CN112402808A

CN112402808A - Parasite inactivation device

Info

Publication number: CN112402808A
Application number: CN202011299673.3A
Authority: CN
Inventors: 尹垚懿
Original assignee: Individual
Current assignee: Individual
Priority date: 2020-11-18
Filing date: 2020-11-18
Publication date: 2021-02-26

Abstract

The invention relates to a parasite inactivation device comprising: a base portion and a handle portion; one end of the lamp holder part is connected with one end of the handle part in a clamping way; the lamp head part is internally provided with a light source generator, the handle part is internally provided with a photoelectric control unit and a power supply, or the lamp head part is internally provided with a light source generator and a photoelectric control unit, and the handle part is internally provided with a power supply; the photoelectric control unit is detachably connected with the power supply; the photoelectric control unit is detachably connected with the light source generator and can control the light source generator to emit light; the light source generator is provided with a first light source generator and a second light source generator. The light source generator is matched with the visual protective cover for use, and the visual protective cover has a light filtering function and can see bacterial fluorescence through a light filtering effect. The device provided by the invention not only can detect parasites and germs on plants and vertebrates, but also can kill germs and pests on plants and vertebrates.

Description

Parasite inactivation device

Technical Field

The invention belongs to the technical field of medical instruments, and particularly relates to a parasite inactivating device.

Background

There are more than about 200 parasites of humans and animals, such as superficial fungi;

superficial mycoses are skin diseases caused by pathogenic fungi that parasitize keratin tissues, the pathogenic bacteria of which can be classified as:

firstly, dermatophyte: pathogenic fungi that parasitize the keratin tissue of the skin are collectively referred to as dermatophytes. The bacterium is subdivided into the following three genera according to the difference of invading tissues and the difference of culture characteristics:

(one) Trichophyton: invading the skin, hair and nails. The genus has been identified with 13 species that cause disease in humans. Common trichophyton fulvum, trichophyton rubrum, trichophyton clinopodium, trichophyton purpureum, trichophyton gypseum, etc.; they are characterized by a rod-shaped macromolecular spore with smooth walls.

(II) Sporosporium: attack hair and skin, and are most common in China, such as microsporidia ferruginea, microsporidia laniformis and the like; these cultures are characterized by spindle-shaped macromolecular spores with thorns on the walls. There are 8 types reported to cause disease in humans.

(III) Epidermophyton genus: invading the skin and nails. Only one species of Epidermophyton floccosum is responsible for the pathogenesis of human beings, and its culture shows pestle-like or pear-like macrospores.

The dermatophytes of the three genera can cause tissue reaction after infecting human body, and then the papule, blister, scale, broken hair, alopecia, deck change and the like can occur. According to the difference of the invasion sites, the clinical classification can be tinea capitis, tinea corporis, tinea cruris, tinea manus, tinea pedis and onychomycosis.

II, trichophyton hornerianum: the pathogenic fungi that parasitize the cuticle or hair shaft surface of the skin can be called trichophyton. The ringworm is divided into two types, namely horny layer type and hairline type. The former includes tinea versicolor, Microcorynebacterium erythraeum, Trichophyton mansoni and Trichophyton Venezans; the latter type has Trichophyton axillaris.

Since the fungus Trichophyton angustifolium is parasitic on the surface of human tissue, it generally does not cause inflammatory reaction of the tissue, and is very slight if any.

[ epidemiology ]

Superficial mycosis is very popular, is spread in all regions of the world and is a common frequently-occurring disease in China. The incidence of the skin diseases accounts for the second or third, some or even the first place of the total number of the patients out-patient department of dermatology according to the reports of several hospitals in Shanghai; according to the data of investigating the disease condition of the Yunnan Gui front line officers in 1985, the data of a certain team is as follows: 2370 officers and soldiers in a certain group army are subjected to physical examination by the hospital, and the result shows that 1414 people suffering from superficial mycosis account for 59.66% of the examined people; in the statistics of 40731 cases of primary clinic cost in 1964 with 1955-. Superficial mycosis occurs in connection with the type of species, the antibacterial power of an individual and the environment, but the skin diseases are prevalent and have high incidence, and the reason for this is probably related to the following factors:

the fungus has extremely strong viability: the strain does not contain chlorophyll and has no photosynthesis ability, and only depends on parasitism or saprophytosis for survival. The fungi prefer a moist and warm environment, the optimum growth temperature is 25-26 ℃, and the fungi can grow under the condition of the pH value of 3.0-10.0. Although weak against high temperature (above 45 ℃), it has strong adaptability to temperature below 4 ℃, and also has considerable resistance to ultraviolet rays, radioactive rays, etc. It follows that fungi are not critical to the living conditions. Therefore, people can culture and detect pathogenic fungi from the atmosphere, animal and plant bodies, human excrement, floors, soil and the like, in short, the fungi have extremely strong living capability and almost do not exist everywhere in the nature, so the opportunity of the fungi to infect human beings is naturally increased.

Secondly, the bacteria carriers are the main reason for epidemic propagation of superficial mycosis: because people do not know enough about the harmfulness of tinea, people do not pay attention to it and often have diseases to do its development. In terms of tinea pedis, most patients have a very mild and itching condition, so the patients do not seek medical attention actively; even if some patients have more obvious symptoms, the patients still cannot be treated actively, and are familiar with the symptoms due to long-term illness; some patients are cured after treatment, but the infection source is not controlled and no preventive measures are taken, so the patients are frequently relapsed. The three people exemplified above are carriers, which ultimately have consequences: tinea may be caused by autoinfection in other places; can be spread to the surrounding people through various ways for the society.

Thirdly, the spread of pathogenic fungi is so extensive that prevention is rather difficult: the spread of the disease is quite extensive, and pathogenic bacteria can be spread widely through public articles such as slippers, bathtubs, footbaths, towels, haircutting tools and the like. Therefore, the public places need strict management and disinfection measures, otherwise, the control of the disease incidence is not easy to realize.

Fourthly, the strength and weakness of the body resistance also have the effect of being not overlooked to the epidemic: although it is considered that the fungal infectivity is low, the disease is not easily developed even between toes if no wound is locally generated. But it is well known that people who are not personal hygiene are not concerned; patients with systemic diseases, such as diabetes, malignant tumor, etc.; for long-term use of corticosteroids, immunosuppressants and antibiotics. The above will certainly promote the occurrence of tinea.

Fifthly, the external environment also has an important relationship with the prevalence of tinea: fungi prefer to grow and reproduce in a humid environment, so the disease is easy to occur among toes and is frequently attacked or aggravated in a damp and hot area and in hot summer.

As can be seen from the above description, superficial mycosis is a disease with a certain infectivity, which can be either a self-infection or a contagious infection. The infection mode of the disease is as follows: once infected by direct contact, the onset of tinea capitis is often caused by direct contact with children suffering from tinea capitis or animals suffering from tinea; the second is indirect infection, for example, tinea may occur when the patient frequently uses the articles used by tinea such as slippers, pillow towels, foot-rubbing cloths, etc.

[ clinical types and symptoms ]

Tinea capitis (Tineascapis)

The disease is a superficial mycosis which occurs in the skin and hair of the head, and the tinea capitis is basically divided into four types in China, namely, yellow tinea, white tinea, black tinea and purulent tinea.

Tinea pedis

The pathogenic bacteria of tinea flavum are dermatophytes and Mongolian variants thereof. The morbid stage of the disease is called alopecia, and the disease is called glaucomatosis in south China, the disease is mainly popular in rural areas, and is mostly seen in children of 7-13 years old, and the ratio of male and female is 9: 1, but may also occur in adults and adolescents.

The disease occurs in the scalp, where the initial lesions are papules or pustules, and then dry, crusted and yellowish in color. Scabs can spread and enlarge, being as large as soybeans or larger. The scab has a similar appearance to a dish, and has a slightly raised periphery and a slightly depressed center with hairs penetrating through the scab, which is called a yellow ringworm composed of a group of yellow ringworm, sebum, scales, and dust. Is an important characteristic of psoriasis and is helpful for diagnosis. Meanwhile, the disease is also suggested to have strong infectivity, and isolation treatment is often needed. The scab like bean dregs is easy to be crushed and smells mouse smell, which is another characteristic of the disease. The adjacent scabs can fuse with each other to form a large gray yellow thick scab, and if the scab is scraped off, the scab can be flushed and wet or superficial to a sexual ulcer, if the hair follicle can be damaged without treatment, and the atrophic scars are left after healing.

The affected hair is dry, withered and bent, and has loose hair loss, but no broken hair is found. No matter how serious the patient suffers from the disease around the scalp, a normal hairband about 1 cm wide is still remained at the hairline, and the hair can not be affected at the hairline.

The psoriasis has itching and lingering course of disease, and cannot be self-healed until adults are treated if the psoriasis is not treated. Erosion and suppuration may be accompanied by swelling of the cervical lymph nodes. Except for the head, the face, neck, trunk and nails are occasionally involved. In severe cases, allergic reactions may also occur, and sometimes a rash occurs throughout the body, which is called tinea and fungus rash.

The disease can be detected by ultraviolet lamp irradiation, and the affected part shows dark green fluorescence, and the disease is detected by endoscopic extraction to obtain endophytic hyphae, and spore or antler-like hyphae can be detected by yellow tinea crust.

Tinea alba (II)

Tinea alba is mainly caused by infecting microsporum pucciniae in China. Is often popular in township centers or primary schools. Almost all are childhood onset.

Scalp lesions are scales and speckles, while small ones are like broad beans, large ones are like coins, and the scales spread and enlarged with time, and are mostly irregular in shape. The inflammatory reaction is not obvious in the lesion, but the boundary is clear. The disease is dry and the loss of luster is usually mainly caused by broken hair, which is different from tinea pedis. The hair is broken at a position 2-5 mm away from the scalp, and white fungus sheaths can be seen on the outer surface of the hair shaft of the affected part close to the scalp, and the hair is also formed by fungi and is considered as one of the characteristics of the disease.

The affected area can be irradiated by the filtered ultraviolet lamp to show bright green fluorescence. When the hair is examined by microscope, the appearance of the spore is determined, and when the fungus is examined by microscope at the early stage of picking the dandruff, the spore is always positive. 97% of the culture was microsporidia ferruginea, the rest being other microsporidia.

The patients feel itchy or have no obvious symptoms, the disease course is chronic, the patients can self-heal by the adolescence usually without treatment. This may be associated with vigorous sebum secretion and increased local free fatty acid concentration in young people to inhibit fungi. After the disease is cured, new hair can regenerate without leaving scar.

(III) tinea nigra

The pathogenic bacteria of the disease are purple trichophyton or trichophyton clinopodium polycephalum. Mainly invade children, and the incidence rate of the children is behind tinea alba and tinea flavum.

Head damage is similar to tinea alba and appears as scale plaque, but the lesion area is smaller and the number is more than that of tinea alba. In addition, the diseased hair is slightly different from tinea alba, mainly presents low-position broken hair, is usually broken at a position 1-2 mm away from the scalp, and even breaks when one scalp breaks. The affected part is observed to have only black residual hair roots, so the product is named as tinea versicolor.

The disease has no fluorescent appearance to the test of the filtered ultraviolet lamp. The disease is picked up and detected as endospore, and hypha can be detected in early dandruff. 80% of the cultured trichophyton purpureum is cultured, and 20% of the cultured trichophyton clinopodii is cultured.

The infectivity of this disease is weaker than that of yellow ringworm and white ringworm. Itching or no discomfort. The disease course is slow, a few scars are left after the recovery, and the hair part is bald.

(IV) tinea pedis

Tinea pedis is caused by infection with an animal-tropic fungus, such as microsporidia gypseum or microsporidia lanuginosus.

Most of the skin lesions are large and swollen like large-block-shaped carbuncles, the inflammatory reaction is severe, the hair follicles of the affected parts are suppurative, and pus can be squeezed out from the hair follicles. The hair at the diseased part is easy to break and fall, and the residual hair is very loose and is not easy to pull out. Scars are often left after recovery, and the fungus microscopic examination and culture are positive when the disease is cured.

The subjective symptoms of the disease usually indicate pain or itching. Swelling of the cervical lymph nodes is often accompanied. Some patients also have general symptoms such as fever, lassitude and inappetence.

Second, tinea corporis (Tineaccorporisis)

The skin diseases caused by dermatophytes infected by the smooth skin on the surface of the human body except the head, the palms and soles, the groin, the negative part and the nails are collectively called tinea corporis. Also named as Yuanxuan or jin Qian Xuan Ling. The common pathogenic bacteria of the disease are trichophyton rubrum, trichophyton gypseum, epidermophyton floccosum, trichophyton purpurea and the pathogenic bacteria of tinea capitis.

Tinea corporis is common to children and then teenagers. The clinical manifestations of this disease are related to the species of pathogenic fungi and individual reactions. The rash begins to become erythema or papule, and then the lesion gradually becomes telecentric and expands to the periphery, the center of the lesion tends to be self-healing, and the lesion becomes annular for a long time. The rim of the ring is slightly elevated compared to the adjacent normal skin where inflammation is more pronounced with small papules, blisters or scales attached. Sometimes, the rash may appear in the center of the ring, and the new skin lesion gradually expands into the ring, so that a plurality of concentric rings are formed in succession, which makes the border clear. The difference in the size and number of the skin lesions is also variable, with 1-2 or more lesions, the general occurrence of the disease is less common, and the distribution is also asymmetric. However, if the patient has an immunodeficiency or is chronically administered corticosteroids and immunosuppressants, the rash may appear to spread systemically.

In addition, the clinical common occurrence of the tinea corporis is called as atypical tinea corporis at present, which is caused by adopting corticoid cream to coat the lesion part by the self-advocate of patients who are misdiagnosed with other skin diseases or tinea corporis. After a period of treatment, the typical symptoms of the pathogenic tinea are destroyed, and instead, the inflammation reaction is severe, the lesion range is rapidly enlarged, the form is irregular, the boundary is not clear, and the tinea corporis is not easy to be identified, so the name is. This is due to the spread of pathogenic fungi caused by the decrease of local skin immunity after the use of corticosteroids. In this regard, it is difficult for an inexperienced physician to make a correct diagnosis.

Patients with tinea corporis have itching, and bacterial infection can occur after scratching. The scales around the lesions were scraped and microscopically examined to reveal hyphae or spores.

Tinea cruris (Tineacruris)

This disease can be considered as a special form of tinea corporis occurring on the medial aspect of the upper thigh. The pathogenic bacteria are common epidermophyton floccosum, and other dermatophytes can also cause diseases.

Tinea cruris is mostly male in adults and is rarely seen in women. Usually one-sided, also two-sided symmetrical distribution. In severe cases, the skin lesions can spread upward and directly reach the lower abdomen; spread backward to the buttocks; extending downward to involve elsewhere in the thigh.

The disease is different from tinea corporis in the following points: the tinea cruris damage form is rarely circular or elliptical, and is mostly irregular or arc; secondly, the tinea cruris skin lesion is often expressed as lichenification or acute and subacute eczematoid lesion; tinea cruris is easy to cause bacterial infection; the itching of tinea cruris is more severe.

Tinea cruris is generally caused by tinea pedis or tinea manuum infection, the disease condition is related to seasonal changes, and the tinea cruris usually recurs or aggravates in summer and can be relieved in winter. The course of the disease is lingering, and patients need to be treated with heart-fast treatment to recover, otherwise, the disease is easy to recur.

Tinea pedis and Tinea pedis (Tinea pidis)

The tinea pedis is the most common superficial fungus pathogen caused by foot infection by pathogenic fungi, and is called beriberi or damp among China folks. The main pathogenic bacteria of the disease comprise trichophyton rubrum, epidermophyton floccosum, trichophyton gypseum, trichophyton roseum and the like. In addition, the cause of Candida albicans is also frequently reported.

The tinea pedis is mainly caused by middle-aged and young-aged pathogenic bacteria. Children and older patients are less common, which may be associated with less activity and drier toes in these individuals.

The pathogenic bacteria can be easily found between the toes, especially in the third and fourth toe gaps. This is related to the close contact, wetness, stuffiness of the skin and poor evaporation of sweat from the above mentioned areas. The manifestations of tinea pedis skin damage are generally classified into the following three types:

(one) blister type: the deep blisters from the needle to the large millet are visible between the toes and on the sole, and the blister wall is thick and is scattered or densely distributed, so that the adjacent rashes can be fused to form a large blister. The bleb liquid is naturally absorbed and dried to be converted into scales.

(II) toe erosion type: it is commonly found in the space between the toes, and the affected area is damp and sweaty. The rash is initially soaked, and it is eventually accompanied by erosion, flushing and moistening due to pruritus or damage of epidermis after rubbing. It can cause the leakage of liquid to cause unpleasant odor.

(III) scale keratotic type: it is quite common to invade the sole, lateral foot, toe and heel. Skin damage is manifested as scaling, thickening of cutin, roughness and hardness, and chapping between skin, and the nature of the disease is particularly severe in winter.

The three types of skin lesions are often mixed and seen, but mainly one of them is called tinea pedis. For example, the blister type is marked by blisters, and erosion or scaling is observed in the meantime.

This disease is marked by the symptoms of itching, especially blister type and erosion between toes. The onset of tinea pedis is season dependent. It is usually mild in winter and heavy in summer. In summer, the skin becomes allergic to bacterial infection, which may cause ringworm and fever.

Tinea manuum (Tieea mauns)

Tinea manuum is a superficial mycosis occurring on the palm surface, and is similar to the "fungal infection of the hand" in traditional Chinese medicine. Can be primary, but most are transmitted from tinea pedis itself. The pathogenic bacteria are the same as tinea pedis, and the clinical manifestations are almost the same as tinea pedis. Because the hands are exposed and the ventilation is much better than that of the feet, no interphalangeal erosion type appears clinically, but only the blister type and the scale keratosis type are seen. The clinical occasional erosion appears, but is usually caused by candida infection, but not by dermatophyte.

Sixthly, onychomycosis (Tineauguium)

Onychomycosis is caused by infection of tinea unguium with dermatophytes, commonly known as onychomycosis; if the lesion on nail caused by non-dermatophytes is called onychomycosis. Both onychomycosis and onychomycosis have been frequently mixed.

Onychomycosis lesions begin at the distal end, lateral margin, or fold of the nail. Appearing as abnormal nail color and morphology. Almost grey white and tarnished; the thickness of the deck is obvious, and the surface is uneven. It is loose and crumbly, often with keratin and debris deposited beneath the nail. Sometimes, the deck may be separate from the nail bed. In addition, a special type, fungal onychomycosis, is seen clinically. This pattern appeared not to thicken, but only a punctate white turbidity developed on the nail surface, followed by gradual expansion to spread to the entire nail. Onychomycosis is slow in disease course and can be treated for a lifetime if it is not treated. Onychomycosis is characterized clinically by increased thickness of the nail plate, visible transverse furrows in the surface, and sometimes brown. There was still a shiny feel without the deposition of inferior keratin and debris common to onychomycosis. Meanwhile, paronychia is accompanied with red swelling around the nail, and pain and tenderness are felt. There is little liquid seepage in the paronychia but no suppuration. The pathogenic bacteria of the disease are candida or aspergillus, and the fungi culture is needed to confirm the pathogenic bacteria.

Seventhly, tinea versicolor (Tieeaverersicolor)

The tinea versicolor is named purple-white palatine wind in traditional Chinese medicine because purple spots and white spots exist alternately. In view of the fact that the skin rash is flaccid in summer, the skin rash is also commonly called as sweat stain. This disease is caused by tinea versicolor parasitic to the horny layer of the epidermis. The bacterium is lipophilicity, and the previous culture often fails, so that the successful culture is frequently reported in China. It has also been proposed that tinea versicolor should not fall within the fungal category from the standpoint of fungal classification. Then, the skin disease caused by it should not be called tinea, so it is recommended to change to pityriasis versicolor.

Tinea versicolor is commonly found in the neck and chest and back. Sometimes, the proximal end of the upper limb is also affected. The basic lesion is macula, as large as soybean. The new rash was yellow brown or tan, and the old lesion was off-white. The surface of the skin lesion is attached with trace bran-like scales, and adjacent skin diagnoses can be fused with each other to form a larger irregular focus. Generally, the patients have no subjective symptoms, and occasionally have itching feeling when sweating.

Eight, tinea fungus eruption (Dermatophytides)

The dermatophyte rash refers to a rash of the fungus or its products occurring in other parts of the human body from the primary focus of menstrual blood circulation, and is an allergic reaction. The following conditions must be provided for this disease: there is an active primary fungal lesion; finding a fungus negative in the lesion of the ringworm rash; the condition of the ringworm rash is improved until the disease is resolved along with the control of primary active fungus focus; positive ringworm test must be used, but negative results can be excluded to the diagnosis of ringworm rash.

Tinea and fungus eruption can be divided into general type and limited type. The former rash is lichen-like rash, i.e. symmetrical and disseminated papules appear on the whole body, which are consistent with hair follicles and are about from needle head to chestnut grain; the latter type is mostly manifested as a pompholyx-like eruption, and is characterized by the occurrence of scattered or clustered deep blisters on the palms and the soles of the fingers, which are of different sizes, and the blister walls are not easily broken, and a few blisters, such as those larger than soybeans, can develop into larger blisters.

The disease is hard to feel itchy, and is especially more obvious in limitation type. In addition, other types of eruptions such as erythema multiforme, erythema nodosum are rare.

[ DIAGNOSIS AND IDENTIFICATION ]

Superficial mycosis can be diagnosed easily according to the medical history and clinical manifestations. If necessary, the following auxiliary checks can be carried out:

firstly, fungus microscopic examination: selecting scales or diseased roots at the edge of the skin lesion. Put on a glass slide, add a drop of potassium hydroxide solution, and cover the slide. Then placed on an alcohol lamp and heated for a while to promote keratolysis. And finally, microscopic observation is carried out. A positive fungal test has a diagnostic effect, and a negative fungal test cannot exclude the diagnosis of tinea.

Secondly, fungus culture: the conventional culture medium is Sabourauad (Sabourauad) culture medium. Inoculating scales, hairs or vesicle membranes taken from the focus, and culturing in a thermostat at 25-30 ℃. Colony growth can be seen after about 5 days generally, and then strain identification can be carried out. If no colonies grow after three weeks of culture, culture negatives may be reported.

Thirdly, checking by filtering an ultraviolet lamp: the lamp, also known as wood, is a lamp in which ultraviolet light passes through a glass unit containing nickel oxide, some fungi are visible in the dark, and colored fluorescence is produced under the irradiation of a filtered ultraviolet lamp. Therefore, important reference can be provided for diagnosing superficial mycosis, particularly tinea capitis, clinically according to the existence of fluorescence and different colors. In addition, the lamp is also helpful for group inspection such as nursery.

Superficial mycoses have to be identified with numerous skin diseases, briefly described as follows:

first, tinea capitis should be distinguished from psoriasis, seborrheic dermatitis and alopecia areata. Psoriasis is common in adults and occurs less in children. Except for the pathological changes of the head, it is often affected by the extension of the trunk and limbs. The skin lesions are plaques, and thick silvery white scales are attached to the surfaces of the plaques. The hair at the lesion is in a brush pen shape, but the hair itself has no pathological changes, namely, broken hair, alopecia, dry hair, withered hair and bent hair and the like are not seen; seborrheic dermatitis is common in adults and is better at the eyebrow, nasolabial sulcus, beard, axilla, center of trunk and pubic mons. The rash is erythema, pimple, and greasy scale on the surface. No broken hair and extreme itching; alopecia areata is commonly called as ghost shaving head. Before the disease occurs, mental disorder exists, the diseased part presents in a circle-like alopecia, the boundary is clear, and the alopecia area has no inflammatory reaction and no scale. The subjective feeling of itch was not felt. The above dermatosis trichome tests were all negative.

Secondly, identifying tinea corporis, pityriasis rosea and psoriasis: pityriasis rosea occurs frequently in the proximal ends of the trunk and limbs, and the rash spreads and is distributed symmetrically, mainly manifested as erythema, lesion long axis balanced with dermatoglyph or rib, and chaffy scale attached to the surface. Negative fungus microscopic examination; psoriasis is usually aggravated in winter and remitted in summer. The remainder of the authentication is as described above.

And thirdly, tinea cruris should be identified mainly by neurodermatitis and chronic eczema. The negative results of the fungal tests of both the nervous rash and the chronic eczema show that the two diseases have no slightly higher damaged edge than the adjacent normal skin and have no phenomenon of heavy summer and light winter.

And fourthly, the tinea manus and pedis is identified by eczema and pompholyx. Eczema usually involves the dorsal aspect of the hand and foot and the extending sides of the toes, and is usually distributed symmetrically. Acute rash is polymorphic lesion, and chronic rash often has obvious infiltration, can be changed like moss, has dark red color and is generally clear in boundary; pompholyx is usually associated with hyperhidrosis, which is usually caused by the side of fingers and the edge of palm. The fungal tests of the above diseases are all negative.

[ TREATMENT ]

First, whole body treatment

Ketoconazole (mono): at present, the medicine is taken orally to replace griseofulvin. Ketoconazole is a synthetic broad-spectrum antifungal imidazole. The antifungal mechanism is that the synthesis of lysergol which is an important component of the fungal cell membrane is inhibited, so that the normal function of the fungal cell membrane is lost, the permeability of the membrane is increased, and finally, the fungi are denatured and even die. According to clinical practice, the medicine has good curative effect on superficial mycosis.

Indications are as follows: it is mainly used for treating tinea capitis, generalized tinea corporis, severe tinea cruris and onychomycosis.

Contraindications: the medicine is forbidden for pregnant and lactating women due to liver function disorder.

Dosage: adult, 200mg, 1 time/day. Weight of children is less than 20 kg, 50mg, 1

The times/day; 20-40 kg of 100mg of the compound, 1 time per day; more than 40 kg can be taken according to the dosage of adults.

(II) other imidazoles: the antifungal effect of Itraconazole (Itraconazole) is 5-10 times that of ketoconazole, and the minimum dose for treating dermatophyte is 100mg per day.

Second, local treatment

The superficial mycosis of hair or nail plate is not affected, and topical therapy can be used for treating superficial mycosis, but it needs to be durable and take long time to take medicine. Two groups of drugs are commonly used.

The general antifungal medicine for external use: more commonly, the compound is prepared into ointment, cream and tincture as required by salicylic acid, benzoic acid, tolnaftate, undecylenic acid, zinc undecylenate, sulfur, iodine and the like for clinical selection.

(II) specific broad-spectrum antifungal agents: currently, the largest group of the currently widely used medicines has a common imidazole ring, i.e., imidazoles such as sulconazole (Miconazole), Miconazole (Miconazole), oxime conazole (oxiconazole), econazole (econazole) ketoconazole (ketoconazole) Bifonazole, clotrimazole (chlorotritimidazole) and the like are often prepared into 1-2% cream for clinical application.

Specific treatment method for various superficial mycoses

Tinea capitis

China has summarized good experiences of a set of five-character therapy, namely taking (medicine), washing (hair), applying (medicine), managing (hair) and disinfecting (toxicity). In contrast to this experience, ketoconazole may be administered orally for 4 weeks at the above doses; the hair is washed daily during the treatment period; continuously applying proper antifungal ointment for 1-2 months; haircut once a week until the hair is cured; daily articles of patients, such as hats, towels, pillow towels, combs and the like, need to be disinfected regularly.

(II) tinea corporis, tinea cruris, tinea manus and pedis

It is believed that the local treatment of tinea would be effective, but different forms of tinea drugs are used according to different conditions and different manifestations of skin injury.

1. Infection should be controlled first when tinea occurs with infection.

2. When swelling and exudation of the lesion are obvious, 3% boric acid water or 0.02% furan cinnoline solution can be selected for wet application, and after swelling and exudation are reduced, an external medicine for effectively treating tinea is selected.

3. For erosion and slight exudation, Huanglian Lian Zi oxide oil should be applied externally for 2-3 days, and then proper tinea ointment should be replaced.

4. When the lesion is of scale keratosis type, the tinea treating ointment is in the form of ointment or cream.

5. If the damaged part has chap phenomenon, and the tincture is not applied externally, ointment or cream is still selected.

6. The rash is mainly erythema and pimple, and can be made into tincture, ointment or cream.

7. High concentration of keratin exfoliants is prohibited for skin lesions on the face, medial thigh, etc. to avoid irritation and dermatitis.

8. As long as the formulation is chosen without errors, the external use medicine should not be frequently changed for many principal and secondary tonics. Each tinea treating ointment should be used for at least one week.

9. For patients with intractable or extensive skin damage and difficult healing by local therapy, ketoconazole can be orally taken for 200mg for 1 time/day for 4 weeks.

(III) onychomycosis

The disease should be treated locally in principle, but the thick nail plate is not easy to permeate medicine, so the common method for treating tinea cannot be adopted. The nail plate should be removed as much as possible before topical application, and then antifungal agents should be applied. The general method is as follows:

the first nail scraping method comprises the following steps: scraping the brittle part of the diseased nail with a knife as much as possible every day, and then externally applying 5% iodine tincture, 30% glacial acetic acid or Weishi liquid for 1-2 times every day until the disease is cured.

(II) A dissolving method: firstly, the skin around the nail is protected by adhesive plaster, then 25-40% of urea ointment is coated on the nail plate, and finally, a plastic film is covered and fixed by the adhesive plaster. Changing the dressing once every 2 days, removing the nail plate with forceps when the nail plate is softened and has floating feeling, changing the dressing according to the conventional method every day, and externally applying the tinea ointment after the wound surface is healed until the nail grows out.

If the treatment fails or the number of the diseased nails is large by the method, ketoconazole can be taken orally, and the disease can be cured in about half a year as the dosage is the same as the dose.

(IV) tinea versicolor

The disease is easy to cure, but it also recurs frequently. The traditional medicine, such as 20-40% sodium thiosulfate solution and 2.5% selenium sulfide emulsion, can be used for external application, and in addition, the imidazole cream can also obtain satisfactory effects.

(V) tinea fungus eruption

The whole body treatment of the disease can be processed according to the treatment principle of allergic skin diseases; antifungal drugs are not required for local application, and particularly, strong-irritation tinea drugs are not used. The preparation can be applied topically with mild zinc oxide oil or wet-applied with 3% boric acid water. In addition, active tinea foci must be treated actively.

In conclusion, the process of killing the skin parasites is complex, the method and the drugs are diversified, pathogens are distinguished before treatment, and the cost is high. The parasites have dormant bodies, and the drugs are not absorbed during the dormancy period, so that relapse is caused.

Disclosure of Invention

Technical problem to be solved

In order to solve the problems in the prior art, the invention provides a parasite killing device, which not only can see germs and pests on plants and vertebrates, but also can assist in killing parasites and germs on the plants and the vertebrates, and has the advantages of low cost, environmental protection and remarkable effect in the whole process.

(II) technical scheme

In order to achieve the purpose, the invention adopts the main technical scheme that:

a parasite inactivation device comprising: a base portion and a handle portion;

one end of the lamp holder part is connected with one end of the handle part in a clamping way;

the lamp head part is internally provided with a light source generator, the handle part is internally provided with a photoelectric control unit and a power supply, or the lamp head part is internally provided with a light source generator and a photoelectric control unit, and the handle part is internally provided with a power supply;

the photoelectric control unit is detachably connected with the power supply;

the photoelectric control unit is detachably connected with the light source generator and can control the light source generator to emit light;

the light source generator is provided with a first light source generator and a second light source generator;

the first light source generator can emit light with the wave band comprising 253.7nm +/-10 nm wavelength;

the second light source generator is capable of emitting light in a wavelength band including 385nm to 450 nm.

Preferably, when the lamp head part is internally provided with the light source generator, and the handle part is internally provided with the photoelectric control unit and the power supply,

the power supply is a battery;

the light source generator further includes: lamp stand, first circuit board and first butt joint:

the first circuit board is arranged on the lamp holder and is electrically connected with the lamp holder;

the first circuit board is electrically connected with the first pair of connectors;

the lamp holder, the circuit board and the first butt joint are all arranged in a shell of the lamp holder part;

wherein the lamp holder is located on the shell at one end of the emission light source, and the first butt joint is located at the other end of the shell.

Preferably, the first light source generator and the second light source generator are both mounted on the lamp socket and electrically connected thereto;

the first light source generator comprises two LED lamp beads with 253.7nm +/-10 nm of emitted light wavelength;

the second light source generator comprises two LED lamp beads with the emission light wavelength of 385nm-450 nm.

Preferably, the handle portion further comprises: a handle housing;

the handle shell is of a hollow cylindrical structure;

the photoelectric control unit and the battery are arranged in the handle shell.

Preferably, the photoelectric control unit includes: the second circuit board, the control key assembly, the battery charging contact head and the second pair of joints;

the second circuit board is respectively connected with the control key assembly, the battery charging contact head and the second pair of joints.

Preferably, one end of the second pair of joints is in power supply connection with the battery, and the other end of the second pair of joints is provided with two spring type electrodes;

the second pair of joints are arranged at the end part of the handle shell;

the first pair of joints of the lamp holder part can be clamped into the handle shell and is in conductive connection with the two spring type electrodes on the second pair of joints.

Preferably, the ends of the second pair of joints are provided with concentric annular electrode contact surfaces;

the inner circle of the concentric ring electrode contact surface is a positive electrode, and the outer circle is a negative electrode.

Preferably, the battery charging contact is provided with two electrodes, is connected with the tail of the battery and is used for charging the battery;

the control key assembly is arranged on the outer wall of the handle shell;

the control key assembly includes: a start key and a stop key.

Preferably, the base part further comprises: a housing and a visual shield;

the light source generator is arranged in the shell;

one end of the shell is connected with the handle part, and the other end of the shell is provided with the visual protective cover;

the visual protective cover is made of ultraviolet and blue light filtering materials, and other light sources are reserved to pass through the visual protective cover.

The end part of the visual protective cover is provided with a sponge.

Preferably, the method further comprises the following steps: a light guide rod;

the light guide rod is connected with the light source generator;

the power supply includes: one or more of dry batteries, storage batteries and external power supplies.

(III) advantageous effects

The invention has the beneficial effects that: the parasite killing device provided by the invention not only can find germs and pests on plants and vertebrates, but also can assist in killing the parasites and germs on the plants and vertebrates, and the whole process has low cost, environmental protection and obvious effect.

Drawings

Fig. 1 is a schematic overall structure diagram of a parasite inactivating apparatus provided by the present invention;

fig. 2 is a schematic overall structure diagram of a parasite inactivating device provided by the present invention;

[ description of reference ]

1. A first light source generator; 2. a second light source generator; 3. a sponge; 4. a visual shield; 5. connecting and clamping positions; 6. a clamping position; 7. a start key; 8. a stop key; 9. a regulator; 10. direction indication; 11. a support frame.

Detailed Description

For the purpose of better explaining the present invention and to facilitate understanding, the present invention will be described in detail by way of specific embodiments with reference to the accompanying drawings.

As shown in fig. 1 and 2: the embodiment discloses a parasite inactivation device, including: a base portion and a handle portion.

One end of the lamp holder part is connected with one end of the handle part in a clamping way; the lamp head part is internally provided with a light source generator, the handle part is internally provided with a photoelectric control unit and a power supply, or the lamp head part is internally provided with a light source generator and a photoelectric control unit, and the handle part is internally provided with a power supply; the photoelectric control unit is detachably connected with the power supply; the photoelectric control unit is detachably connected with the light source generator and can control the light source generator to emit light.

Wherein, the light source generator is provided with a first light source generator 1 and a second light source generator 2; the first light source generator 1 is capable of emitting light in a wavelength band including 253.7nm ± 10 nm; the second light source generator 2 is capable of emitting light in a wavelength band including 385nm to 450 nm.

Specifically, in this embodiment, when the light source generator is disposed in the light head portion, and the photoelectric control unit and the power supply are disposed in the handle portion, the power supply is a battery; the light source generator further comprises: lamp stand, first circuit board and first butt joint: the first circuit board is arranged on the lamp holder and is electrically connected with the lamp holder, and the first circuit board is electrically connected with the first pair of connectors; the lamp holder, the circuit board and the first butt joint are all arranged in a shell of the lamp holder part; wherein the lamp holder is located on the shell at one end of the emission light source, and the first butt joint is located at the other end of the shell.

In the present embodiment, the first light source generator 1 and the second light source generator 2 are both mounted on the lamp socket and electrically connected to the lamp socket; the first light source generator 1 comprises two LED lamp beads with 253.7nm +/-10 nm of emitted light wavelength; the second light source generator 2 comprises two LED lamp beads with the emission light wavelength of 385nm-450 nm.

Specifically, in this embodiment, the handle portion further includes: a handle housing; the handle shell is of a hollow cylindrical structure; the photoelectric control unit and the battery are arranged in the handle shell. The photoelectric control unit includes: the second circuit board, the control key assembly, the battery charging contact head and the second pair of joints; the second circuit board is respectively connected with the control key assembly, the battery charging contact head and the second pair of joints. One end of the second pair of joints is in power supply connection with the battery, and the other end of the second pair of joints is provided with two spring type electrodes; the second pair of joints are arranged at the end part of the handle shell; the first pair of joints of the lamp holder part can be clamped into the handle shell and is in conductive connection with the two spring type electrodes on the second pair of joints.

Specifically, the end parts of the second pair of joints are provided with concentric annular electrode contact surfaces; the inner circle of the concentric ring electrode contact surface is a positive electrode, and the outer circle is a negative electrode. The battery charging contact is provided with two electrodes, is connected with the tail of the battery and is used for charging the battery; the control key assembly is arranged on the outer wall of the handle shell; the control key assembly includes: a start key 7 and a stop key 8.

Finally, it should be noted that: the lamp cap part further comprises: a housing and a visual shield 4; the light source generator is arranged in the shell; one end of the shell is connected with the handle part, and the other end of the shell is provided with the visual protective cover 4; the end part of the visual protective cover is provided with a sponge 3.

The parasite inactivation device provided in this embodiment further comprises: a light guide rod; the light guide rod is connected with the light source generator. The power supply includes: one or more of dry batteries, storage batteries and external power supplies.

It should be noted that: the using method is that the light source generated by the device provided by the embodiment is directly adopted to directly irradiate the affected area. In practical application, the illumination intensity is determined according to the thickness required to penetrate, the adjustment is carried out, the illumination is changed from weak to strong, and each layer is irradiated sufficiently as far as possible. The size of the illumination area of the light source is determined according to the size of the area of the lesion. The single time and the number of times of irradiation are designed according to the pathogen and the self condition of the host. The distance of light source contact is set according to the condition of the parasite and the host.

The parasites described in this example include: bacteria, viruses, fungi, spores and parasites.

The principle for the visual and light-activated parasite inactivation in this example is as follows:

1. biomolecules such as nucleic acids and proteins are the material basis of life;

2. the light with the wavelength of 253.7nm or more can be absorbed by pyrimidine and purine in nucleic acid of microorganisms, so that the nucleic acid is promoted to generate a plurality of photoproducts through a base dimerization mode, and the molecular structure of deoxyribonucleic acid (DNA) and ribonucleic acid (RNA) is damaged. When the molecular structure is disrupted, the nucleic acid is difficult to replicate, and even if replication is possible, it often has defects, causing death of microorganisms and micro-invertebrates.

3. Organisms are composed of cells, except for viruses, which are RNA structures.

4. The most basic characteristic of organisms is metabolism, and most of microbial proteins contain porphyrin rings, wherein coproporphyrin can be activated into high-energy unstable porphyrin by blue light (wavelength of more than or equal to 405 nm), and then is combined with triplet oxygen to form unstable singlet oxygen, and the triplet oxygen is combined with compounds on cell membranes to damage the cell membranes, so that the cells die. Under the same condition, the porphyrin can also generate fluorescence, and can be clearly seen under the action of filtering.

5. Blue light can also kill microorganisms and micro-invertebrates by affecting transmembrane ion influx and altering intracellular PH.

6. The total amount of cells of vertebrates and plants is far larger than that of parasites, the reproducible amount is far more than that of parasites, and the parasites of microorganisms and micro invertebrates can be safely and reliably solved by adopting artificial light.

Therefore, the artificial light source with the 200-280nm wave band is directed to the nucleic acid of the pathogen; the artificial light source with the wave band of 405nm-450nm aims at the oxidized protein photolyzed by pathogens.

In the embodiment, the shell of the base part is provided with a connecting clamp 5 of the base part, which can be connected with a perspective protective cover or a light guide rod, as shown in fig. 1; one end of the light source generator emits light, the other end of the light source generator is provided with a positive electrode and a negative electrode, the light source generator can be operated after being electrified, the end is connected with a battery through a quick connection design, a clamping position 6 is shown in figure 1, a switch is arranged on a handle part, a start key 7 is pressed down, the device is started and defaulted to be in a strong light mode, light reaches a required value instantly, meanwhile, the default working time of the device is 40 seconds, the device automatically stops working when the working time is up, a prompt tone is generated every 2 seconds during the working period, and if the required period is ended, a stop key 8 is pressed down, and the. If the start key is pressed for 5 seconds, the device is changed into a flash mode, the start key is pressed again, the device is started, the light reaches a required value instantly, meanwhile, the default working time of the device is 40 seconds, the device stops for 1 second every 2 seconds, namely, the light sounds a prompt tone every 2 seconds, stops for 1 second and lights for 2 seconds, and the operation is stopped after the default setting time is completed for 40 seconds. During which a stop key is pressed if necessary to terminate. If necessary, switch back to the highlight mode and press the start key for 5 seconds. One side of the handle part is connected with the light source generator, the other side is provided with a charging port and an illumination intensity adjuster 9 which is a circular ring, 4 gears of 800lx, 1200lx, 2200lx and 3200lx are arranged on the circular ring, and as shown in a direction indicator 10 in fig. 1, the circular ring can be adjusted by twisting to a fixed position.

As shown in fig. 2, a low sodium lamp with heat dissipation is installed at one end of the parasite inactivating device in the embodiment by a spiral interface, wherein 1 wavelength is 253.7nm ± 10 nm; wherein 2, the wavelength is 405nm +/-10 nm; the light source generator and the photoelectric control unit are both arranged in the lamp holder part, according to the position of the circuit component, a switch is arranged on the lamp holder part, the start key 7 is pressed, the device is started and is in a high light mode in a default mode, light reaches a required value instantly, meanwhile, the device has a working time of 200 seconds in a default mode, the device stops working automatically when the time is up, during the period, a prompt tone is generated every 2 seconds, and if the working time is up, the stop key 8 is pressed, and the working stops. If the start key is pressed for 5 seconds, the device is changed into a flash mode, the start key is pressed again, the device is started, the light reaches a required value instantly, meanwhile, the default working time of the device is 200 seconds, every 2 seconds is spaced in the period, the device stops for 1 second, namely, every 2 seconds the light is on, a prompt sound is sounded, the device stops for 1 second and then lights for 2 seconds, the operation is circulated, the default setting time is completed for 200 seconds, and the work is stopped. During which a stop key is pressed if necessary to terminate. If necessary, switch back to the highlight mode and press the start key for 5 seconds. The photoelectric control unit of the device and the power supply assembly are connected with the light source generator on one side, the charging port and the illumination intensity regulator 9 are arranged on the other side, the photoelectric control unit of the device is a circular ring, 4 gears including 3200lx, 5000lx, 12000lx and 20000lx are arranged on the circular ring, and the circular ring can be adjusted to a fixed position by twisting. The blower is embedded in the lamp head part and blows towards the lamp bulb, and the blower is started when the light source starts to work until the work stops. The other end of handle portion has fast interface, can connect support frame 11 or unmanned aerial vehicle.

Example 1

The experimental person is free of photosensitivity, and aims at killing pathogens of trichophyton rubrum, trichophyton mentagrophytes, trichophyton floccosum, filamentous fungi, yeast-like bacteria, yeasts, sporomycetes, fusarium and candida albicans which are parasitic in human onychomycosis, the device uses a rechargeable battery as a power supply, adopts a 10W power LED as an inlaid luminous element of a light source generator, simultaneously adopts a wave band of 253.7nm +/-10 nm and a wave band of two 385nm-450nm, has the illumination intensity of 2200lx, uses a shading element as a transparent protective cover, covers one large side of the protective cover in a diseased region, compresses sponge on the protective cover, prevents radiation leakage, opens a light source to obviously fluoresce bacteria, accurately finds the position of a parasitic source, irradiates 10 seconds of light at the same position, uses a flash mode, three times per day and 6 days later, and has no activity and no dormancy of the parasitic. The newly grown nails of experimenters are normal, and sick nails are gradually sheared off according to the newly grown nails, so that healthy nails can be obtained, which is a successful parasite killing case.

Example 2

The experimental person is free of photosensitivity, aiming at Propionibacterium Acnes (PA) parasitized on the human face, the equipment uses a rechargeable battery as a power supply, adopts a 10w power LED as an inlaid light-emitting piece of a light source generator, simultaneously generates a band I253.7 nm +/-10 nm and a band II 385nm-450nm, and has the illumination intensity of 800-. After the metabolism of the experimenter, no inflammation phenomenon occurs in the affected area 30 days after the irradiation is finished, and the facial health is obtained. This is a successful parasite inactivation case.

Example 3

The experimental person is free of photosensitivity, aiming at Propionibacterium Acnes (PA) parasitized on the human face, the equipment is connected with a fixed power supply, a 10w power LED is used as an inlaid light emitting piece of a light source generator, a band I253.7 nm +/-10 nm and a band II 385nm-450nm occur simultaneously, the illumination intensity is 800-. After the metabolism of the experimenter, no inflammation phenomenon occurs in the affected area 30 days after the irradiation is finished, and the facial health is obtained. This is a successful parasite inactivation case.

Example 4

The experimental house has no light allergy, and aims at subcutaneous parasitic scabies, itch mites and demodex of livestock, the equipment uses a rechargeable battery as a power supply, adopts a 10w power LED as an inlaid luminous element of a light source generator, simultaneously generates a first 253.7nm +/-10 nm wave band and a second 385nm-450nm wave band, has the illumination intensity of 800-. Healthy skin is obtained. This is a successful parasite inactivation case.

EXAMPLE 5

Aiming at cryptotrichum amabilis, trichina, trichodina, fish lice, ductus obliquus, ichthyophthirius multifiliis, sipunculus glaucosus and sipunculus siphonatus of fish, the equipment is connected with a fixed power supply, a 10w power LED is used as an embedded luminous element of a light source generator, the first 253.7nm +/-10 nm waveband and the second 385nm-450nm waveband are generated simultaneously, the illumination intensity is 2000lx, the shading element is used as a goggles, the light source is connected with a light guide rod because the fish is in water, other parts except the light guide rod are on the water surface, the bacterial fluorescence can be obviously seen by turning on the light source, the position of a parasite source can be accurately found, the parasite source is moved once every 30 seconds in the same position until the disease area is comprehensively irradiated for three times every day by matching with an antibody, and no active parasite and no dormancy exist in the irradiation area after. After two weeks healthy fish were obtained. This is a successful parasite inactivation case.

Example 6

Aiming at plant anthracnose, the equipment is connected with a solar charging battery, a low-sodium lamp is used as an inlaid light-emitting piece of a light source generator, a 253.7nm +/-10 nm waveband and a 385nm-450nm waveband are generated simultaneously, the illumination intensity is 20000lx, the light-shielding piece is used as goggles in operation, a use area can be separated by a light shielding plate, a support is used for the equipment to be fixed within a range of 1 m away from a plant, an intense light mode is used, the period of a disease high-incidence area can be repeated every day for 40-200 seconds every time, after one week, the diffusion of the anthracnose in the irradiation area is obviously slowed down, and the area of the anthrax infection is obviously. This is a successful parasite inactivation case.

Example 7

Aiming at pseudomonas, xanthomonas, agrobacterium, corynebacterium, erwinia and mycoplasma in a high-incidence stage of plant parasitic bacteria, the equipment is connected with a solar charging battery, a low sodium lamp is used as an embedded luminous element of a light source generator, a first waveband of 253.7nm +/-10 nm and a second waveband of 385nm-450nm occur simultaneously, the illumination intensity is 20000lx, the operation and use shading element is a goggles, a use area can be separated by a shading plate, an equipment use bracket is fixed in a range of 1 m away from a plant, a strong light mode is used, each time is 40-200 seconds, the high-incidence stage of diseases can be carried out for multiple times every day, and after one week, bacteria in the irradiation area can not cause agricultural damage. This is a successful parasite inactivation case.

Example 8

Aiming at parasitic aphids, white spiders and borers cubs of hot peppers, the device shown in the figure 2 is used, qualified goggles are selected as a main radiation protection tool, the lamp holder part is connected with the support frame which is a 4-meter long rod, so that a person is away from a light emitting opening by more than 2 meters, a strong light mode is selected, the illumination intensity is selected to be 20000lx, a light source is started, parasitic fluorescence, such as green fluorescence, can be obviously seen under the action of a wave band of two 385nm-450nm through the goggles, the operator holds the rod and sends the light source to a diseased region for irradiation, the sound is prompted for 15 times every 30 seconds, and after the parasitic aphids, the operator can listen to the prompting sound without seeing the light source. Under the irradiation of 253.7nm +/-10 nm wave band and 385-450 nm wave band and 20000lx illumination intensity, parasites and cubs die rapidly and worm eggs cannot escape.

The technical principles of the present invention have been described above in connection with specific embodiments, which are intended to explain the principles of the present invention and should not be construed as limiting the scope of the present invention in any way. Based on the explanations herein, those skilled in the art will be able to conceive of other embodiments of the present invention without inventive efforts, which shall fall within the scope of the present invention.

Claims

1. A parasite inactivation device, comprising: a base portion and a handle portion;

the photoelectric control unit is detachably connected with the power supply;

2. A parasite inactivation device according to claim 1,

when the light source generator is arranged in the lamp holder part and the photoelectric control unit and the power supply are arranged in the handle part,

the power supply is a battery;

3. A parasite inactivation device according to claim 2,

the first light source generator and the second light source generator are both mounted on the lamp holder and electrically connected with the lamp holder;

4. A parasite inactivation device according to claim 2,

the handle portion further includes: a handle housing;

the handle shell is of a hollow cylindrical structure;

5. A parasite inactivation device according to claim 4,

the photoelectric control unit includes: the second circuit board, the control key assembly, the battery charging contact head and the second pair of joints;

6. A parasite inactivation device according to claim 5,

one end of the second pair of joints is in power supply connection with the battery, and the other end of the second pair of joints is provided with two spring type electrodes;

the second pair of joints are arranged at the end part of the handle shell;

7. A parasite inactivation device according to claim 6,

the end parts of the second pair of joints are provided with concentric annular electrode contact surfaces;

8. A parasite inactivation device according to claim 6,

the battery charging contact is provided with two electrodes, is connected with the tail of the battery and is used for charging the battery;

the control key assembly is arranged on the outer wall of the handle shell;

the control key assembly includes: a start key and a stop key.

9. A parasite inactivation device according to claim 1,

the lamp cap part further comprises: a housing and a visual shield;

the light source generator is arranged in the shell;

the end part of the visual protective cover is provided with a sponge.

10. A parasite inactivation device according to claim 1,

further comprising: a light guide rod;

the light guide rod is connected with the light source generator;