; N 8.91; Found: C, 42.15; H, 3.57; N 8.71.
Example 2 5- [ (1, 3-benzodioxol-5-yl)methylene] -2-thioxo-4-thiazolidinone Twenty grams (133.2 mmol) of piperonal were reacted with 17.74 g (133.2 mmol) of rhodanine in 38.24 g (466.2 mmol) of glacial acetic acid at reflux for about 3 hours. The mixture was then poured into water and stirred overnight. A precipitate formed which was recovered by filtration and then air dried overnight to provide 27.8 g of title product, m.p. 194-195°C.
Analysis for C11H7N1O3S2: Calculated: C, 49.80; H, 2.66; N 5.28; S, 24.17; Found: C, 50.04; H, 2.38; N 5.27; S, 23.98.
Example 3 5-[ (4-quinolinyl) methylene] -2-thioxo-4-thiazolidinone Rhodanine (2.2 g; 16.5 mmol), 1.3 ml of concentrated ammonium hydroxide and 1 g of ammonium chloride in 20 ml of ethanol were heated on a steam bath for 15 minutes. 4-Quinoline carboxaldehyde (2.6 g; 16.5 mmol) was added and the resulting mixture was heated on the steam bath for another hour. Upon cooling to 5°C a precipitate formed. This precipitate was recovered by X-6715 -32- filtration and then washed with water to provide 4 g of title compound, m.p. 325-328°C.
Analysis for C13H8N2OS2: Calculated: C, 57.33; H, 2.96; N 10.29; Found: C, 57.11; H, 3.11; N 10.21.
Example 4 5- (diphenylmethylene) -2-thioxo-4-thiazolidinone One hundred and ninety grams (1.05 mol) of diphenyl ketimine, 140 grams (1.05 mol) of rhodanine, 5 ml of acetic acid and 1500 ml of toluene were heated at reflux for 3 hours. Crystals formed upon cooling. The solvent was decanted, fresh toluene was added to the residue and the resulting suspension was filtered. The recovered crystals were recrystallized from methanol to provide 172.0 g of title product, m.p. 192-194°C.
Analysis for C16H11NOS2: Calculated: C, 64.62; H, 3.73; 0, 5.38; N 4.71; S, 21.56; Found: C, 64.13; H, 3.84; 0, 5.57; N 4.59; S, 22.38.
Example 5 5- [ (4-phenoxyphenyl) methylene] -2-thioxo-4-thiazolidinone A mixture of 9.9 g (50.0 mmol) of 4-phenoxybenzaldehyde, 6.8 g (51.1 mmol) of rhodanine, 15.5 g of sodium acetate and 60 ml of acetic acid was heated on a steam bath for two hours. The reaction solution was then poured into water causing crude product to precipitate. The precipitate was filtered and then washed successively with water followed by diethyl ether to provide 8.6 g of title product, m.p. 195-200°C.
X-6715 -33- Analysis for C16H11NO2S2: Calculated: C, 61.32; H, 3.54; N 4.47; Found: C, 61.07; H, 3.63; N 4.47.
The following compounds were synthesized using methods substantially equivalent to those described in Examples 1-5 above or as described elsewhere herein.
Example 6 5- (phenylmethylene) -2-thioxo-4-thiazolidinone, m.p. 202-203.5°C Example 7 5- [ (2-hydroxyphenyl) methylene] -2-thioxo-4-thiazolidinone, m.p. 220-222°C Example 8 5- [ (4-hydroxyphenyl) methylene] -2-thioxo-4-thiazolidinone, m.p. 287-290°C Example 9 5- [ (2-nitrophenyl) methylene] -2-thioxo-4-thiazolidinone, m.p. 197.5-199°C Example 10 5- [ (3 -nitrophenyl) methylene] -2-thioxo-4-thiazolidinone, m.p. 277-280°C Example 11 5-[ (3 -hydroxyphenyl) methylene] -2-thioxo-4-thiazolidinone, m.p. 242-244°C Example 12 5- [ (2, 4-dimethoxyphenyl) methylene] -2-thioxo-4-thiazolidinone, m.p. 253-255°C X-6715 -34- Example 13 5- [ (4-fluorophenyl) methylene] -2-thioxo-4-thiazolidinone, m.p. 225-227°C Example 14 5- [ (2 -thienyl) methylene] -2-thioxo-4-thiazolidinone, m.p. 231-233°C Example 15 5- [ (2-furanyl) methylene] -2-thioxo-4-thiazolidinone, m.p. 217-219°C Example 16 5- [ ( 4-pyridyl) methylene] -2-thioxo-4-thiazolidinone, m.p. 297-298°C Example 17 5- [ (3 , 4, 5-trimethoxyphenyl) methylene] -2-thioxo 4-thiazolidinone, m.p. 203-205°C Example 18 5-[ ( 4-methoxyphenyl ) methylene] -2-thioxo-4-thiazolidinone, m.p. 252-254°C Example 19 5- [ (3 , 4, 5-trimethoxyphenyl) methylmethylene] -2-thioxo-4-thiazolidinone, m.p. 210-213°C Example 2Q 5- [ (3 -methoxy-4-hydroxyphenyl) methylene] -2-thioxo-4-thiazolidinone, m.p. 229-231°C X-6715 -35- Exaimole 21 5- [ (4-methoxyphenyl)phenylmethylene] -2-thioxo 4-thiazolidinone, m.p. 169-171°C Example 22 5-[ (3 -pyridyl) methylene] -2-thioxo-4-thiazolidinone, m.p. ~286°C Example 23 5- [ (3 -chlorophenyl)methylene] -2-thioxo-4-thiazolidinone, m.p. 233-235°C Example 24 5-[ (2 ,3-dimethoxyphenyl) methylene] -2-thioxo-4 thiazolidinone xam le 25 5- [ (3 -methoxyphenyl) methylene] -2-thioxo-4-thiazolidinone Example 26 5- [ (2-methoxyphenyl) methylene] -2-thioxo-4-thiazolidinone Example 27 5-[ (3 -methyl-4-methoxyphenyl) methylene] -2-thioxo-4-thiazolidinone Ex m le 29 5-[ [3,5-bis(l,l-dimethylethyl)-4-hydroxyphenyl]methylene] -2-thioxo-4-thiazolidinone, m.p. ~260°C X-6715 -36- Examole 29 5-[ (1, l'-biphenyl] -2 -yl) methylene) -2-thioxo-4-thiazolidinone Example Q 5-[ ( 3 -methoxy-4-hydroxyphenyl) methylene] -3- (2-propenyl) -2-thioxo-4-thiazolidinone, m.p. 146-148°C Example 31 5-[ ( 3 -methoxy-4-heptoxyphenyl) methylene] -2-thioxo-4 -thiazolidinone, m.p. 130-132°C Example 32 5-[ (3-ethoxy-4-hydroxyphenyl)methylene] -2-thioxo-4-thiazolidinone, m.p. 217-217.5°C Example 33 5- [ (3 -methylphenyl) methylene] -2-thioxo-4-thiazolidinone, m.p. 197-202°C Example 5-[ ( 4 -methylphenyl) methylene] -2-thioxo-4-thiazolidinone, m.p. 229-234°C Example 35 5-[ (2-naphthalenyl) methylene) -2-thioxo-4-thiazolidinone, m.p. 224-225°C Example 36 5-[ (3 , 4-dichlorophenyl) methylene] -2-thioxo-4-thiazolidinone Example 37 4- [ (2-thioxo-4-thiazolidinone) methylene] benzoic acid, m.p. ~320°C X-6715 37- Exaimole 38 5- [ (3 , 4-diethoxyphenyl) methylene] -2-thioxo-4-thiazolidinone Example 39 5- [ (lH-indol-3-yl) methylene] -2-thi xo-4-thiazolidinone Example 40 5-[ (3 -hydroxy-4-methoxyphenyl) methylene] -2-thioxo- -thiazolidinone, m.p. 218-220°C Example 41 5- [ (3-methoxy-4-butoxyphenyl) methylene] -2-thioxo-4-thiazolidinone, m.p. 175-176°C Example 42 5- [ [ (1, 1 ' -biphenyl) -4-yl] methylene] -2-thioxo-4 thiazolidinone, m.p. 245-250°C Example 43 5-[ (3 -hydroxy-4-nitrophenyl) methylene] -2-thioxo-4-thiazolidinone, m.p. ~224°C Example 44 5- [ (3-hydroxyphenyl)methylmethylene] -2-thioxo-4-thiazolidinone Example 45 5-[ (3 -methoxy-4-pentoxyphenyl) methylene] -2-thioxo-4-thiazolidinone, m.p. 170-171°C X-6715 -38- Exanrole 46 5-[ (3 -hydroxy-4-ethoxyphenyl) methylene] -2-thioxo-4-thiazolidinone, m.p. >225°C Example 47 5- [ (4-pentoxyphenyl) methylene] -2-thioxo-4-thiazolidinone, m.p. 158.5-160°C Example 48 5-[ (3 -methoxy-4-ethoxyphenyl) methylene] -2-thioxo-4-thiazolidinone, m.p. 207-207.5°C.
Example 49 5-[ (3 -ethoxy-4-propbxyphenyl) methylene] -2-thioxo-4-thiazolidinone, m.p. 156-157°C Example 50 5- [ (3 -propoxy-4 -ethoxyphenyl) methylene] -2-thioxo-4-thiazolidinone, m.p. 186.5-188CC Example 51 5- [ (3 , 4-dipropoxyphenyl) methylene] -2-thioxo-4-thiazolidinone, m.p. 167.5-168.5°C Example 52 5- [ (3 -methoxy-4-butoxypheny1) methylene] -2-thioxo-4-thiazolidinone, sodium salt m.p. >225°C X-6715 -39- Example 53 5- [ [3 , 5-bis (1, 1-dimethylethyl) -4-hydroxy-phenyl] methylene] -4-oxo-2-thioxo-3-thiazolidine acetic acid, m.p. ~265°C Example 54 5- [ ( 3 -methoxy-4-butoxyphenyl) methyl] -2-thioxo 4-thiazolidinone, m.p. 152-153.5°C Example 55 5- [ (3 , 5-dichloro-4-hydroxyphenyl)methylene] -2 thioxo-4-thiazolidinone, m.p. >260°C Example 56 5- [ (3-ethoxy-4-butoxyphenyl)methylene] -2-thioxo-4-thiazolidinone Example 57 5- [ (3 -methoxy-4-pentoxyphenyl) methylene] -2-thioxo-4-thiazolidinone sodium salt, m.p. ~254°C Example 58 5-[ (3-ethoxy-4-methoxyphenyl)methylene] -2-thioxo-4-thiazolidinone, m.p. >225°C X-6715 -40- Ex¾mple 9 5- [ [3, 5-bis (1-methylpropyl) -4-hydroxy-phenyl] methylene] -4-oxo-2-thioxo-3-thiazolidine acetic acid, m.p. 191-193°C Example 60 5- [ (3 , 4-dimethoxyphenyl) methylene] -2-thioxo-4-thiazolidinone Example 61 5- [ ( 4-butoxyphenyl) methylene] -2-thioxo-4-thiazolidinone, m.p. 180°C Example 62 5- [ (3 , 5-dimethyl-4-hydroxyphenyl) methylene] -2-thioxo-4-thiazolidinone, m.p. 260°C Example 63 5- [ (3 , 5-dimethoxy-4-hydroxyphenyl) methylene] -2 thioxo-4-thiazolidinone, m.p. 230°C Example 6 5- [ (3-methoxy-4-pentoxyphenyl)methyl] -2-thioxo 4-thiazolidinone, m.p. 163-164°C Example 65 5- [ (3-methoxy-4-pentoxyphenyl)methylene] -2-thioxo-3-methyl-4-thiazolidinone, m.p. 117-118°C X-6715 -41- Example 66 5-[ (3 -methoxy-4-pentoxyphenyl) methylene] -4-thiazolidinone, m.p. 174-175°C Example $7 5- [ (3-methoxy-4-pentoxyphenyl) methyl] -4-thiazolidinone, m.p. 108-109°C Example 68 5-[ (3-methoxy-4-hexoxyphenyl) methylene] -2-thioxo-4-thiazolidinone Example 69 5- [ (3 -methoxy-4-octoxyphenyl) methylene] -2-thioxo-4-thiazolidinone, m.p. 125-127°C Example 70 5- [ (3 , 5-dimethoxy-4-pentoxyphenyl)methylerie] -2-thioxo-4-thiazolidinone, m.p. 166-167°C Example 71 5- [ [3- (1, 1-dimethylethyl) -4-hydroxy-5-(methy11hiopheny1) phenyl]methylene]-2-thioxo-4-thiazolidinone, m.p. 181-184°C X-6715 -42- Example 72 5- [ [3-ethoxy-4-hydroxy-5- (methylthio-pheny1) phenyl] methylene] -2-thioxo-4-thiazolidinone, m. 190-192°C Example 73 5- [ [3-ethoxy-4-hydroxy-5- (methylthio-pheny1)phenyl] methylene] -2-thioxo-3 -methy1-4-thiazolidinone, m.p. 137°C Example 74 5- [ [3-ethoxy-4-hydroxy-5- (methylthio-phenyl)phenyl] methylene] -4-oxo-2-thioxo-3 -thiazolidine acetic acid m.p. 202-206°C Exam le 75 5- [ (1-naphthyl) methylene] -2-thioxo-4-thiazolidinone, m.p.224-225°C Example 76 5- [ (2-naphthyl)methylmethylene] -2-thioxo-4-thiazolidinone Exampl 77 5- [ (3-phenoxyphenyl)methylene] -2- hioxo-4-thiazolidinone X-6715 -43- Examole 78 5-[ (3-phenoxyphenyl)methylmethylene] -2-thioxo-4-thiazolidinone Example 79 5- [ [3- (methyloxyphenyl) phenyl] methylene] -2-thioxo-4-thiazolidinone, m.p. 177-180°C.
Example 80 5-[ (3-methoxy-4-heptoxyphenyl) methylene] -2-thioxo-3-amino-4-thiazolidinone, m.p. 118-121°C (dec) .
Example 81 5-[ ( 3 -methoxy-4-heptoxyphenyl) methylene] -2-thioxo-3-dimethylamino-4-thiazolidinone Two hundred and fifty milligrams (1 mmol) of 3-methoxy-4-hep oxy benzaldehyde, 233 mg (1.2 mmol) of 2- (N-dimethylamino-dithiocarboxamido) acetic acid (a compound of formula VI, above), 330 mg (4 mmol) of anhydrous sodium acetate and 5 ml of acetic acid were stirred while heating at reflux for 15 hours. The reaction was then quenched by pouring the reaction solution into 10 ml of an ice/water mixture. The resulting solids were recovered by filtration, washed with ethyl acetate and then water to provide 450 mg of impure title compound. The impure compound was purified via recrystallization from hexane/methylene chloride to provide 180 mg of pure title compound, m.p. 105-108°C.
X-6715A -44- Example 82 5- [ [4- (dimethylamino) phenyl] methylene] -2-thioxo-4-thiazolidinone Exam le 83 5- [ ( -heptoxyphenyl) methylene] -2-thioxo-3-dimethylamino-4-thiazolidinone, m.p. 80°C.
The compounds of the present invention may be used as medicaments, for example for lowering blood glucose levels in mammals, for example by administering a therapeutically effective amount of a compound of formula I . The term "therapeutically effective amount", as defined herein, means the amount of compound necessary to provide a hypoglycemic effect following administration, preferably to a human susceptible to adult onset diabetes.
The hypoglycemic activity of the compounds of the present invention was determined by testing the efficacy of the compounds in vivo in male viable yellow obese-diabetic mice. The test procedure is described in detail below.
Test formulations were prepared by dissolving the test compound in a saline solution containing 2% Emulphor (a polyoxyethylated vegetable oil surfactant from GAF Corp . ) to provide the dose level desired. Each test formulation was administered to six viable yellow obese-diabetic mice intraperitoneally at the beginning of the experiment. Blood glucose levels were determined immediately before the first dose and at 2 and 4 hours thereafter using glucose oxidase . A mean was taken of the 6 values obtained before the first dose and at the 2 and 4 hour intervals. The 2 and 4 hour mean values, calculated as a percentage of the first dose mean value, are reported r:\0library\karchive\up-4500\4107nopc.doc X-6715 -45- in Table 1, below. In Table 1, Column 1 provides Che example number of the test compound, Column 2 provides the dose level of compound tested, and Columns 3 and 4 provide a measurement of the test animal's blood glucose level 2 and 4 hours after test compound administration, respectively, as a percentage of the test animal's pre-administration blood glucose level.
X-6715 -46- TABLE 1 HYPOGLYCEMIC ACTIVITY OF TEST COMPOUNDS IN OBESE DIABETIC MICE Example # of Percent of Initial Compound Dose Blood Glucose Level Tested (mg/kg) After After 2 hrs. 4 hrg 1 50 82 + 5 75 ± 2 2 50 96 ± 1 82 ± 3 3 50 90 ± 10 73 ± 3 4 50 91 ± 4 72 + 7 5 50 79 ± 4 71 ± 3 6 50 85 + 6 72 ± 4 6 50 92 ± 4 79 ± 4 7 50 80 ± 4 91 ± 7 8 50 94 ± 4 84 ± 6 9 50 91 ± 8 83 + 6 10 50 89 ± 4 80 ± 4 11 50 84 + 3 85 + 6 12 50 90 ± 7 69 + 6 13 50 94 ± 4 88 + 5 14 50 84 ± 7 71 ± 8 15 50 73 + 5 62 ± 4 16 50 94 + 8 96 ± 9 17 50 88 ± . 8 89 + 10 18 50 89 ± 4 88 ± 5 19 50 85 ± 14 75 ± 4 20 50 76 + 3 70 ± 5 21 50 99 + 4 81 ± 6 22 50 77 ± 5 67 + 2 X-6715 -47- Table 1 (cont'd) Example # of Percent of Initial Compound Dose Blood Glucose Level Tested (mg/kg) After After 2 hrs 4 hrs 22 50 77 ± 6 69 + 6 23 50 74 + 6 90 + 6 24 50 78 + 4 80 + 5 25 50 78 + 4 74 ± 4 25 25 84 + 5 87 ± 6 26 50 80 + 4 75 ± 2 27 50 93 + 3 84 ± 6 28 50 83 + 9 79 + 7 29 50 84 ± 5 77 ± 6 30 .- 50 78 ± 7 81 + 5 31 50 76 ± 7 76 + 5 32 50 75 ± 4 80 ± 8 32 50 80 + 18 66 + 11 33 50 91 ± 6 86 ± 7 34 50 85 ± 8 79 + 9 35 50 83 + 5 85 ± 6 36 50 81 + 7 90 + 8 37 50 89 + 4 80 + 4 38 50 60 ± 5 59 ± 4 38 50 96 ± 6 80 ± 3 38 50 86 + 4 81 ± 5 38 25 69 ± 9 65 + 7 38 10 72 ± 4 71 ± 6 38 10 73 + 8 59 + 7 39 50 83 + 4 76 ± 4 40 50 78 ± 5 72 + 4 X-6715 -48- Table 1 (cont ' d) Example # of Percent of Initial Compound Dose Blood Glucose Level Tested (mg/kg) After After 2 hrs. 4 hrs . 41 50 61 + 3 51 + 4 41 50 64 ± 6 54 + 5 41 50 77 ± 5 62 + 5 41 50 77 + 5 72 + 8 41 25 58 + 6 45 ± 5 41 25 72 + 7 64 + 4 41 25 74 ± 7 70 ± 8 41 25 87 + 5 85 ± 6 41 10 80 ± 7 59 ± 4 41 10 97 + 7 75 + 5 41 10 92 + 7 92 + 7 41 5 93 + 10 71 + 4 41 5 95 + 4 97 ± 5 42 50 87 + 8 70 ± 8 43 50 92 + 7 88 ± 4 44 50 98 + 4 88 + 5 45 50 76 + 7 57 + 3 45 50 68 + 2 66 ' + 4 45 25 93 ± 4 87 ± 5 45 25 83 + 10 78 ± 12 46 50 79 ± 4 77 ± 5 47 50 99 + 14 76 + 8 48 50 70 ± 3 65 + 3 48 25 87 + 4 81 ± 5 49 50 83 + 5 77 + 7 50 50 75 + 5 69 + 5 51 50 89 + 7 85 ± 8 X-6715 -49- Table 1 (cont'd) Example # of Percent of Initial Compound Dose Blood Glucose Level Tested (mg/kg) After After '. 2 hrs. 4 hrs 52 50 73 + 3 61 + 4 53 100 83 ± 9 80 + 14 53 50 73 ± 4 55 + 5 54 50 76 ± 7 74 + 6 55 50 81 ± 3 75 ± 3 56 50 78 + 4 72 ± 3 56 25 81 + 8 75 ± 3 56 10 94 ± 4 97 + 4 57 50 63 ± 6 58 + 7 57 . 50 69 + 5 63 + 7 57 25 67 ± 7 66 + 7 57 25 79 ± 10 70 ± 4 57 10 95 ± 3 87 + 6 57 5 82 + 6 68 ± 5 58 50 67 + 2 75 ± 5 59 50 62 ± 5 59 + 9 60 50 85 ± 4 78 ± 3 60 50 102 ± 6 81 ± 5 60 25 87 ± 7 89 ± 6 61 50 76 ± 5 61 ± 5 61 50 98 ± 8 79 ± 4 The hypoglycemic activity of the compounds of the present invention was confirmed in a second in vivo test system; namely, the normal fed rat system. The procedure used in this test system is described below.
Male Sprague Dawley rats (Charles River Laboratories) weighing 175-200 g were used in this test system. Test formulations were prepared by suspending the X-6715 -50- test compound in 5% acacia (concentration of the drug was adjusted such that 0.25 ml/100 g body weight administered orally gave the desired dose on a body weight basis) . The desired dose level of each test formulation was administered to four rats by oral gavage at the beginning of the experiment. Blood glucose levels were determined immediately before the first dose and at 3 and 5 hours thereafter by an enzymatic procedure employing glucose oxidase and peroxidase coupled with a chromogenic oxygen acceptor. A mean was taken of the 4 values obtained before the first dose and at the 3 and 5 hour intervals. The 3 and 5 hour mean values, calculated as a percentage of the first dose mean value, are reported in Table 2, below. In Table 2, Column 1 provides the example number of the test compound, Column 2 provides the dose level of compound tested, and Columns 3 and 4 provide a measurement of the test animal's blood glucose level 3 and 5 hours after test compound administration, respectively, as a percentage of the test animal's pre-administration blood glucose level.
TABLE 2 HYPOGLYCEMIC ACTIVITY OF TEST COMPOUNDS TN NORMAL FED RATS Example # of Percent of Initial Compound Dose Blood Glucose Level . Tested (mg/kg) After After 3 rs. 5 hrs. 15 167 84 87 16 200 92 79 17 200 78 68 22 200 84 68 24 200 100 100 25 200 100 100 26 200 100 100 31 200 95 92 32 200 100 96 38 200 90 74 41 160 76 67 45 167 61 63 47 200 82 73 48 167 87 81 49 200 100 98 56 150 79 65 57 200 84 73 58 200 100 100 61 200 89 82 62 200 78 53 63 200 69 52 64 200 91 89 65 200 100 91 X-6715 -52- Table 2 (cont'd) Example # of Percent of Initial Compound Dose Blood Glucose T.eveT Tested (mg/kg) ' After After '. 3 hrs, 5 hrs 66 200 100 86 67 200 92 88 68 200 88 89 69 200 93 88 The hypoglycemic activity of the compounds of the present invention was confirmed in yet a third in vivo test system; namely, the obese diabetic Zucker rat (Zucker Diabetic Fatty Rat) test system. The rats used in this test system were 6 to 8 months old, weighed between 550 to 625 grams and had a pre-drug blood glucose level between 250 to 350 mg/dl. The procedure used in this test system is the same as that described for the normal fed rat test system, above. The results of such tests are set forth in Table 3, below. The format of Table 3 is the same as that used in Table 2.
ABLE 3 HYPOGLYCEMIC ACTIVITY OF TEST COMPOUNDS IN OBESE DIABETIC ZUCKER RATS Example # of Percent of Initial Compound Dose Blood Glucose Level Tested (mg/kg) After After 3 hrs, 5 hrs 22 50 53 56 45 167 30 20 47 167 74 66 56 50 79 66 Finally, the long-term hypoglycemic activity of the compounds of the present invention was tested in yet another jji vivo test system. This long-term test system entailed incorporating test compound into the test animal's diet at various concentrations (control animal's diet contained no test compound) . Such diet was then fed to the test or control animals for either 14 or 21 days. Each test or control animal was then bled from the tail (200-400 μΐ sample of blood) at 0 (before diet started) , 7, 14 and, if appropriate, 21 and 28 days after diet administration was started. Plasma samples were then obtained from each blood sample collected and the glucose concentration of such plasma samples was determined enzymatically .
The results of the long-term hypoglycemic test system described above are set forth in Table 4, below. In Table 4, Column 1 describes the type of rodent used in the test system, Column 2 provides the example number of the test compound or indicates that the numbers reported are for a control animal, Column 3 provides the concentration, in percent, of test compound in the test or X-6715 -54- control animal's diet. Columns 4-8 provide the plasma glucose concentration at days 0, 7, 14 and, if appropriate, 21 and 28, respectively, for the animals tested. Glucose lowering was not associated with depressed diet consumption.
TABLE 4 LONG-TERM HYPOGLYCEMIC , ACTIVITY OF TEST COMPO Type of Rodent* Example No. of Concentration Plasma Glucos Cmpd. Tested of Test Cmpd. (mg/dl) in Diet (%) 0 7 14 ZDF 45 0.1 388 140 1 ZDF control -- 416 364 4 ZDF 45 0.1 464 215 2 ZDF 45 0.025 467 451 4 ZDF control 478 499 5 ZDF 45 0.1 357 171 1 ZDF 64 0.1 339 187 1 ZDF control 343 423 4 ZDF 45 0.1 309 137 1 ZDF 71 0.1 311 237 2 ZDF 70 0.1 300 190 1 ZDF control 317 286 2 Male A a 45 0.1 438 338 3 (Harlan) Male A a 38 0.1 340 351 3 (Harlan) Male A a control 429 414 4 (Harlan) * ZDF = 8 week old male Zucker Diabetic Fatty rat; ΑΥΥ/Ά = viable X-6715A -56- The invention of application no. 106877 provides compounds of formula (la) for use as medicaments for treating Alzheimer's disease in mammals, for example by administering a therapeutically effective amount of a compound of formula la. The term "therapeutically effective amount", as defined for this use, means the amount of compound necessary to reduce, eliminate or prevent the physiological effects or, causes of Alzheimer's disease following administration, preferably to a human suffering from or susceptible to Alzheimer's disease.
Alzheimer's disease is a degenerative disorder of the human brain. Clinically, it appears as a progressive dementia Its histopathology is characterized by degeneration of neurons, gliosis, and the abnormal deposition of proteins in the brain. Proteinaceous deposits (called "amyloid") appear as neurofibrillary tangles, amyloid plaque cores, and amyloid of the congophilic angiopathy. [For reviews, see, Alzheimer's Disease, (B. Reisberg, ed., The Free Press 1983) . ] While there is no general agreement as to the chemical nature of neurofibrillary tangles, the major constituent of both the amyloid plaque cores and the amyloid of the congophilic angiopathy has been shown to be a 4500 Dalton protein originally termed β-protein or amyloid A4.
Throughout this document this protein is referred to as S-amyloid peptide or protein. β-amyloid peptide is proteolytically derived from a transmembrane protein, the amyloid precursor protein (APP) . Different splice forms of the amyloid precursor protein are encoded by a widely expressed gene, see, e.g., K. Beyreuther and B. Muller-Hill, Annual Reviews in Biochem-istryr 58:287-307 (1989) . β-amyloid peptide consists, in its longest forms, of 42 or 43 amino acid residues. J. Kang, et al ..
Nature (London) , 325:733-736 (1987). These peptides, however, vary as to their nVOlibrar Vkarchive\up-4S00\4107nopc.doc X-6715 -57- amino-termini. C. Hilbich, et al . , Journal of Molecular Biolocrv, 218:149-163 (1991).
Because senile plaques are invariably surrounded by dystrophic neurites, it was proposed early that β-amyloid peptide is involved in the loss of neuronal cells that occurs in Alzheimer's disease. B. Yankner and co-workers were the first to demonstrate that synthetic β-amyloid peptide could be neurotoxic in vitro and in vivo. B. A. Yankner, et al .. Science, 245:417 (1989); see also, N. W. Kowall, et al.. Proceedings of the National Academy of Sciences. U.S.A.. 88:7247 (1991). Other research groups, however, were unable to consistently demonstrate direct toxicity with β-amyloid peptide, see, e.g., Neurobiology of Aging, 13:535 (K. Kosik and P. Coleman, eds. 1992) . Even groups receiving β-amyloid peptide from a common source demonstrate conflicting results. D.
Price, et al.. Neurobiology of Aging, 13:623-625 (1991) (and the references cited therein) .
As mentioned supra , cells have alternative mechanisms for processing APP which can result in the formation of the β-amyloid protein and subsequently, the senile plaques. It is likely that this alternative processing route occurs in the lysosomes. It has been found that compounds that inhibit lysosomal enzymes inhibit the fragment formation, see, e.g., Science, 155:689 (1992).
A lysosome is a membranous reservoir of hydrolytic enzymes responsible for the intracellular digestion of macromolecules . Lysosomes are known to contain approximately forty hydrolytic enzymes, including proteases, nucleases, glycosidases, lipases, phospholipases, phosphatases and sulfatases. These enzymese are all acid hydrolases which are optimally active at about pH 5. Therefore, it is necessary to determine which enzyme or enzymes are responsible for this X-6715 -58- alternative processing of the APP and the consequent formation of the β-amyloid protein.
Abnormally high concentrations of the proteases cathepsins D and B have been observed in the brains of patients with early-onset Alzheimer's disease. Yu Nakamura, et al .. Neuroscience Letters. 130, 195-198 (1991) . Furthermore, elevated activity for cathepsin D has been observed in the brains of Alzheimer's patients. M. Takeda, et al . , Neurochemistrv Research, (abstract) , 11:117 (1986) . Cathepsin D is a lysosomal endoprotease that is present in all mammalian cells, see, e.g., "Proteinases in Mammalian Cells and Tissues," ed. (A. J. Barret, ed. 1977) pp. 209-248. It is the only aspartyl protease that is known to be a lysosomal enzyme.
The cathepsins are a family of hydrolase enzymes that are usually located in the lysosomes. These enzymes are endopeptidases with an acidic optimum pH. .. Cathepsin A is a serine carboxypeptidase, cathepsin C [EC 3.4.14.1] is a dipeptidyl peptidase, cathepsin D [EC 3.4.23.5] is an aspartyl protease, and cathepsin B2 [EC 3.4.16.1] is a serine carboxypeptidase. Cathepsin B [EC 3.4.22.1] (also known as cathepsin Βχ) and cathepsin L [EC 3.4.22.15] are thiol proteases having activity within the lysosomes .
It has been found that inhibition of cathepsin D using an aspartyl protease inhibitor reduces the formation of β-amyloid protein and the resultant senile plaque. As such, compounds which inhibit cathepsins (and, in particular, cathepsin D) or reduce the formation of β-amyloid protein would be expected to be useful in treating Alzheimer's disease. Such activities were demonstrated in the following test systems.
X-6715 -59- CATHEPSIN D PERCENT INHIBITION ACTIVITY A fluorometric assay was adapted from the method disclosed by Murakami et al., Anal. Biochem.
H _: 232-239 (1981) for measuring renin activity. Human liver cathepsin D (Athens Research and Technology, Athens, GA) was diluted in assay buffer, 200mM. NaOAc, pH 4.5, 150mM NaCl to 500 ng/mL and then 100 UL of this cathepsin D solution was added to each well of a 96 well plate with the exception of control wells which received just 100 1 of assay buffer. Compound stocks were prepared by dissolving a sufficient quantity of the particular compound to be tested in DMSO such that a 10 g/ml concentration of test compound in DMSO was obtained and then 5 ΐ* of the compound stock was added to each of the wells prepared above. Blank and enzyme control wells each received 5 μΐ. of the DMSO vehicle.
Following a ten minute incubation at 25°C to allow enzyme/compound interaction, 5 μ]_ι of a 500μ solution of a derivative of a known porcine renin tetradecapeptide fluorometric substrate (Bachem Biosciences, Inc. 1993 Catalog ID No. 1-1340; Bachem Biosciences, Philadelphia, PA) in DMSO was added per well to initiate the reaction. After incubation at 37°C for 30 minutes, cathepsin D activity was terminated by the addition of 100 [1L per well of 400 mU/mL microsomal leucine aminopeptidase (EC 3.4.11.2, Sigma, St. Louis, MO) in 1M Tris-HCl, pH 8.0.
The plates were then analyzed in a fluorometer (CytoFluor 2350, Millipore, Bedford, MA) with an excitation wavelength of 360nm and an emission wavelength of 460nm, in order to check for background fluorescence due to test compounds. Following a two hour incubation at 37°C, to allow the aminopeptidase to release the fluorophore, 7-amido-4-methylcoumarin (AMC) from the X-6715 -60- products of cathepsin D cleavage, the plates were again analyzed in the fluorometer. In order to check for potential false positives, i.e., inhibitors of microsomal leucine aminopeptidase, residual aminopeptidase activity was monitored directly in each well by the addition of 20 jj/well of 2.5mM Leu-pNA (Bachem Biosciences, Philadelphia, PA) in 10% DMSO. Aminopeptidase activity was measured as an increase in the absorbance of 405nm in a UVmax microplate reader (Molecular Devices, Menlo Park, CA) .
Cathepsin D activity was linear under these conditions and the results are expressed as a percentage of the controls in Table 5, below. All results presented are the mean and standard deviation of at least four replicate assays.
X-6715 -61- TABLE 5 CATHEPSIN D INHIBITION ACTIVITY % Inhibition Example No. of Cathepsin D 1 36 4 50 5 74 6 29 8 64 18 38 42 88 45 62 50 40 55 90 75 43 76 32 77 87 81 21 82 79 82 68 83 47 CATHEPSIN D INHIBITION IC50 ACTIVITY The above assay was repeated with the exception that the compound stocks were prepared in various concentrations other than 10 g ml so that IC50 values (concentration of test compound at which 50% inhibition of cathepsin D was obtained) for the test compounds could be determined. The results obtained from such assay system are set forth in Table 6 below. Χ-6715 -62- TABLE 6 Example No. IC50 (Mg/Ml 5 2.6 28 3.1 31 1.6 35 8.9 42 1.6 47 5.2 56 >4.15 68 3.4 68 1.4 69 2.4 71 1.2 71 4.8 77 4.5 78 25.0 79 3.7 80 47.0 S-AMYLOID PROTEIN PRODUCTION INHIBITION Two cell lines (human kidney cell line 293 and Chinese hamster ovary cell line CHO) were stably transfected with the gene for APP751 containing the double mutation Lys-651-Met-652 to Asn-651-Leu-652 (APP-751 numbering) commonly called the Swedish mutation using the method described in Citron et al., Nature 360:672-674 (1992) . The transfected cell lines were designated as 293 751 SWE and CHO 751 SWE, and were plated in Corning 96 well plates at 2.5xl04 or lxlO4 cells per well respectively in Dulbecco's minimal essential media (DMEM) X-6715 -63- plus 10% fetal bovine serum. Following overnight incubation at 37°C in an incubator equilibrated with 10% carbon dioxide (CO2)., the media were removed and replaced with 200 |1L per well of conditioned media (media containing compound stocks; compound stocks diluted with media such that the concentration of DMSO in the media/compound stock solution did not exceed 0.5%) for a two hour pretreatment period during which the cells were incubated as described above. These compound stocks were prepared by dissolving a sufficient quantity of the particular compound to be tested in DMSO such that a 10 μg/ml concentration was obtained. After this pretreatment period, the conditioned media was removed and replaced with fresh conditioned media and the cells were incubated for an additional two hours .
After treatment, plates were centrifuged in a Beckman GPR at 1200 rpm for five minutes at room temperature to pellet cellular debris from the conditioned media. From each well, 100 |1L of conditioned media were transferred into an ELISA plate precoated with antibody 266 [Seubert et al.. Nature . 359:325-327 (1992)] and stored at 4°C overnight prior to the completion of the ELISA assay the next day.
Cytotoxic effects of the compounds were measured by a modification of the method of Hansen ejt ai. , J. Immun. Meth. 119:203-210 (1989). To the cells remaining in the tissue culture plate, was added 25 \1L of a 3- (4, 5-dimethylthiazol-2-yl) -2 , 5-diphenyltetrazolium bromide (MTT) stock solution (5 mg/mL) to a final concentration of 1 mg/mL. Cells were incubated at 37°C for one hour, and cellular activity was stopped by the addition of an equal volume of MTT lysis buffer (20% w/v sodium dodecylsulfate in 50% DMF, pH 4.7) . Complete extraction was achieved by overnight shaking at room temperature. The difference in the OD562nm a d the ODgsonm X-6715 -64- was measured in a Molecular Devices UVmax microplate reader as an indicator of the cellular viability.
The results of the β-amyloid protein ELISA were fit to a standard curve and expressed as ng/mL β-amyloid protein peptide. In order to normalize for cytotoxicity, these β-amyloid protein results were divided by the cytotoxicity results and expressed as a percentage of the results from a drug-free control.
TABLE 7 β-AMYLOID PROTEIN INHIBITION % Inhibition of Example No. β-Amyloid Prote 5 47 5 58 31 58 42 52 70 38 71 65 77 25 81 100 As can be seen from the data in Tables 5, 6 and 7, the compounds of formula la can be administered for prophylactic and/or therapeutic treatment of diseases related to the deposition of β-amyloid protein such as Alzheimer's disease, Down's syndrome, and advanced aging of the brain. In therapeutic applications, the compounds are administered to a host already suffering from the disease. The compounds will be administered in an amount sufficient to inhibit further deposition of β-amyloid protein plaque.
X-6715A -65- For prophylactic applications, the compounds of formula la are administered to a host susceptible to Alzheimer's disease or a β-amyloid protein related disease, but not already suffering from such disease . Such hosts may be identified by genetic screening and clinical analysis, as described in the medical literature, see e.g., Goate, Nature 349:704-706 (1991). The compounds will be able to inhibit or prevent the formation of the β-amyloid protein plaque at a symptomatically early stage, preferably prevent-ing even the initial stages of the S-amyloid protein disease.
The compounds of the present invention and the compounds utilized according to the invention of application no. 106877 are effective over a wide dosage range. For example, dosages per day will normally fall within the range of about 0.5 to about 500 mg/kg of body weight. In the treatment of adult humans, the range of about 1.0 to about 100 mg/kg, in single or divided doses, is preferred.
However, it will be understood that the amount of the compound actually administered will be determined by a physician in light of the relevant circumstances including the condition to be treated, the choice of compound to be administered, the age, weight, and response of the individual patient, the severity of the patient's symptoms and the chosen route of administration. Therefore, the above dosage ranges are not intended to limit the scope of the invention in any way. While the present compounds are preferably administered orally, the compounds may also be administered by a variety of other routes such as the transdermal, subcutaneous, intranasal, intramuscular and intravenous routes .
While it is possible to administer a compound of the invention, or a compound used as a medicament according to this invention, directly, the compounds are preferably employed in the form of a pharmaceutical formulation n\01ibrar \karchive\up-4500\4107nopc.doc X-6715 -66- comprising a pharmaceutically acceptable carrier, diluent or excipient and a compound of the invention. Such formulations will contain from about 0.01 percent to about 90 percent of a compound of the invention.
In making the formulations of the present invention, the active ingredient will usually be mixed with at least one carrier, or diluted by at least one carrier, or enclosed within a carrier which may be in the form of a capsule, sachet, paper or other container. When the carrier serves as a diluent, it may be a solid, semisolid or liquid material which acts as a vehicle, excipient or medium for the active ingredient. Thus, the formulations can be in the form of tablets, granules, pills, powders, lozenges, sachets, cachets, elixirs, emulsions, solutions, syrups, suspensions, aerosols (as a solid or in a liquid medium) and soft and hard gelatin capsules.
Examples of suitable carriers, diluents and excipients include lactose, dextrose, sucrose, sorbitol, mannitol, starches, gum acacia, calcium phosphate, alginates, liquid paraffin, calcium silicate, microcrystalline cellulose, polyvinyl pyrrolidone, cellulose, tragacanth, gelatin, syrup, methyl cellulose, methyl- and propyl-hydroxybenzoates , vegetable oils, such as olive oil, injectable organic esters such as ethyl oleate, talc, magnesium stearate, water and mineral oil. The formulations may also include wetting agents, lubricating, emulsifying and suspending agents, preserving agents, sweetening agents, perfuming agents, stabilizing agents or flavoring agents. The formulations of the invention may be formulated so as to provide quick, sustained or delayed release of the active ingredient after administration to the patient by employing procedures well-known in the art.
X-6715 -67- For oral administration, a compound of this invention, or a compound used in the methods of this invention, ideally can be admixed with carriers and diluents and molded into tablets or enclosed in gelatin capsules .
The compositions are preferably formulated in a unit dosage form, each dosage containing from about 1 to about 500 mg, more usually about 5 to about 300 mg, of the active ingredient. The term "unit dosage form" refers to physically discrete units suitable as unitary dosages for human subjects and other mammals, each unit containing a predetermined quantity of active material calculated to produce the desired therapeutic effect, in association with a suitable pharmaceutical carrier, diluent or excipient therefor.
In order to more fully illustrate the operation of this invention, the following examples of formulations are provided. The examples are illustrative only and are not intended to limit the scope of the invention. The formulations may employ as active con¾xmnds any of the compounds of the present invention.
X-6715 -68- FQRMULATION 1 Hard gelatin capsules suitable for use in treating Alzheimer's disease or reducing glucose concentration are prepared using the following ingredients : Amt. per Concentration by- Capsule Weight (percent) Compound of Example No. 5 250 mg 55.0 Starch dried 220 mg 43.0 Magnesium stearate 10 ma 2.0 460 mg 100.0 The. above ingredients are mixed and -filled into hard gelatin capsules in 46.0 mg quantities.
FQFMUI^ IQ ¾ Capsules each containing 20 mg of medicament are made as follows: Amt. per Concentration by Capsule Weight (percent) Compound of Example No. 1 20 mg 10.0 Starch 89 mg 44.5 Microcrystalline 89 mg 44.5 cellulose Magnesium stearate 2 ma 1.0 200 mg 100.0 The active ingredient, cellulose, starch and magnesium stearate are blended, passed through a No. 45 mesh U.S. sieve and filled into a hard gelatin capsule.
X-6715 -69- FORMULATIQN 3 Capsules each containing 100 mg of active ingredient are made as follows: Amt. per Concentration by Capsule Weight (percent) Compound of Example No. 45 100 mg 29.0 Polyoxyethylenesorbitan 50 meg 0.02 monooleate Starch powder 250 ma 71.0 250.05 mg 100.02 The above ingredients are thoroughly mixed and placed in an empty gelatin capsule.
X-6715 -70- FQRMULATION 4 Tablets each containing 10 mg of active ingredient are made up as follows: Amt. per Concentration by Capsule Weight (percent) Compound of Example No. 71 10 mg Starch 45 mg Macrocrystalline 35 mg cellulose Polyvinyl 4 mg pyrrolidone (as 10% solution in water) Sodium carboxyethyl 4.5 mg starch Magnesium stearate 0.5 mg Talc 1 mg 100 mg 100.0 The active ingredient, starch and cellulose are passed through a No. 45 mesh U.S. sieve and mixed thoroughly. The solution of polyvinylpyrrolidone is mixed with the resultant powders which are then passed through a No. 14 mesh U.S. sieve. The granule so produced is dried at 50°-60°C and passed through a No. 18 mesh U.S. sieve. The sodium carboxymethyl starch, magnesium stearate and talc, previously passed through a No. 60 mesh U.S. sieve, are then added to the granule which, after mixing, is compressed on a tablet machine to yield a tablet weighing 100 mg.
X-6715 71- FQRMULATION 5 A tablet formula may be prepared using the ingredients below: Amt. per Concentration by Capsule Weight (percent) Compound of Example No. 2 250 mg 38.0 Cellulose 400 mg 60.0 microcrystalline Silicon dioxide 10 mg 1.5 fumed Stearic acid 5 mg 0.5 665 mg 100.0 The components are blended and compressed to form tablets each weighing 665 mg.
X-6715 -72- FQRMULATION 6 Suspensions each containing 5 mg of medicament per 40 ml dose are made as follows: Per 5 ml of suspension Compound of Example No. 59 5 mg Sodium carboxymethyl 50 mg cellulose Syrup 1.25 ml Benzoic acid solution 0.10 ml Flavor q.v.
Color q.v.
Water q.s. to 5 ml The medicament is passed through a No. 45 mesh U.S. sieve and mixed with the sodium carboxymethylcellulose and syrup to form a smooth paste. The benzoic acid solution, flavor and color is diluted with some of the water and added, with stirring. Sufficient water is then added to produce the required volume.
X-6715 -73- FORMULA IQN 7 An aerosol solution is prepared containing .the following components: Concentration by Weight Compound of Example No. 53 0.25 Ethanol 29.75 Propellant 22 70.00 (Chlorodifluoromethane) 100.00 The active compound is mixed with ethanol and the mixture added to a portion of the propellant 22, cooled to . -30°C and transferred to a filling device. The required amount is then fed to a stainless steel container and diluted further with the remaining amount of propellant. The valve units are then fitted to the container.
Claims (8)
1. A compound of the formula wherein : Ar is (i) phenyl, (ii) phenyl substituted with from one to three substituents independently selected from Ci-Cs alkyl, Ci-Cs alkoxy, Ci-Cs alkylthio, trifluoromethyl, C2-C4 alkylphenyl, NO2, F, CI, phenoxy, C1-C4 alkyloxyphenyl , thiophenyl, C1-C4 alkylthiophenyl, -C0OR7, -N(R7)S02R7 or -N(R7)2, where each R7 is independently hydrogen or C1-C6 alkyl, (iii) 1- or 2-naphthyl, (iv) 2- or 3-benzofuranyl,' (v) 2- or 3-benzothiophenyl, (vi) 2- or 3-thienyl, (vii) 2-, 3- or 4-pyridyl, (viii) 2- or 3-furanyl, (ix) 1, 3 -benzodioxanyl, (x) substituted 1, 3 -benzodioxanyl , (xi) quinolinyl, (xii) 2- or 3-indolyl or (xiii) N-substituted 2- or 3-indolyl; R1 is C1-C6 alkyl, C1-C4 alkylphenyl, hydrogen, phenyl or phenyl substituted with one or two substituents independently selected from CI, Br, F, I, C1-C4 alkyl, Ci-C4 alkoxy, hydroxy, trifluoromethyl, - H2, - H(Ci~C4 alkyl), -N(Ci-C4 alkyl) 2 or C1-C4 alkylthio; R2 and R3 are each hydrogen or when taken together form a bond; R4 and R5 are each hydrogen or when taken together are =S, or when one of R4 and R5 is hydrogen, the other is -SCH3; is hydrogen, C1-C6 alkyl, C3-C8 cycloalkyl, C2 C6 alkenyl, -SO2CH3 or -(CH2)p-Y where p is 0, 1, 2, or 3 and Y is cyano, tetrazolyl, - R10R11, C4 alkylthio or \ -75- alkyl) where R8 is hydrogen, C1-C4 alkyl, or °(Ci-C4)alkyl _ r9 ^ hydrogen, C1-C4 alkyl or H2; an(i r1° and R11 are each independently hydrogen, Ci-Cg alkyl, C2-C5 alkenyl, -(CH2)qOH, - (CH2)qN(Ci-C4 alkyl)2, - (CH2) qS (C1-C4 alkyl) , C2-C6 alkynyl, phenyl, or C1-C4 alkylphenyl, where q is an integer from 1 to 6, both inclusive, or R10 and R11, taken together with the nitrogen atom to which they are attached, form a morpholinyl, piperidinyl, piperazinyl or N-methylpiperazinyl ring; and HI is 0, 1, or 2; and the pharmaceutically acceptable salts thereof; with the provisos that when Ar is (i) phenyl, (ii) phenyl substituted with from one to three substituents independently selected from C1-C8 alkyl, Ci-Cs alkoxy, F, Cl, trifluoromethyl, phenoxy, C1-C4 alkyloxyphenyl , Ci-Cs alkylthio, NO2, -N(R7)2 or -COOR7, where each R7 is independently hydrogen or C±-Cs alkyl, (iii) 1- or 2-naphthyl, (iv) 2- or 3 -benzofuranyl , (v) 2- or 3-benzothiophenyl, (vi) 2- or 3-thienyl, (vii) 2-or 3-indolyl, (viii) 2- or 3-furanyl, (ix) quinolinyl or (x) 2-, 3- or 4-pyridyl; R1 is hydrogen or C1-C6 alkyl; R2 and R3 taken together form a bond; m is 0; and R4 and R5 taken together are =S, then R6 must be other than hydrogen or Ci-C6 alkyl; when Ar is phenyl; R1 is hydrogen, methyl or ethyl; R2 and R3 taken together form a bond; m is 0; and R4 and R5 taken together are =S, then R6 must be other than phenyl or C1-C4 alkylphenyl; Ar cannot be phenyl substituted solely with one chloro substituent at the 4-position of the phenyl ring; when Ar is phenyl substituted with two ethoxy moieties at the 3- and -positions of the phenyl ring, R1 must be hydrogen; 119119/2 -76- Ar cannot be phenyl substituted with a COOH moiety at the 2-position of the phenyl ring; and when and are each hydrogen cannot be Ci~ C6 alkyl.
2. The compound.5- [ ( 3-methoxy-4-pentoxyphenyl ) -methylene] -2-thioxo-4-thiazolidinone or a pharmaceutically acceptable salt thereof.
3. The compound 5- [ [ 3- ( 1 , 1-dimethylethyl ) -4-hydroxy-5- (methylthiophenyl) phenyl]methylene] -2-thioxo-4-thiazolidinone or a pharmaceutically acceptable salt thereof .
4. A pharmaceutical formulation comprising a compound of any one of claims 1 to 3 associated with one or more pharmaceutically acceptable carriers.
5. A compound according to any one of claims 1 to 3 for use as a medicament.
6. Use of a compound according to any one of claims 1 to 3 in the preparation of a medicament, substantially as described in the specification.
7. A process for preparing a compound according to any one of claims 1 to 3 which comprises: (A) reacting a compound of the formula B I I Ar—C-Rl wherein: B is 0 or NH and Ar and R1 are as defined in claim 1, with a compound of the formula: 119119/2 -77- wherein: R4 , R5, m and R6 are as defined in claim 1, so as to provide a compound of the formula (B) reducing a compound of claim 1 wherein R4 and R5 taken together are =S so as to prepare a compound of claim 1 in which R4 and R5 are hydrogen; (C) reducing a compound of claim 1 in which R2 and R3 taken together form a bond so as to prepare a compound claim 1 in which R2 and R3 are hydrogen; (D) reducing a compound of claim 1 in which R2 and R3 taken together form a bond and R4 and R5 taken together are =S so as to prepare a compound of claim 1 in which R2, R3, R4 and R5 are all hydrogen; (E) alkylating a compound of claim 1 in which R6 is hydrogen so as to prepare a compound of claim 9 in which R6 is Ci-C6 alkyl, C2-C6 alkenyl, C3-C8 cycloalkyl or - (CH2) -PY (where p is an integer from 0 to 3, both inclusive, and Y is cyano, OR8, -SH, C1-C alkylthio, -NR10R11 or 119119/2 -78- alkyl) where R is as defined in claim 1); (F) acylating a compound of claim 1 in which R5 is hydrogen so as to prepare a compound of claim 1 in which R6 is -(CH2)p-Y, where p is an integer from 0 to 3, both inclusive, and Y is O II -CR9 9 , where R is as defined in claim 1; (G) oxidizing a compound of claim 1 wherein m is 0, so as to prepare a compound of claim 1 wherein m is 1; (H) oxidizing a compound of claim 1 wherein m is 0, so as to prepare a compound of claim 1 wherein m is 2 ; (I) oxidizing a compound of claim 1 wherein m is 1, so as to prepare a compound of claim 1 wherein m is 2; (J) reacting a compound of the formula B II Ar—C-Rl wherein : B is 0 or NH and Ar and R1 are as defined in claim 1, with a compound of the formula 119119/2 -79- wherein R4, R5 and m are as defined in claim 1, and R6 is -(CH2)P-Y (where p is an integer from 0 to 3, both inclusive, and Y is OR8, where R8 is hydrogen) so as to provide a compound of the formula wherein : Ar, R1, R4, R5 and m are as set forth in claim 1 and R6 is -(CH2)p-Y (where p is an integer from 0 to 3, both o II -PPH inclusive, and Y is OR8, where R8 is 3 ) ; (K) reducing a comopund of claim 1 in which R6 is -(CH2)P-Y, wherein p is 0 to 3, both inclusive, and Y is OR8, o II c ■ -C(C,-C4 alkyl) ^_ , c where R is , so as to prepare a compound of claim 1 in which R6 is -(CH2)p-Y, wherein p is 0 to 3, both inclusive, and Y is OR8, where R8 is hydrogen; (L) reacting a compound of the formula set forth in claim 1 in which R6 is -(CH2)P-Y, wherein p is 0 to 3, both inclusive, and Y is OR8, where R8 is tosyl, with an amine of the formula HNR10R11 (where R10 and R11 are as defined in claim 1) so to prepare a compound of claim 1 in which R6 is -(CH2)P-Y, wherein p is 0 to 3, both inclusive, and Y -NR10Rn; (M) treating a compound of claim 1 in which R6 is -(CH2)P-Y, wherein p is 0 to 3, both inclusive, and Y is cyano with tri-n-butyl tin azide so as to prepare a compound 119119/2 -80- of claim in which R6 is -(CH2)p-Y, wherein p is 0 to 3, both inclusive, and Y is tetrazolyl; (N) reacting a compound of the formula B II Ar—C-Ri wherein : B is 0 or NH and Ar and R1 are as defined in claim 1, with a compound of the formula wherein : R10 and R11 are as set forth in claim 1, so as to provide a compound of the formula wherein R4 and R5 taken together are =S and Ar, R1, R10 and R11 are as defined in claim 1; (0) heating a compound of claim 1 in which R6 is -(CH2)p-Y, wherein p is 0 to 3, both inclusive, and Y is -NR10R (neither of R10 or R11 being hydrogen) in an 119119/2 -81- ethanol/water mixture in the presence of a catlyst so as to prepare a compound of claim 8 in which R6 is -(CH2)P-Y wherein p is 0 to 3, both inclusive, and Y is -NR10R1:L (where one of R10 and R11 is hydrogen and the other is not hydrogen) ; (P) salifying a compound of any one of claims 1 through 3 by reacting the non-salt form of the compound with either a strong acid or a strong base.
8. A process for preparing a compound as claimed in any one of claims 1 to 3, substantially as described and illustrated in any one of the examples.
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Application Number | Priority Date | Filing Date | Title |
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US94335392A | 1992-09-10 | 1992-09-10 | |
IL106877A IL106877A (en) | 1992-09-10 | 1993-09-02 | Rhodanine derivatives for use as medicaments for the treatment of alzheimer's disease |
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IL119119A true IL119119A (en) | 1998-08-16 |
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IL11911993A IL119119A (en) | 1992-09-10 | 1993-09-02 | Rhodanine derivatives process for their preparation and pharmaceutical compositions containing them |
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