CN112289438A

CN112289438A - Method for identifying abnormal development of eye diopter and device for restraining excessive development of eye diopter

Info

Publication number: CN112289438A
Application number: CN202011181424.4A
Authority: CN
Inventors: 李浩天; 刘斌; 刘魏; 钱晶; 夏磊
Original assignee: Jianshi Optometry Technology Changzhou Co ltd
Current assignee: Jianshi Optometry Technology Changzhou Co ltd
Priority date: 2020-10-29
Filing date: 2020-10-29
Publication date: 2021-01-29

Abstract

The invention relates to a method for identifying abnormal development of eye diopter and a device for restraining excessive rapid development of eye diopter, which are used for acquiring a plurality of groups of cornea and eye axis parameters in the second stage of preset time, simulating the eye diopter growth and development by adopting a West-Gemmerd function on the plurality of groups of cornea and eye axis parameters, and if the fitted parameter gamma is larger than the parameter gamma of a normally developed eyeball_stdIf yes, judging that the diopter is abnormal in development, otherwise, judging that the diopter is normal in development. The device for restraining the excessive rapid development of the eye diopter comprises a multi-stage defocusing compensation training lens and a device for performing adaptability on the multi-stage defocusing compensation training lensThe training sighting target instrument utilizes the multistage defocusing compensation training lens to perform triggering compensation training, becomes a stable constraint state, controls diopter development to be lower than a set threshold value, and achieves the purposes of prevention and treatment.

Description

Method for identifying abnormal development of eye diopter and device for restraining excessive development of eye diopter

Technical Field

The invention relates to a method for identifying abnormal development of eye diopter and a device for restraining excessive rapid development of eye diopter, belonging to the field of eye diopter identification and correction.

Background

Human beings use dioptric systems inside eyes to focus light emitted by objects outside the eyes on retinas to form optical signals, and then the optical signals are transmitted to cerebral cortex along optic nerves to form object images. The location of the focus of the dioptric system is exactly on the fovea of the retina, called focus. Otherwise, it is called defocus. The defocus position is in front of the retina and is called myopic defocus. The defocus position is behind the retina and is called hyperopic defocus.

The dioptric system mainly comprises dioptric media such as cornea, crystal, vitreous body and the like. Dioptric media has the ability to bend the direction of propagation of light, which is measured in diopters by classical optics theory. Therefore, refractive media such as cornea, crystal, vitreous body, etc. have independent diopters. The object image can be presented in the fovea of the retina in a clear focusing manner by means of corneal diopter, lens diopter and vitreous diopter in an optimized cooperative combination, the diaphragm action of the pupil and the relaxation action of the extraocular muscles (commonly called 'triple linkage'). It must be emphasized that the quality of clear focus depends on the diopter-accurate compensation in the out-of-focus state (referred to as "out-of-focus compensation"), including accommodative compensation of the lens with dynamically changeable diopter, and vergence compensation of the dynamically relaxable extraocular muscles. The focusing quality of the eye dioptric system can be evaluated by testing the size of the eye dioptric power. For myopic eyes, the lower the test result of eye diopter, the worse the focusing quality.

Vision is a high-level endowment that human beings differentiate from other organisms during the evolution of the organisms, because the formation of vision requires the participation of learning thinking. Learning thinking is a mechanism unique to humans. After an external object is imaged on a cerebral cortex, the human brain can conduct three-dimensional expansion on the imaging of the object by referring to the touch sense formed by the contact of limbs, skin and the object, and finally three-dimensional vision is formed. The learning thinking is developed in the process of three-dimensional expansion. Ophthalmologists in the 16 th century find that the object image transmitted to the cerebral cortex by the retina of a human is inverted, and the human feels the size, the distance, the direction and the position of the object by groping and exploring the limbs, learns and thinks in a centripetal conductive visual central system consisting of 'hand → brain → vision', and finally turns over the visual inverted image to form an upright three-dimensional world.

Besides, the human vision learning thinking mechanism also represents the relearning and processing of completely new imaging signals. George starton (George Stratton) reverses the imaging of his visual central system and causes severe dizziness by wearing a telescope that can image backwards (the telescope invented by keplerian). Stalton, with the aid of a supporting tool, reconstructs the processing mechanisms of the visual central system after a few weeks, using learning-thinking mechanisms, thus dispelling dizziness and dyskinesias. However, after the telescope is removed, Steton returns to the extreme vertigo and mobility impaired state, and then resumes normal living ability after several weeks by means of the learning thinking again. The starton test (Stratton Experiment) shows that the brain visual central system can form a new processing result for a brand new imaging signal by relearning the thinking, and the learning thinking is named as visual psychology.

Visual psychology is widely applied in daily life, for example, a myopic patient wearing optical glasses for the first time feels tension and even dizziness of eye muscles when wearing the optical glasses, and a vision doctor can recommend that the patient can be effectively improved after adapting to the condition for 1 week, which is a process for enabling the visual psychology of a visual central system. The visual learning thinking mechanism can be deeply applied to the work of restraining the rapid development of the eye diopter.

As shown in fig. 1, the human eye optical imaging path and defocus compensation principle. During the long-term evolution of organisms, humans evolved the eye as an extremely sophisticated optical imaging device; whether a distant macro scenery is watched or a near fine substance is watched, light emitted by a target enters the eye dioptric system through the cornea and the pupil, and under the action of adjustment and convergence, the fovea of the retina obtains an imaging signal and transmits the imaging signal to the brain. The brain performs aberration grading on the imaging quality, and the grading result is fed back to the fovea of the retina. Then, the retina starts a defocusing compensation mechanism, sends a chemical signal to the choroid which is close to the retina, causes structural phase change of the choroid, further causes structural phase change of the sclera which is close to the choroid, pushes the physical position of the retina to change, and completes compensation. Among them, the fovea of the retina has densely arranged cone cells to receive light signals and image, and chemical factors are secreted to issue a choroidal phase change command. The choroid is composed primarily of 3 layers of blood vessels, including the large blood vessel Layer near the sclera (Haller Layer), the capillary blood vessel Layer near the retina, and the middle vascular sandwich (Satfler Membrane). The choroidal layer 3 vessels may change in thickness (thicken or thin) upon receiving a phase change command. The choroid is thickened, corresponding to myopic defocus compensation. The choroid is thinned, corresponding to the hyperopic defocus compensation. The sclera is composed of compact smooth collagen and elastic fibers, can be elastically deformed or plastically deformed under the action of the pushing force of the choroid, and the elastic deformation can be recovered while the plastic deformation can not be recovered. Myopes, especially those with over-developed diopters, are often accompanied by choroidal thinning and scleral plastic deformation.

FIG. 2 illustrates the compensation path when the human eye receives a sharpened imaging signal; the sharp imaging signal means that the imaging edge line is thin, the edge angle of the imaging outline is clear, the line is clear, and the detail is gorgeous. In contrast to fig. 1, fig. 2 represents a situation where the eye has a preceeding accommodation, i.e. the eye responds to excessive excitation of the light emitted by the object in front of the eye, the dioptric system provides an amount of accommodation that is more than necessary, the imaging signal delivered to the fovea of the retina is too sharp, the brain levels the quality of the image and feeds back higher order sharp aberrations, and the retina issues a choroidal thickening command in order to balance the eye accommodation. The choroid increases its thickness through a series of actions including increasing surface area and osmotic activity of proteoglycan complexes to allow water to enter the vascular layer, increasing the number of osmotically active molecules by increasing the capillary fenestration, draining fluid from the anterior chamber into the choroid, and allowing fluid in the retina to pass through the pigment epithelium layer into the choroid. The thickened choroid pushes the retina forward (i.e., toward the vitreous), producing myopic defocus compensation, thereby counteracting accommodation overstimulation and relaxing the refractive tone of the eye. The retina moves towards the vitreous body, the sclera fiber can be pulled to generate recoverable elastic compression, the sclera can not generate plastic deformation, and the diopter of the eye is not changed. This suggests that the ophthalmologist can keep eye diopter stable by triggering eye myopic defocus compensation.

In contrast, as shown in fig. 3, the compensation path when the human eye receives the blurred imaging signal also shows the path of the human eye with too fast diopter development due to the imaging blur; in contrast to sharp imaging, blurred imaging has thicker imaging edge lines, deformed imaging contours, blurred lines and details. There is a lag in accommodation in the eye, the dioptric system provides less than the desired amount of accommodation, the brain feeds back higher order blur aberrations, and the retina issues a choroidal thinning command. The choroid reduces blood flow in the vascular layer, releases water, reduces protein permeation, causes the thickness of the retina to be thin, and the retina moves along the direction away from the vitreous body to generate hyperopic defocus compensation, thereby making up for insufficient adjustment caused by adjustment lag. The eye will initiate convergence compensation of the extraocular muscles to elongate the axis of the eye, increasing the distance the retina moves backwards. The retina moves towards the back of the vitreous body, tears scleral fibers and generates unrecoverable plastic deformation, which causes rapid development of eye diopter. The greater the amount of accommodative lag, the greater the degree of choroidal and scleral thinning, the longer the axial length of the eye, the faster the diopter development of the eye, and the higher the myopic power. It is clear that blurred imaging of the fovea is a key factor responsible for the rapid development of eye diopters, while hyperopic defocus compensation triggered by accommodative lag causes progressive progression of myopia power.

At present, myopia is high, the number of people with myopia in China exceeds 6 hundred million, and the myopia is even impregnated to teenagers and preschool children. The medical community calls for establishing a dynamic file of the eye diopter of the teenagers, carries out regular monitoring on the eye diopter of the teenagers, and if necessary, measures are taken to implement constraint on the rapidly developed diopter, so that the proportion of low diopter is limited below a set threshold value, and excellent visual focusing quality is ensured.

Disclosure of Invention

The invention aims to solve the technical problem of providing a method for identifying the abnormal development of the diopter of the eye, which comprises the following steps:

1) acquiring multiple groups of cornea and ocular axis parameters in the second stage in preset time, and adopting West Gem function for the multiple groups of cornea and ocular axis parameters

The growth and development of the diopter of the eye are simulated,

wherein ref. (n) is an ocular diopter function, corresponding to an ocular diopter of n years of age, and metric units are D;

delta is the diopter of the first stage, corresponding to the diopter of the eyes at the age of 0-3, and the metric unit is D; the invention delta is the diopter of the eye at the first stage, preferably corresponding to the diopter of the eye at age 1.

Gamma is a curve curvature parameter and is dimensionless, the parameter gamma is used for representing the change slope of the curve at the second stage, and the larger the parameter gamma is, the higher the slope of the curve is; x, represents the multiplication of the two;

max is the diopter of the eye at the third stage, corresponding to the diopter of the eye after the age of 16, and the metric unit is D;

n is age, metric unit is, age;

the first stage is an initial stage of 0-3 years old, the second stage is a development stage of 4-15 years old, and the third stage is a stabilization stage of 16-35 years old,

2) performing West-Gemmer function nonlinear fitting on diopter dynamic development values represented by cornea and eye axis parameters in the second stage within preset time to obtain curve parameter gamma, and if the fitted parameter gamma is larger than the parameter gamma of normally-developed eyeball_stdIf yes, judging that the diopter is abnormal in development, otherwise, judging that the diopter is normal in development.

The invention has the beneficial effects that: the invention provides a change mechanism of eye diopter during development, and defines a method for identifying abnormal development of eye diopterObtaining a fitting parameter gamma by adopting a Westingmader function, and obtaining a parameter gamma of a normally developing eyeball according to the fitting parameter gamma and the parameter gamma of the normally developing eyeball_stdComparing if gamma is larger than gamma_stdDiopter dysplasia can be judged; otherwise, judging the development to be normal; the method can dynamically monitor and judge the diopter development condition, and control the diopter development to be lower than a set threshold value. When the ametropia is judged to be normal, the method enters a prevention and treatment stage, the diopter development is controlled to be lower than a set threshold value by adopting the method, and the aim of prevention and control is achieved through regular treatment.

On the basis of the technical scheme, the invention can be further improved as follows.

The invention also provides a device for restraining the over-rapid development of eye diopter, a multi-stage defocusing compensation training lens and a sighting target instrument, wherein the multi-stage defocusing compensation training lens is obtained by the following method: step (1), testing the adjustment reserve and the fusion reserve of the vision to be tested by adopting optometry equipment to obtain the upper limit of the practicable compensation triggering total amount; the upper limit of the total executable compensation triggering amount is 1/3-1/2 of the adjustment reserve and the fusion reserve obtained by the optometry equipment.

Step (2), the implementable compensation triggering total quantity is subjected to n-level differential series discrete, and a discrete function is provided

Wherein Acco is the total amount of compensation triggers that can be implemented, Acco_iCompensating for the triggering quantity, tolerance, for each stage

Obtaining multiple levels of myopic defocus compensation training lenses, i.e. including obtaining by acco₁Ophthalmic lens, acco₂Ophthalmic lens, acco₃Ophthalmic lenses, acco₄Lens … … acco_nThe lens comprises a plurality of stages of myopic defocus compensation training lenses.

First-stage compensation lens acco₁Starting to perform trigger compensation training, and after clear imaging is acquired, starting to gradually increase the trigger compensation training of the compensation lens step by step. First-stage compensation lens acco₁The size of the optical system can be obtained by implanting an emmetroscope in front of eyes of a user through the comprehensive optometry table for relative adjustment test; the relative regulation test process includes the first obtaining the best near vision of the testee, the subsequent increasing the positive spherical lens in the interval of +0.125DS before the testee until the testee cannot see the visual target, and the added spherical lens degree, i.e. acco₁. The n-level differential stage number is discrete and can be according to acco₁And the tolerance and the compensation total amount are jointly calculated and determined.

The invention obtains the diopter development status by identifying the method of the abnormal development of the diopter, wears a multi-stage defocusing compensation training lens on a user by adopting the device for restraining the excessively fast development of the diopter, and carries out triggering compensation training by matching with a visual target on a visual target instrument for observing the set distance, so that the diopter is changed into a stable restraint state, the parameter gamma is reduced, the diopter development is controlled to be lower than the set threshold value, and the purposes of prevention and treatment are realized.

The device for restraining the excessive diopter development further comprises a light box and a sighting mark display screen, wherein the sighting mark display screen is provided with sighting marks filled with green light.

The device for restraining the excessive development of diopter is characterized in that the sighting target display screen is provided with a sighting target filled with red light.

The device for restraining the excessive development of diopter further comprises an E-shaped sighting mark in a triangular area, an E-shaped sighting mark in an arc area and a digital sighting mark in a circle area of the sighting mark display screen.

According to the device for restraining the over-rapid development of diopter, the size of the sighting target E in the arc-shaped area is 5 meters; the size of the digital sighting mark in the circular area is designed according to a 30 cm near vision sighting mark; the size of the E-shaped sighting mark in the triangular area is set to be 3 m, 4 m and 5 m standard sighting marks according to the sight distance.

The invention provides a method for identifying abnormal development of eye diopter, which obtains a curve curvature parameter gamma through the fitting of a West grammes function, further judges whether the diopter is normally or abnormally developed, intervenes through a device for restraining the excessive development of the diopter, and triggers the defocusing compensation of human eyeballs through set conditions on the basis of a visual learning thinking mechanism so as to realize clear focusing, restrain the excessive development of the eye diopter, limit the development of the eye diopter below a set threshold value and reduce the curve curvature parameter gamma of the West grammes.

Drawings

FIG. 1 is a schematic diagram of an optical imaging path and defocus compensation for the human eye;

FIG. 2 is a diagram of a compensation path for a sharpened imaging signal received by the human eye;

FIG. 3 is a compensation path for a human eye receiving a blurred imaging signal, and also shows a path diagram of the human eye with too fast diopter development due to imaging blur;

fig. 4 is a diagram illustrating that the device for restricting the excessively rapid diopter development triggers the defocus compensation mechanism to restrict the path of the excessively rapid diopter development.

Fig. 5 shows the curve characteristics of the human eye diopter development change with age under different diopter parameters and the fitting results, wherein the curve includes diopter development curves under normal or abnormal cornea diopter parameters and normal or abnormal ocular axial length parameters.

Figure 6 is an experimental result of eye diopter versus age curves and effectiveness for normal individuals, myopic individuals and interventional myopic individuals employing the device for constraining excessive diopter development of the present invention.

Fig. 7 is a flowchart of interventional therapy of a method for identifying abnormal diopter development and a device for restricting excessive diopter development according to an embodiment of the present invention.

Fig. 8 is a schematic diagram of the arrangement of optotypes in an optotype meter in the apparatus for restraining excessive diopter development.

Fig. 9 is an enlarged schematic view of fig. 8A.

In the drawings, the components represented by the respective reference numerals are listed below:

1. the E-shaped sighting target in the triangular area, the E-shaped sighting target in the arc area, and the digital sighting target in the circle area are respectively arranged at the three sides of the triangular area and the arc area.

Detailed Description

The principles and features of this invention are described below in conjunction with the following drawings, which are set forth by way of illustration only and are not intended to limit the scope of the invention.

Example 1

As shown in fig. 5, the human eye's diopter changes with age in different dioptric parameters. This curve can be roughly classified into 3 stages according to age, an initial stage (first stage) of 0-3 years, a developmental stage (second stage) of 4-15 years, and a stable stage (third stage) of 16-35 years.

The diopter change characteristics corresponding to the first stage have universality, namely, the diopter of most human eyes changes from +12.00DS to +3.00 DS. The diopter change corresponding to the third stage is characterized by constant and small change rate, and the diopter development of the eyes at the stage is basically stable. For all human eyes, the diopter change corresponding to the second stage is severe and is influenced by genetic factors and environmental factors to different degrees.

The change of the curvature of the curve in the second stage directly determines the final diopter height of the human eyes. The genetic factors determine the corneal diopter, generally speaking, the corneal diopter is more than 45.00D and is called as keratoconus; corneal power less than 41.00D, called applanation; the normal human cornea is approximately 43.50D in diopters. Environmental factors influence the development of the length of the ocular axis.

According to the demographic average, the ophthalmology sets up the normal standard eye axis length reference of each age, and the eye axis length is over-long when the eye axis length is larger than the standard value. The human eyes are divided into 4 types according to the combination of the cornea diopter and the eye axis length, including normal cornea + normal eye axis (combination 1), normal cornea + extra long eye axis (combination 2), flat cornea + extra long eye axis (combination 3), and normal cornea cone + eye axis (combination 4). It is found from fig. 5 that the change in curvature of the curve in the second stage of eye power is larger in all of the other 3 combinations than in combination 1, and is largest in combination 2.

Table 1 shows the diopter development history of the 4 combinations of corneal diopter and axial length of the eye without intervention, and records the test results at different stages from 1 year to 30 years of age.

TABLE 1 combination of 4 refractive parameters without intervention treatment the refractive development history of the eye

The method for identifying the abnormal development of the diopter comprises the following steps:

The growth and development of the diopter of the eye are simulated,

δ is the ocular diopter of the first stage, corresponding to ocular diopter at age 1, in metric units, D;

gamma is a curve curvature parameter and is dimensionless, the parameter gamma is used for representing the change slope of the curve at the second stage, and the larger the parameter gamma is, the higher the slope of the curve is;

n is age, metric unit is, age;

2) and performing West-Grammer function nonlinear fitting on diopter dynamic development values represented by cornea and eye axis parameters in a preset time in the second stage to obtain a curve parameter gamma, and obtaining the curve parameter gamma under 4 combinations of refraction parameters, wherein the curve parameter gamma is shown in a table 2.

Table 24 refractive parameter combinations sigma function fitting results

From the fitting result, the control parameter gamma of the curvature change of the second stage of the 4 combined curves₁＜γ₄＜γ₃＜γ₂The fitting conclusion perfectly matches the actual development status, i.e. combining 2 parameters gamma₂Maximum, representing combination 2 with extra long axis and most intense diopter development, and combination 1 with parameter γ₁At a minimum, representative combination 1 is normal eye diopter development and normal eye axial length. It can be seen that the curve parameter γ of the sagger's function is a core parameter for evaluating the dysplasia of the diopter of the eye. Although the existing view proposes the length of the axis of the eye as the basis for judging the excessive diopter of the myopia, the axis of the eye is a static parameter, and the curve parameter gamma of the West-Gummer function is a dynamic parameter for representing the change rate, so that the aim of dynamically monitoring the diopter of the teenagers is better matched.

Therefore, the invention provides a curve parameter gamma of the West Gem's function as a basis for identifying the abnormal development of the diopter of the eye, the nonlinear fitting of the West Gem's function is carried out on the dynamic development value of the diopter of the teenagers to obtain the curve parameter gamma, and if the fitted parameter gamma is larger than the parameter gamma of the normally developed eyeball_stdAnd diopter dysplasia can be judged. Otherwise, judging the development to be normal.

Example 2

As shown in fig. 6, curves of the diopter scale of the eye versus the age of the normal individuals, the myopic individuals (control group) and the myopic individuals (test group) to which the intervention treatment representing the device for restraining excessive diopter development of the present invention was applied were also experimental results representing the effectiveness of the device for restraining excessive diopter development of the present invention and the intervention treatment.

The diopter of the eyes of the myope develops into the geometric superposition of the genetic factor influence and the environmental factor influence, the genetic factor and the environmental factor influence simultaneously act, the genetic factor is controlled by the congenital condition, and the subsequent cannot form constraint on the genetic factor, so that the method only makes constraint aiming at the influence of the environmental factor.

Table 3 shows the refractive parameters for the different refractive conditions in figure 6. Through the parameter fitting of the sitcomold curve, the parameter gamma of the control group is-0.17753, the parameter gamma of the sitcomold curve representing the diopter of the normally developed eye is-0.2309, the former is obviously larger than the latter, and the condition that gamma is larger than gamma is met_sadThe control group was judged as ametropia.

TABLE 3 Normal individuals, myopic individuals (control group) and interventional myopic individuals

The test group and the control group were sampled in parallel. The trial set was first tested for amplitude of accommodation and vergence amplitude using a phoropter stand, and the total amount of compensation triggers were obtained as 1/2 for amplitude of accommodation and vergence amplitude, i.e., 13.2/2 ═ 6.10D. For compensation triggering, n-21 arithmetic progression deviations (tolerance d-0.25 DS) are carried out, 0.25 being a customary tolerance in optical optics, and acco is determined₁= 1.25DS to obtain acco₁The lens is used for setting the distance to observe the sighting target on the sighting target instrument, carrying out trigger compensation training for a preset number of days (such as two days), causing the focusing position of the eye of the patient to move towards the front of the retina, and forming sharpened imaging sharpen₁. Administering the acco to the wearer₂Ophthalmic lenses, acco₂And (5) carrying out trigger compensation training to obtain a second-level optical signal sharpening sharpen₂Progressively graded in an internal cyclic manner, acco₃，acco₄……acco₂₁Finally, the accumulated total sharpening amount of the optical signal is 6.25DS (the maximum approximately equals 6.1DS)

A tolerance of 0.125 may also be used, and finallyTo accumulate to 6.125 DS. Because the sharpening of the optical signal can eliminate the negative increase of the diopter of the eyes of the patient caused by environmental factors in the annual development period, the sharpening becomes a constraint state which is kept stable.

Under the geometric superposition of the constraint state and the genetic factors, the gradual decline of the eye diopter year by year is formed. Starting from age 4, the test group developed year-by-year trigger compensation training and achieved diopter restraint success. The final reduction in diopter after 16 years of stabilization was 6.00 diopters with a moderate down-level control of myopia (-2.25DS) compared to the control, greatly reducing the incidence of high myopia and a range of retinal complications.

And carrying out Schugermore function nonlinear fitting on the test group to obtain a second-stage curve change rate parameter gamma-0.1912. The main improvement of the proposed intervention is the curve rate of change of the second phase of the sigma-delta function, gamma, compared to the control group, gamma-0.17753. Specifically, the parameter γ should be reduced, reducing the diopter development rate. Therefore, the emphasis of the present invention is applied to the age group corresponding to the development of diopters during the age of 4-15 which controls the second stage in the curve of the West Gimmed function.

As shown in fig. 7, the method for identifying abnormal diopter development and the device for restricting diopter development faster than that of the present invention are schematic in the flow of triggering compensation training.

As shown in fig. 8 and 9, the visual target display screen in the device for restraining the excessive diopter development comprises an E-shaped visual target in a triangular area, an E-shaped visual target in an arc area and a digital visual target in a circle area. The size of the sighting mark in the arc-shaped area E is 5 meters of standard sighting marks; the size of the digital sighting mark in the circular area is designed according to the 30 cm near vision sighting mark; the size of the E-shaped sighting mark in the triangular area is set to be 3 m, 4 m and 5 m standard sighting marks according to the sight distance.

In order to enhance the treatment effect of the sighting mark in the area, the E-shaped sighting mark in the triangular area can be filled with green or red. Filling in green may cause the optotype to emit green light and filling in red may cause the optotype to emit red light. The green light and the red light have different corresponding wavelengths, and by utilizing the chromatic aberration principle of a human eye dioptric system, the green light can trigger the myopic defocusing when being focused in front of the retina, and the red light can trigger the hyperopic defocusing when being focused behind the retina.

Therefore, the treatment effect can be enhanced by using green light in the treatment process of the myopia patients. In addition, the size of the visual target in the arc-shaped area E is 5 meters of standard visual targets, so that the visual target is convenient for a patient to test and judge the quality of vision. When the vision test result is stable, the diopter development stability can be indirectly inferred; when the vision test result is reduced, the diopter development can be indirectly presumed to be faster. For the size of the digital optotype within the circular area, a 30 cm near vision optotype design may be used to treat the diopter constraint of a accommodative lag patient.

The above description is only for the purpose of illustrating the preferred embodiments of the present invention and is not to be construed as limiting the invention, and any modifications, equivalents, improvements and the like that fall within the spirit and principle of the present invention are intended to be included therein.

Claims

1. A method of identifying abnormal development of eye diopters, comprising the steps of:

The growth and development of the diopter of the eye are simulated,

delta is the diopter of the first stage, corresponding to the diopter of the eyes at the age of 0-3, and the metric unit is D;

n is age, metric unit is, age;

2) performing West-Gem function nonlinear fitting on diopter dynamic development values represented by cornea and eye axis parameters in a preset time in the second stage to obtain a curve parameter gamma, and if the fitted parameter gamma is larger than the parameter gamma of the normally developed eyeball_stdIf yes, judging that the diopter is abnormal in development, otherwise, judging that the diopter is normal in development.

2. The device for restraining the excessive rapid development of the diopter of the eye is characterized by comprising a multi-stage defocusing compensation training lens and a sighting target instrument, wherein the multi-stage defocusing compensation training lens is obtained by the following method: testing adjustment storage and fusion image storage of vision to be tested by adopting optometry equipment to obtain an upper limit of an implementable compensation triggering total amount, wherein the upper limit of the implementable compensation triggering total amount is 1/3-1/2 of the adjustment storage and the fusion image storage obtained by the optometry equipment;

And obtaining the multi-stage myopic defocus compensation training lens.

3. The apparatus of claim 2, wherein the optotype instrument comprises a light box and an optotype display screen, and the optotype display screen has optotypes filled with green light.

4. The apparatus of claim 3, wherein the visual target display screen has a red-filled visual target.

5. The apparatus according to claim 3, wherein the sighting target display screen comprises an E-shaped sighting target in a triangular area, an E-shaped sighting target in an arc area and a digital sighting target in a circle area.

6. The apparatus according to claim 5, wherein the size of the E optotype in the arc area is 5 m standard optotype; the size of the digital sighting mark in the circular area is designed according to a 30 cm near vision sighting mark; the size of the E-shaped sighting mark in the triangular area is set to be 3 m, 4 m and 5 m standard sighting marks according to the sight distance.